Tsuyoshi Narimiya

409 total citations
9 papers, 328 citations indexed

About

Tsuyoshi Narimiya is a scholar working on Molecular Biology, Rheumatology and Genetics. According to data from OpenAlex, Tsuyoshi Narimiya has authored 9 papers receiving a total of 328 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 3 papers in Rheumatology and 3 papers in Genetics. Recurrent topics in Tsuyoshi Narimiya's work include Bone Metabolism and Diseases (7 papers), Bone and Dental Protein Studies (3 papers) and Oral microbiology and periodontitis research (2 papers). Tsuyoshi Narimiya is often cited by papers focused on Bone Metabolism and Diseases (7 papers), Bone and Dental Protein Studies (3 papers) and Oral microbiology and periodontitis research (2 papers). Tsuyoshi Narimiya collaborates with scholars based in Japan. Tsuyoshi Narimiya's co-authors include Satoshi Wada, Hiroyuki Kanzaki, Yuta Katsumata, Yuuki Yamaguchi, Kanako Itohiya, Yutaka Miyamoto, Sari Fukaya, Yoshiki Nakamura, Yoshiki Nakamura and Hiroshi Tomonari and has published in prestigious journals such as Frontiers in Physiology, Polymers and Journal of Cellular and Molecular Medicine.

In The Last Decade

Tsuyoshi Narimiya

9 papers receiving 326 citations

Peers

Tsuyoshi Narimiya

MB

PERIO

CR

IMMUN

RHEUM

Yuta Katsumata Japan

Hirohito Kato Japan

Keqing Pan China

Kanako Itohiya Japan

Yiyang Jiang China

Tetsuya Yoshimoto Japan

Fuhua Yan China

Satoru Shindo Japan

E.‐C. Kim South Korea

Hongbing He China

Yuta Katsumata Japan

Tsuyoshi Narimiya

205 ×1.2

MB

98 ×1.0

PERIO

50 ×1.0

CR

59 ×1.3

IMMUN

38 ×1.0

RHEUM

Citations per year, relative to Tsuyoshi Narimiya Tsuyoshi Narimiya (= 1×) peers Yuta Katsumata

Countries citing papers authored by Tsuyoshi Narimiya

Since Specialization

Citations

This map shows the geographic impact of Tsuyoshi Narimiya's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tsuyoshi Narimiya with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tsuyoshi Narimiya more than expected).

Fields of papers citing papers by Tsuyoshi Narimiya

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tsuyoshi Narimiya. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tsuyoshi Narimiya. The network helps show where Tsuyoshi Narimiya may publish in the future.

Co-authorship network of co-authors of Tsuyoshi Narimiya

This figure shows the co-authorship network connecting the top 25 collaborators of Tsuyoshi Narimiya. A scholar is included among the top collaborators of Tsuyoshi Narimiya based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tsuyoshi Narimiya. Tsuyoshi Narimiya is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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9 of 9 papers shown

1.

Wada, Satoshi, Hiroyuki Kanzaki, Yuta Katsumata, et al.. (2020). Bach1 Inhibition Suppresses Osteoclastogenesis via Reduction of the Signaling via Reactive Oxygen Species by Reinforced Antioxidation. Frontiers in Cell and Developmental Biology. 8. 740–740. 19 indexed citations

2.

Kanzaki, Hiroyuki, Satoshi Wada, Yuuki Yamaguchi, et al.. (2019). Compression and tension variably alter Osteoprotegerin expression via miR-3198 in periodontal ligament cells. BMC Molecular and Cell Biology. 20(1). 6–6. 26 indexed citations

3.

Narimiya, Tsuyoshi, Hiroyuki Kanzaki, Yuki Yamaguchi, et al.. (2019). Nrf2 activation in osteoblasts suppresses osteoclastogenesis via inhibiting IL-6 expression.. Bone Reports. 11. 100228–100228. 31 indexed citations

4.

Katsumata, Yuta, Hiroyuki Kanzaki, Yoshitomo Honda, et al.. (2018). Single Local Injection of Epigallocatechin Gallate-Modified Gelatin Attenuates Bone Resorption and Orthodontic Tooth Movement in Mice. Polymers. 10(12). 1384–1384. 19 indexed citations

5.

Kanzaki, Hiroyuki, Satoshi Wada, Tsuyoshi Narimiya, et al.. (2017). Pathways that Regulate ROS Scavenging Enzymes, and Their Role in Defense Against Tissue Destruction in Periodontitis. Frontiers in Physiology. 8. 351–351. 142 indexed citations

6.

Wada, Satoshi, Hiroyuki Kanzaki, Tsuyoshi Narimiya, & Yoshiki Nakamura. (2017). Novel device for application of continuous mechanical tensile strain to mammalian cells. Biology Open. 6(4). 518–524. 14 indexed citations

7.

Yamaguchi, Yuuki, Hiroyuki Kanzaki, Yuta Katsumata, et al.. (2017). Dimethyl fumarate inhibits osteoclasts via attenuation of reactive oxygen species signalling by augmented antioxidation. Journal of Cellular and Molecular Medicine. 22(2). 1138–1147. 60 indexed citations

8.

Narimiya, Tsuyoshi, et al.. (2017). Orthodontic tensile strain induces angiogenesis via type IV collagen degradation by matrix metalloproteinase‐12. Journal of Periodontal Research. 52(5). 842–852. 16 indexed citations

9.

Itohiya, Kanako, Hiroyuki Kanzaki, Satoshi Wada, et al.. (2016). Occlusal hypofunction mediates alveolar bone apposition via relative augmentation of TGF-βsignaling by decreased Asporin production in rats. 3(1). 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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