Togo Ikuta

2.1k total citations
31 papers, 1.7k citations indexed

About

Togo Ikuta is a scholar working on Molecular Biology, Health, Toxicology and Mutagenesis and Oncology. According to data from OpenAlex, Togo Ikuta has authored 31 papers receiving a total of 1.7k indexed citations (citations by other indexed papers that have themselves been cited), including 19 papers in Molecular Biology, 9 papers in Health, Toxicology and Mutagenesis and 5 papers in Oncology. Recurrent topics in Togo Ikuta's work include Toxic Organic Pollutants Impact (9 papers), Effects and risks of endocrine disrupting chemicals (6 papers) and Nuclear Structure and Function (6 papers). Togo Ikuta is often cited by papers focused on Toxic Organic Pollutants Impact (9 papers), Effects and risks of endocrine disrupting chemicals (6 papers) and Nuclear Structure and Function (6 papers). Togo Ikuta collaborates with scholars based in Japan, Sweden and United States. Togo Ikuta's co-authors include Kaname Kawajiri, Yasuhito Kobayashi, Hidetaka Eguchi, Yoshihiro Yoneda, Taro Tachibana, Yoshiaki Fujii‐Kuriyama, Osamu Gotoh, Manabu Koike, Tadahiro Shiomi and Takashi Miyasaka and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Molecular and Cellular Biology.

In The Last Decade

Togo Ikuta

30 papers receiving 1.7k citations

Peers

Togo Ikuta
Markus R. Probst United States
Jennifer Marlowe United States
Eli V. Hestermann United States
Brett D. Hollingshead United States
Ellen C. Henry United States
Chunhua Qin United States
Soona Shin United States
Togo Ikuta
Citations per year, relative to Togo Ikuta Togo Ikuta (= 1×) peers Cornelia Dietrich

Countries citing papers authored by Togo Ikuta

Since Specialization
Citations

This map shows the geographic impact of Togo Ikuta's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Togo Ikuta with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Togo Ikuta more than expected).

Fields of papers citing papers by Togo Ikuta

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Togo Ikuta. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Togo Ikuta. The network helps show where Togo Ikuta may publish in the future.

