Uǧur Akar

1.3k total citations
26 papers, 1.0k citations indexed

About

Uǧur Akar is a scholar working on Molecular Biology, Epidemiology and Hematology. According to data from OpenAlex, Uǧur Akar has authored 26 papers receiving a total of 1.0k indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 7 papers in Epidemiology and 6 papers in Hematology. Recurrent topics in Uǧur Akar's work include Retinoids in leukemia and cellular processes (8 papers), Autophagy in Disease and Therapy (5 papers) and Acute Myeloid Leukemia Research (4 papers). Uǧur Akar is often cited by papers focused on Retinoids in leukemia and cellular processes (8 papers), Autophagy in Disease and Therapy (5 papers) and Acute Myeloid Leukemia Research (4 papers). Uǧur Akar collaborates with scholars based in United States, Türkiye and Japan. Uǧur Akar's co-authors include Bülent Özpolat, Gabriel Lopez‐Berestein, Kapil Mehta, Yasuko Kondo, Gabriel N. Hortobágyi, Esen Özkaya‐Bayazit, Banu Arun, Banu Arun, Angelica M. Gutierrez‐Barrera and Ana M. Tari and has published in prestigious journals such as Blood, Journal of the American Academy of Dermatology and Autophagy.

In The Last Decade

Uǧur Akar

24 papers receiving 992 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Uǧur Akar United States 14 571 272 190 172 106 26 1.0k
Sung‐Chul Lim South Korea 20 551 1.0× 155 0.6× 305 1.6× 151 0.9× 56 0.5× 56 1.0k
Srinivas Chava United States 20 614 1.1× 251 0.9× 154 0.8× 400 2.3× 54 0.5× 41 1.2k
Chenglong Li China 20 668 1.2× 148 0.5× 152 0.8× 187 1.1× 47 0.4× 66 1.1k
Donald P. Lombardi United States 16 643 1.1× 90 0.3× 298 1.6× 188 1.1× 91 0.9× 26 1.4k
Yuhui Zang China 19 593 1.0× 214 0.8× 121 0.6× 389 2.3× 87 0.8× 44 1.2k
Manuela Minguzzi Italy 21 383 0.7× 176 0.6× 106 0.6× 190 1.1× 160 1.5× 33 997
Yasutaka Shuno Japan 17 393 0.7× 388 1.4× 344 1.8× 129 0.8× 47 0.4× 53 1.1k
Matheus Dyczynski Sweden 7 554 1.0× 446 1.6× 111 0.6× 235 1.4× 37 0.3× 8 928
Yongsheng Ge China 22 542 0.9× 133 0.5× 236 1.2× 207 1.2× 40 0.4× 50 1.0k
Lijing Wang China 20 668 1.2× 119 0.4× 178 0.9× 299 1.7× 45 0.4× 66 1.2k

Countries citing papers authored by Uǧur Akar

Since Specialization
Citations

This map shows the geographic impact of Uǧur Akar's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Uǧur Akar with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Uǧur Akar more than expected).

Fields of papers citing papers by Uǧur Akar

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Uǧur Akar. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Uǧur Akar. The network helps show where Uǧur Akar may publish in the future.

