Tomoya Ozaki
- Molecular Biology
- Reproductive Medicine top 5%
- Genetics
- Cell Biology top 10%
- Public Health, Environmental and Occupational Health
- Co-authors
- Haruhiko KanasakiKentaro TakahashiKohji MiyazakiHiroko KuriokaRitsuto FujiwakiKohkichi HataMasako OkadaKenji Kadomatsu
- Topics
- Ovarian function and disorders (12 papers)Reproductive Biology and Fertility (9 papers)Proteoglycans and glycosaminoglycans research (7 papers)
- Partner nations
- JapanUnited StatesFrance
In The Last Decade
Tomoya Ozaki
41 papers receiving 811 citations
Peers
Comparison fields: 5 of 87
- Molecular Biology 320
- Reproductive Medicine 227
- Genetics 116
- Cell Biology 111
- Public Health, Environmental and Occupational Health 102
Countries citing papers authored by Tomoya Ozaki
This map shows the geographic impact of Tomoya Ozaki's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tomoya Ozaki with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tomoya Ozaki more than expected).
Fields of papers citing papers by Tomoya Ozaki
This network shows the impact of papers produced by Tomoya Ozaki. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tomoya Ozaki. The network helps show where Tomoya Ozaki may publish in the future.
Co-authorship network of co-authors of Tomoya Ozaki
This figure shows the co-authorship network connecting the top 25 collaborators of Tomoya Ozaki. A scholar is included among the top collaborators of Tomoya Ozaki based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tomoya Ozaki. Tomoya Ozaki is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 5 | |
| 2 | 11 | |
| 3 | 5 | |
| 4 | 24 | |
| 5 | 15 | |
| 6 | 12 | |
| 7 | 36 | |
| 8 | 6 | |
| 9 | 8 | |
| 10 | 5 | |
| 11 | 20 | |
| 12 | 4 | |
| 13 | 19 | |
| 14 | 108 | |
| 15 | A case of a pregnant woman with luteal insufficiency and a mutation in the beta-subunit of luteinizing hormone. | 5 |
| 16 | 4 | |
| 17 | 37 | |
| 18 | 32 | |
| 19 | 14 | |
| 20 | 9 |
About Tomoya Ozaki
Tomoya Ozaki is a scholar working on Reproductive Medicine, Sensory Systems and Endocrine and Autonomic Systems, having authored 42 papers that have together received 828 indexed citations. Recurring topics across this work include Ovarian function and disorders (12 papers), Reproductive Biology and Fertility (9 papers) and Proteoglycans and glycosaminoglycans research (7 papers). The work is most often cited by research in Reproductive Medicine (227 citations), Obstetrics and Gynecology (83 citations) and Endocrine and Autonomic Systems (49 citations). Tomoya Ozaki has collaborated with scholars based in Japan, United States and France. Frequent co-authors include Haruhiko Kanasaki, Kentaro Takahashi, Kohji Miyazaki, Hiroko Kurioka, Ritsuto Fujiwaki, Kohkichi Hata, Masako Okada, Kenji Kadomatsu, Kazuma Sakamoto and K. Miyazaki. Their work appears in journals such as Journal of Biological Chemistry, The Journal of Physiology and Scientific Reports.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.