Co-authorship network of co-authors of Togo Ikuta

This figure shows the co-authorship network connecting the top 25 collaborators of Togo Ikuta. A scholar is included among the top collaborators of Togo Ikuta based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Togo Ikuta. Togo Ikuta is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Ikuta, Togo, et al.. (2024). Abortion in AhR-knockout mice and fetomaternal immunity. Reproductive Biology. 24(4). 100952–100952.
2.
Fujimoto, Hirofumi, Togo Ikuta, Aki Koike, & Manabu Koike. (2022). Acetylation of the nuclear localization signal in Ku70 diminishes the interaction with importin-α. Biochemistry and Biophysics Reports. 33. 101418–101418. 3 indexed citations
3.
Ikuta, Togo, Aki Koike, & Manabu Koike. (2021). Detection of double-stranded DNA breaks and apoptosis induced by bleomycin in mouse intestine. The Journal of Toxicological Sciences. 46(12). 611–618. 1 indexed citations
4.
Ikuta, Togo, Masafumi Kurosumi, Toshimasa Yatsuoka, & Yoji Nishimura. (2016). Tissue distribution of aryl hydrocarbon receptor in the intestine: Implication of putative roles in tumor suppression. Experimental Cell Research. 343(2). 126–134. 37 indexed citations
5.
Ikuta, Togo, Yasuhito Kobayashi, Masato Kitazawa, et al.. (2013). ASC-associated inflammation promotes cecal tumorigenesis in aryl hydrocarbon receptor-deficient mice. Carcinogenesis. 34(7). 1620–1627. 52 indexed citations
6.
Ikuta, Togo, Motoi Ohba, Christos C. Zouboulis, Yoshiaki Fujii‐Kuriyama, & Kaname Kawajiri. (2010). B lymphocyte-induced maturation protein 1 is a novel target gene of aryl hydrocarbon receptor. Journal of Dermatological Science. 58(3). 211–216. 30 indexed citations
7.
Ikuta, Togo, Takeshi Namiki, Yoshiaki Fujii‐Kuriyama, & Kaname Kawajiri. (2008). AhR protein trafficking and function in the skin. Biochemical Pharmacology. 77(4). 588–596. 59 indexed citations
8.
Ikuta, Togo & Kaname Kawajiri. (2006). Zinc finger transcription factor Slug is a novel target gene of aryl hydrocarbon receptor. Experimental Cell Research. 312(18). 3585–3594. 72 indexed citations
9.
10.
Ikuta, Togo, et al.. (2005). Growth suppression of Leydig TM3 cells mediated by aryl hydrocarbon receptor. Biochemical and Biophysical Research Communications. 331(4). 902–908. 8 indexed citations
11.
Ikuta, Togo, Yasuhito Kobayashi, & Kaname Kawajiri. (2004). Cell Density Regulates Intracellular Localization of Aryl Hydrocarbon Receptor. Journal of Biological Chemistry. 279(18). 19209–19216. 102 indexed citations
12.
Kawajiri, Kaname & Togo Ikuta. (2004). Regulation of Nucleo-Cytoplasmic Transport of the Aryl Hydrocarbon Receptor. JOURNAL OF HEALTH SCIENCE. 50(3). 215–219. 6 indexed citations
13.
Kawana, Katsuyoshi, Togo Ikuta, Yasuhito Kobayashi, et al.. (2003). Molecular Mechanism of Nuclear Translocation of an Orphan Nuclear Receptor, SXR. Molecular Pharmacology. 63(3). 524–531. 95 indexed citations
14.
Watanabe, Junko, Tsutomu Shimada, Elizabeth M. J. Gillam, et al.. (2000). Association of CYP1B1 genetic polymorphism with incidence to breast and lung cancer. Pharmacogenetics. 10(1). 25–33. 148 indexed citations
15.
Koike, Manabu, Togo Ikuta, Takashi Miyasaka, & Tadahiro Shiomi. (1999). Ku80 can translocate to the nucleus independent of the translocation of Ku70 using its own nuclear localization signal. Oncogene. 18(52). 7495–7505. 67 indexed citations
16.
Koike, Manabu, Togo Ikuta, Takashi Miyasaka, & Tadahiro Shiomi. (1999). The Nuclear Localization Signal of the Human Ku70 Is a Variant Bipartite Type Recognized by the Two Components of Nuclear Pore-Targeting Complex. Experimental Cell Research. 250(2). 401–413. 53 indexed citations
17.
Ikuta, Togo, et al.. (1998). Cholesterol Sulfate Activates Transcription of Transglutaminase 1 Gene in Normal Human Keratinocytes. Journal of Investigative Dermatology. 111(6). 1098–1102. 36 indexed citations
18.
Eguchi, Hidetaka, Togo Ikuta, Taro Tachibana, Yoshihiro Yoneda, & Kaname Kawajiri. (1997). A Nuclear Localization Signal of Human Aryl Hydrocarbon Receptor Nuclear Translocator/Hypoxia-inducible Factor 1β Is a Novel Bipartite Type Recognized by the Two Components of Nuclear Pore-targeting Complex. Journal of Biological Chemistry. 272(28). 17640–17647. 101 indexed citations
19.
Kasahara, Kohji, et al.. (1993). Rapid phosphorylation of 28‐kDa heat‐shock protein by treatment with okadaic acid and phorbol ester of BALB/MK‐2 mouse kerationocytes. European Journal of Biochemistry. 213(3). 1101–1107. 14 indexed citations
20.
Ikuta, Togo, Yoshio Honma, Junko Okabe‐Kado, Takashi Kasukabe, & Motoo Hozumi. (1989). Normal mouse lung tissue produces a growth-inhibitory factor(s) preferential for mouse monocytic leukemia cells. Cancer Immunology Immunotherapy. 30(3). 139–144. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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