Sonja Schneppenheim

1.3k total citations
39 papers, 880 citations indexed

About

Sonja Schneppenheim is a scholar working on Hematology, Immunology and Genetics. According to data from OpenAlex, Sonja Schneppenheim has authored 39 papers receiving a total of 880 indexed citations (citations by other indexed papers that have themselves been cited), including 32 papers in Hematology, 15 papers in Immunology and 7 papers in Genetics. Recurrent topics in Sonja Schneppenheim's work include Platelet Disorders and Treatments (29 papers), Complement system in diseases (15 papers) and Blood groups and transfusion (15 papers). Sonja Schneppenheim is often cited by papers focused on Platelet Disorders and Treatments (29 papers), Complement system in diseases (15 papers) and Blood groups and transfusion (15 papers). Sonja Schneppenheim collaborates with scholars based in Germany, United States and Canada. Sonja Schneppenheim's co-authors include Ulrich Budde, Reinhard Schneppenheim, Tobias Obser, Wolf Hassenpflug, Carsten Baldauf, Frauke Gräter, Florian Oyen, Jihan Zhou, Wolfram Stacklies and E. Drewke and has published in prestigious journals such as Circulation, Blood and PLoS ONE.

In The Last Decade

Sonja Schneppenheim

39 papers receiving 862 citations

Peers

Sonja Schneppenheim
Shizuko Tsuji Japan
Maneesh Arya United States
Runhui Wu China
Nobuyuki Minami Japan
Ann Dahlberg United States
Jean‐Pierre Farcet France
Lambros Kordelas Germany
J. P. Jouet France
Gavivann Veerakul Thailand
SA Krilis Australia
Shizuko Tsuji Japan
Sonja Schneppenheim
Citations per year, relative to Sonja Schneppenheim Sonja Schneppenheim (= 1×) peers Shizuko Tsuji

Countries citing papers authored by Sonja Schneppenheim

Since Specialization
Citations

This map shows the geographic impact of Sonja Schneppenheim's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sonja Schneppenheim with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sonja Schneppenheim more than expected).

Fields of papers citing papers by Sonja Schneppenheim

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sonja Schneppenheim. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sonja Schneppenheim. The network helps show where Sonja Schneppenheim may publish in the future.