Co-authorship network of co-authors of Uǧur Akar

This figure shows the co-authorship network connecting the top 25 collaborators of Uǧur Akar. A scholar is included among the top collaborators of Uǧur Akar based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Uǧur Akar. Uǧur Akar is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Tekedereli, İbrahim, Uǧur Akar, S. Neslihan Alpay, Gabriel Lopez‐Berestein, & Bülent Özpolat. (2018). Autophagy is Required to Regulate Mitochondria Renewal, Cell Attachment, and All-trans–Retinoic Acid–Induced Differentiation in NB4 Acute Promyelocytic Leukemia Cells. Journal of Environmental Pathology Toxicology and Oncology. 38(1). 13–20. 5 indexed citations
2.
Arun, Banu, Uǧur Akar, Angelica M. Gutierrez‐Barrera, Gabriel N. Hortobágyi, & Bülent Özpolat. (2015). The PARP inhibitor AZD2281 (Olaparib) induces autophagy/mitophagy in BRCA1 and BRCA2 mutant breast cancer cells. International Journal of Oncology. 47(1). 262–268. 88 indexed citations
3.
4.
Özpolat, Bülent, Uǧur Akar, İbrahim Tekedereli, et al.. (2012). PKCδRegulates Translation Initiation through PKR and eIF2αin Response to Retinoic Acid in Acute Myeloid Leukemia Cells. PubMed. 2012. 1–17. 8 indexed citations
5.
Ekmekçioğlu, Sühendan, et al.. (2011). Zyflamend Mediates Therapeutic Induction of Autophagy to Apoptosis in Melanoma Cells. Nutrition and Cancer. 63(6). 940–949. 11 indexed citations
6.
Arun, Banu, Vicente Valero, Diane Liu, et al.. (2011). Short-Term Biomarker Modulation Prevention Study of Anastrozole in Women at Increased Risk for Second Primary Breast Cancer. Cancer Prevention Research. 5(2). 276–282. 1 indexed citations
7.
Bayraktar, Soley, Angelica M. Gutierrez‐Barrera, Diane Liu, et al.. (2011). Outcome of triple-negative breast cancer in patients with or without deleterious BRCA mutations. Breast Cancer Research and Treatment. 130(1). 145–153. 84 indexed citations
8.
Zhang, Dongwei, Ana M. Tari, Uǧur Akar, et al.. (2010). Silencing Kinase-Interacting Stathmin Gene Enhances Erlotinib Sensitivity by Inhibiting Ser10 p27 Phosphorylation in Epidermal Growth Factor Receptor–Expressing Breast Cancer. Molecular Cancer Therapeutics. 9(11). 3090–3099. 17 indexed citations
9.
Akar, Uǧur, Bülent Özpolat, Kapil Mehta, et al.. (2010). Targeting p70S6K Prevented Lung Metastasis in a Breast Cancer Xenograft Model. Molecular Cancer Therapeutics. 9(5). 1180–1187. 41 indexed citations
10.
Akar, Uǧur, Ana M. Tari, Ángela Sanguino, et al.. (2008). Silencing of Bcl-2 expression by small interfering RNA induces autophagic cell death in MCF-7 breast cancer cells. Autophagy. 4(5). 669–679. 215 indexed citations
11.
12.
Özpolat, Bülent, Uǧur Akar, Michael Steiner, et al.. (2007). Programmed Cell Death-4 Tumor Suppressor Protein Contributes to Retinoic Acid–Induced Terminal Granulocytic Differentiation of Human Myeloid Leukemia Cells. Molecular Cancer Research. 5(1). 95–108. 77 indexed citations
13.
Özpolat, Bülent, Uǧur Akar, Kapil Mehta, & Gabriel Lopez‐Berestein. (2007). PKCδ and Tissue Transglutaminase are Novel Inhibitors of Autophagy in Pancreatic Cancer Cells. Autophagy. 3(5). 480–483. 68 indexed citations
14.
Akar, Uǧur, Bülent Özpolat, Nancy H. Colburn, & Gabriel Lopez‐Berestein. (2006). p38 MAPK Signaling Mediates Retinoic Acid-Induced Expression of a Novel Tumor Suppressor Protein Programmed Cell Death 4 (PDCD4) in Acute Promyelocytic Leukemia Cells.. Blood. 108(11). 1942–1942.
15.
Özpolat, Bülent, Uǧur Akar, Yasuko Kondo, & Gabriel Lopez‐Berestein. (2006). All-Trans-Retinoic Acid (ATRA) Induces Autophagy in Acute Promyelocytic Leukemia Cells.. Blood. 108(11). 2012–2012. 1 indexed citations
16.
Demokan, Semra, et al.. (2005). Polymorphisms of the XRCC1 DNA repair gene in head and neck cancer. Pathology & Oncology Research. 11(1). 22–25. 28 indexed citations
17.
Özkaya‐Bayazit, Esen & Uǧur Akar. (2001). Fixed drug eruption induced by trimethoprim-sulfamethoxazole: Evidence for a link to HLA-A30 B13 Cw6 haplotype. Journal of the American Academy of Dermatology. 45(5). 712–717. 61 indexed citations
18.
Türkmen, Aydın, Gülsüm Ak, Yavuz Furuncuoğlu, et al.. (2000). Relationship between Gingival Hyperplasia and Class II Histocompatibility Antigens in Renal Transplant Recipients. ˜The œNephron journals/Nephron journals. 84(1). 29–31. 6 indexed citations
19.
Kavak, Ayşe, Can Baykal, Güzin Özarmağan, & Uǧur Akar. (2000). HLA in alopecia areata. International Journal of Dermatology. 39(8). 589–592. 59 indexed citations
20.
Neyzi, Olcay, et al.. (1991). An educational intervention on promotion of breast feeding. Paediatric and Perinatal Epidemiology. 5(3). 286–298. 19 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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