Tobias Obser

2.7k total citations
70 papers, 1.9k citations indexed

About

Tobias Obser is a scholar working on Hematology, Immunology and Genetics. According to data from OpenAlex, Tobias Obser has authored 70 papers receiving a total of 1.9k indexed citations (citations by other indexed papers that have themselves been cited), including 55 papers in Hematology, 22 papers in Immunology and 16 papers in Genetics. Recurrent topics in Tobias Obser's work include Platelet Disorders and Treatments (52 papers), Blood groups and transfusion (30 papers) and Complement system in diseases (21 papers). Tobias Obser is often cited by papers focused on Platelet Disorders and Treatments (52 papers), Blood groups and transfusion (30 papers) and Complement system in diseases (21 papers). Tobias Obser collaborates with scholars based in Germany, United States and France. Tobias Obser's co-authors include Reinhard Schneppenheim, Ulrich Budde, Florian Oyen, Sonja Schneppenheim, Frauke Gräter, Carsten Baldauf, Michael C. Frühwald, Martin Hasselblatt, Reiner Siebert and Stefan Gesk and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Circulation and The Journal of Chemical Physics.

In The Last Decade

Tobias Obser

70 papers receiving 1.9k citations

Peers

Countries citing papers authored by Tobias Obser

Since Specialization

Citations

This map shows the geographic impact of Tobias Obser's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tobias Obser with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tobias Obser more than expected).

Fields of papers citing papers by Tobias Obser

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tobias Obser. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tobias Obser. The network helps show where Tobias Obser may publish in the future.

Co-authorship network of co-authors of Tobias Obser

This figure shows the co-authorship network connecting the top 25 collaborators of Tobias Obser. A scholar is included among the top collaborators of Tobias Obser based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tobias Obser. Tobias Obser is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Title	Journal	Authors	Indexed citations
1	Single Molecule Force Spectroscopy at Cell Surfaces to Study Physical Properties of Heparan Sulfate Chains and Protein‐Heparan Sulfate Interactions		Tobias Obser, Christian Gorzelanny et al.	1
2	Medulloblastoma in children with Fanconi anemia: Association with FA-D1/FA-N, SHH type and poor survival independent of treatment strategies	Neuro-Oncology	Denise Obrecht, Pablo Hernáiz Driever et al.	1
3	Platelet adhesion and aggregate formation controlled by immobilised and soluble VWF	BMC Molecular and Cell Biology	Matthias F. Schneider, Christian Meß et al.	9
4	Multiplexed protein force spectroscopy reveals equilibrium protein folding dynamics and the low-force response of von Willebrand factor	Proceedings of the National Academy of Sciences	Achim Löf, Steffen M. Sedlak et al.	65
5	Evidence for the Misfolding of the A1 Domain within Multimeric von Willebrand Factor in Type 2 von Willebrand Disease	Journal of Molecular Biology	Alexander Tischer, Maria A. Brehm et al.	11
6	The von Willebrand factor Tyr2561 allele is a gain-of-function variant and a risk factor for early myocardial infarction	Blood	Reinhard Schneppenheim, Maria A. Brehm et al.	19
7	Enhanced Local Disorder in a Clinically Elusive von Willebrand Factor Provokes High-Affinity Platelet Clumping	Journal of Molecular Biology	Alexander Tischer, Venkata R. Machha et al.	20
8	Mutation G1629E Increases von Willebrand Factor Cleavage via a Cooperative Destabilization Mechanism	Biophysical Journal	Camilo Aponte‐Santamaría, Judith J. Mittag et al.	9
9	Force Sensing by the Vascular Protein Von Willebrand Factor is Tuned by a Strong Intermonomer Interaction	Biophysical Journal	Achim Löf, Tobias Obser et al.	1
10	pH-Dependent Interactions in Dimers Govern the Mechanics and Structure of von Willebrand Factor	Biophysical Journal	Achim Löf, Tobias Obser et al.	16
11	Shear-Induced Unfolding and Enzymatic Cleavage of Full-Length VWF Multimers	Biophysical Journal	Matthias Radtke, Tobias Obser et al.	58
12	Identification and characterization of the elusive mutation causing the historical von Willebrand Disease type IIC Miami	Journal of Thrombosis and Haemostasis	Tobias Obser, Florian Oyen et al.	7
13	Mechanosensitive Von Willebrand Factor Protein-Protein Interactions Regulate Hemostasis	Biophysical Journal	Volker Huck, Agnieszka K. Bronowska et al.	1
14	Performance evaluation and multicentre study of a von Willebrand factor activity assay based on GPIb binding in the absence of ristocetin	Blood Coagulation & Fibrinolysis	Ulrich Budde, A Huber et al.	56
15	Common large partial VWF gene deletion does not cause alloantibody formation in the Hungarian type 3 von Willebrand disease population	Journal of Thrombosis and Haemostasis	Z Boda, Hajna Losonczy et al.	11
16	Germline Nonsense Mutation and Somatic Inactivation of SMARCA4/BRG1 in a Family with Rhabdoid Tumor Predisposition Syndrome	The American Journal of Human Genetics	Reinhard Schneppenheim, Michael C. Frühwald et al.	222
17	Shear‐induced unfolding activates von Willebrand factor A2 domain for proteolysis	Journal of Thrombosis and Haemostasis	Carsten Baldauf, Reinhard Schneppenheim et al.	98
18	Genetic defects in von Willebrand disease type 3 in Indian and Greek patients	Blood Cells Molecules and Diseases	Pawan Gupta, Renu Saxena et al.	20
19	Recombinant expression of mutations causing von Willebrand disease type Normandy: characterization of a combined defect of factor VIII binding and multimerization	Thrombosis and Haemostasis	H. Lenk, Tobias Obser et al.	19
20	Dissecting the molecular mechanism of ADAMTS13 deficiency by expression of recombinant protein		Reinhard Schneppenheim, Tobias Obser et al.	1

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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