Co-authorship network of co-authors of Sonja Schneppenheim

This figure shows the co-authorship network connecting the top 25 collaborators of Sonja Schneppenheim. A scholar is included among the top collaborators of Sonja Schneppenheim based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sonja Schneppenheim. Sonja Schneppenheim is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Doevelaar, Adrian, Bodo Hölzer, Felix S. Seibert, et al.. (2023). Generation of potentially inhibitory autoantibodies to ADAMTS13 in coronavirus disease 2019. Scientific Reports. 13(1). 10501–10501. 3 indexed citations
2.
Große, Gerrit M., Jan‐Thorben Sieweke, Sonja Schneppenheim, et al.. (2023). ADAMTS-13 activity in stroke of known and unknown cause: Relation to vascular risk factor burden. Frontiers in Neurology. 13. 1045478–1045478. 2 indexed citations
3.
Doevelaar, Adrian, Bodo Hölzer, Felix S. Seibert, et al.. (2021). von Willebrand Factor Multimer Formation Contributes to Immunothrombosis in Coronavirus Disease 2019. Critical Care Medicine. 49(5). e512–e520. 41 indexed citations
4.
Lehmann, Katja, Christian Meß, Tobias Obser, et al.. (2020). Upshaw-Schulman syndrome-associated ADAMTS13 variants possess proteolytic activity at the surface of endothelial cells and in simulated circulation. PLoS ONE. 15(5). e0232637–e0232637. 3 indexed citations
5.
Löf, Achim, Sonja Schneppenheim, Reinhard Schneppenheim, et al.. (2019). Advancing multimer analysis of von Willebrand factor by single-molecule AFM imaging. PLoS ONE. 14(1). e0210963–e0210963. 4 indexed citations
6.
Budde, Ulrich, Francesco Tona, Antonella Bertomoro, et al.. (2018). Link between von Willebrand factor multimers, relapses and coronary microcirculation in patients with thrombotic thrombocytopenic purpura in remission. Thrombosis Research. 173. 42–47. 1 indexed citations
7.
Obser, Tobias, Julia Bode, Florian Oyen, et al.. (2018). Genetic and Functional Characterization of ADAMTS13 Variants in a Patient Cohort with Upshaw–Schulman Syndrome Investigated in Germany. Thrombosis and Haemostasis. 47(4). 709–722. 9 indexed citations
8.
Lavin, Michelle, Sonia Águila, Sonja Schneppenheim, et al.. (2017). Novel insights into the clinical phenotype and pathophysiology underlying low VWF levels. Blood. 130(21). 2344–2353. 98 indexed citations
9.
Noone, Damien, Magdalena Riedl, Fred G. Pluthero, et al.. (2016). Von Willebrand factor regulates complement on endothelial cells. Kidney International. 90(1). 123–134. 49 indexed citations
10.
Obser, Tobias, Florian Oyen, Maria A. Brehm, et al.. (2016). Identification and characterization of the elusive mutation causing the historical von Willebrand Disease type IIC Miami. Journal of Thrombosis and Haemostasis. 14(9). 1725–1735. 7 indexed citations
11.
Dicke, Christina, Sonja Schneppenheim, Katharina Holstein, et al.. (2016). Distinct mechanisms account for acquired von Willebrand syndrome in plasma cell dyscrasias. Annals of Hematology. 95(6). 945–957. 34 indexed citations
12.
Dicke, Christina, Katharina Holstein, Sonja Schneppenheim, et al.. (2014). Acquired hemophilia A and von Willebrand syndrome in a patient with late-onset systemic lupus erythematosus. Experimental Hematology and Oncology. 3(1). 21–21. 12 indexed citations
13.
Huck, Volker, Camilo Aponte‐Santamaría, Sandra Grässle, et al.. (2014). von Willebrand disease type 2A phenotypes IIC, IID and IIE: A day in the life of shear-stressed mutant von Willebrand factor. Thrombosis and Haemostasis. 112(7). 96–108. 30 indexed citations
14.
Schneppenheim, Sonja, Ulrich Budde, Moritz Seiffert, et al.. (2012). Abstract 17447: Correction of Acquired Von Willebrand Disease in Patients Undergoing Trans-Catheter Aortic Valve Replacement. Circulation. 126(8). 945–53. 1 indexed citations
15.
16.
Solomon, Cristina, Ulrich Budde, Sonja Schneppenheim, et al.. (2011). Acquired type 2A von Willebrand syndrome caused by aortic valve disease corrects during valve surgery. British Journal of Anaesthesia. 106(4). 494–500. 30 indexed citations
17.
Baldauf, Carsten, Reinhard Schneppenheim, Wolfram Stacklies, et al.. (2009). Shear‐induced unfolding activates von Willebrand factor A2 domain for proteolysis. Journal of Thrombosis and Haemostasis. 7(12). 2096–2105. 98 indexed citations
18.
Schröder, Jörg, et al.. (2009). The problem of novel FVIII missense mutations for haemophilia A genetic counseling. Hämostaseologie. 29(2). 158–160. 4 indexed citations
19.
Santer, René, Johannes Rischewski, Sonja Schneppenheim, et al.. (2005). The spectrum of aldolase B (ALDOB) mutations and the prevalence of hereditary fructose intolerance in Central Europe. Human Mutation. 25(6). 594–594. 46 indexed citations
20.
Lenk, H., Tobias Obser, Johannes Oldenburg, et al.. (2004). Recombinant expression of mutations causing von Willebrand disease type Normandy: characterization of a combined defect of factor VIII binding and multimerization. Thrombosis and Haemostasis. 92(7). 36–41. 19 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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