Tomas Jakobsson

1.2k total citations
17 papers, 920 citations indexed

About

Tomas Jakobsson is a scholar working on Surgery, Molecular Biology and Oncology. According to data from OpenAlex, Tomas Jakobsson has authored 17 papers receiving a total of 920 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Surgery, 9 papers in Molecular Biology and 6 papers in Oncology. Recurrent topics in Tomas Jakobsson's work include Cholesterol and Lipid Metabolism (10 papers), Drug Transport and Resistance Mechanisms (6 papers) and Cancer, Lipids, and Metabolism (2 papers). Tomas Jakobsson is often cited by papers focused on Cholesterol and Lipid Metabolism (10 papers), Drug Transport and Resistance Mechanisms (6 papers) and Cancer, Lipids, and Metabolism (2 papers). Tomas Jakobsson collaborates with scholars based in Sweden, United States and France. Tomas Jakobsson's co-authors include Knut R. Steffensen, Eckardt Treuter, Jan-Ακε Gustafsson, Nicolas Venteclef, Anastasios Damdimopoulos, Anna Ehrlund, Paolo Parini, Rongrong Fan, Ewa Ellis and Olli A. Jänne and has published in prestigious journals such as Journal of Biological Chemistry, Genes & Development and Molecular Cell.

In The Last Decade

Tomas Jakobsson

17 papers receiving 908 citations

Peers

Tomas Jakobsson

SURGE

ONCOL

IMMUN

Ayce Yesilaltay United States

Daniel Rodrı́guez-Agudo United States

Hailing Liao United States

Matthew M. Molusky United States

Kamal D. Mehta United States

Judith Magenheim Israel

Tsugumichi Saito Japan

Jia Nie China

M Witt Germany

Maria T. Bengoechea-Alonso Sweden

Ayce Yesilaltay United States

Tomas Jakobsson

503 ×1.0

409 ×0.9

SURGE

168 ×0.7

ONCOL

157 ×0.9

IMMUN

130 ×0.9

Citations per year, relative to Tomas Jakobsson Tomas Jakobsson (= 1×) peers Ayce Yesilaltay

Countries citing papers authored by Tomas Jakobsson

Since Specialization

Citations

This map shows the geographic impact of Tomas Jakobsson's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tomas Jakobsson with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tomas Jakobsson more than expected).

Fields of papers citing papers by Tomas Jakobsson

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tomas Jakobsson. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tomas Jakobsson. The network helps show where Tomas Jakobsson may publish in the future.

Co-authorship network of co-authors of Tomas Jakobsson

This figure shows the co-authorship network connecting the top 25 collaborators of Tomas Jakobsson. A scholar is included among the top collaborators of Tomas Jakobsson based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tomas Jakobsson. Tomas Jakobsson is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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17 of 17 papers shown

Huang, Zhiqiang, Ning Liang, Saioa Goñi, et al.. (2021). The corepressors GPS2 and SMRT control enhancer and silencer remodeling via eRNA transcription during inflammatory activation of macrophages. Molecular Cell. 81(5). 953–968.e9. 32 indexed citations

Jakobsson, Tomas, Francesco Ravaioli, Massoud Vosough, et al.. (2021). Gene Editing Correction of a Urea Cycle Defect in Organoid Stem Cell Derived Hepatocyte-like Cells. International Journal of Molecular Sciences. 22(3). 1217–1217. 17 indexed citations

Pramfalk, Camilla, Tomas Jakobsson, Maria Olin, et al.. (2020). Generation of new hepatocyte-like in vitro models better resembling human lipid metabolism. Biochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids. 1865(6). 158659–158659. 5 indexed citations

Liang, Ning, Tomas Jakobsson, Rongrong Fan, & Eckardt Treuter. (2019). The Nuclear Receptor—Co-repressor Complex in Control of Liver Metabolism and Disease. Frontiers in Endocrinology. 10. 411–411. 28 indexed citations

Santinha, Deolinda, Anna Kłopot, Igor Marques, et al.. (2019). Lipidomic analysis of human primary hepatocytes following LXR activation with GW3965 identifies AGXT2L1 as a main target associated to changes in phosphatidylethanolamine. The Journal of Steroid Biochemistry and Molecular Biology. 198. 105558–105558. 7 indexed citations

Fontana, Raffaella, Laura Raccosta, Lucrezia Rovati, et al.. (2018). Nuclear receptor ligands induce TREM-1 expression on dendritic cells: analysis of their role in tumors. OncoImmunology. 8(3). 1554967–1554967. 14 indexed citations

Jakobsson, Tomas, Lise‐Lotte Vedin, & Paolo Parini. (2017). Potential Role of Thyroid Receptor β Agonists in the Treatment of Hyperlipidemia. Drugs. 77(15). 1613–1621. 20 indexed citations

Treuter, Eckardt, Rongrong Fan, Zhiqiang Huang, Tomas Jakobsson, & Nicolas Venteclef. (2017). Transcriptional repression in macrophages—basic mechanisms and alterations in metabolic inflammatory diseases. FEBS Letters. 591(19). 2959–2977. 29 indexed citations

Jakobsson, Tomas, Nicolas Venteclef, Dario Greco, et al.. (2014). The oxysterol receptor LXRβ protects against DSS- and TNBS-induced colitis in mice. Mucosal Immunology. 7(6). 1416–1428. 44 indexed citations

10.

Steffensen, Knut R., Tomas Jakobsson, & Jan-Ακε Gustafsson. (2013). Targeting liver X receptors in inflammation. Expert Opinion on Therapeutic Targets. 17(8). 977–990. 52 indexed citations

11.

González‐Granillo, Marcela, Knut R. Steffensen, Omar Granados, et al.. (2012). Soy protein isoflavones differentially regulate liver X receptor isoforms to modulate lipid metabolism and cholesterol transport in the liver and intestine in mice. Diabetologia. 55(9). 2469–2478. 41 indexed citations

12.

Jakobsson, Tomas, Eckardt Treuter, Jan-Ακε Gustafsson, & Knut R. Steffensen. (2012). Liver X receptor biology and pharmacology: new pathways, challenges and opportunities. Trends in Pharmacological Sciences. 33(7). 394–404. 263 indexed citations

13.

Venteclef, Nicolas, Tomas Jakobsson, Knut R. Steffensen, & Eckardt Treuter. (2011). Metabolic nuclear receptor signaling and the inflammatory acute phase response. Trends in Endocrinology and Metabolism. 22(8). 333–343. 81 indexed citations

14.

Venteclef, Nicolas, Tomas Jakobsson, Anna Ehrlund, et al.. (2010). GPS2-dependent corepressor/SUMO pathways govern anti-inflammatory actions of LRH-1 and LXRβ in the hepatic acute phase response. Genes & Development. 24(4). 381–395. 154 indexed citations

15.

Jakobsson, Tomas, Nicolas Venteclef, Gudrun Toresson, et al.. (2009). GPS2 Is Required for Cholesterol Efflux by Triggering Histone Demethylation, LXR Recruitment, and Coregulator Assembly at the ABCG1 Locus. Molecular Cell. 34(4). 510–518. 98 indexed citations

16.

Antonson, Per, Tomas Jakobsson, Tova Almlöf, et al.. (2008). RAP250 Is a Coactivator in the Transforming Growth Factor β Signaling Pathway That Interacts with Smad2 and Smad3. Journal of Biological Chemistry. 283(14). 8995–9001. 21 indexed citations

17.

Jakobsson, Tomas, et al.. (2007). Molecular basis for repression of liver X receptor-mediated gene transcription by receptor-interacting protein 140. Biochemical Journal. 405(1). 31–39. 14 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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