Tobias Eisenberger

1.6k total citations
14 papers, 531 citations indexed

About

Tobias Eisenberger is a scholar working on Molecular Biology, Genetics and Sensory Systems. According to data from OpenAlex, Tobias Eisenberger has authored 14 papers receiving a total of 531 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 7 papers in Genetics and 4 papers in Sensory Systems. Recurrent topics in Tobias Eisenberger's work include Retinal Development and Disorders (7 papers), Hearing, Cochlea, Tinnitus, Genetics (4 papers) and Genetic and Kidney Cyst Diseases (4 papers). Tobias Eisenberger is often cited by papers focused on Retinal Development and Disorders (7 papers), Hearing, Cochlea, Tinnitus, Genetics (4 papers) and Genetic and Kidney Cyst Diseases (4 papers). Tobias Eisenberger collaborates with scholars based in Germany, Saudi Arabia and United Kingdom. Tobias Eisenberger's co-authors include Hanno J. Bolz, Christine Neuhaus, Carsten Bergmann, Martin Gliem, Peter Charbel Issa, Johannes Birtel, Christian Betz, Frank G. Holz, Steffen Lenzner and Elisabeth Mangold and has published in prestigious journals such as PLoS ONE, Scientific Reports and Human Molecular Genetics.

In The Last Decade

Tobias Eisenberger

14 papers receiving 522 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Tobias Eisenberger Germany 11 437 175 165 75 66 14 531
Samer Khateb Israel 16 525 1.2× 178 1.0× 147 0.9× 67 0.9× 94 1.4× 43 662
Emma Bedoukian United States 13 238 0.5× 56 0.3× 119 0.7× 61 0.8× 30 0.5× 31 375
Eva Lenassi United Kingdom 17 595 1.4× 436 2.5× 141 0.9× 77 1.0× 54 0.8× 29 859
Polona Le Quesne Stabej United Kingdom 14 348 0.8× 46 0.3× 167 1.0× 123 1.6× 32 0.5× 18 535
Ilaria Zito United Kingdom 10 449 1.0× 117 0.7× 240 1.5× 18 0.2× 68 1.0× 15 560
Krysta Voesenek Netherlands 9 863 2.0× 228 1.3× 524 3.2× 44 0.6× 154 2.3× 11 1.0k
Penny Clouston United Kingdom 12 613 1.4× 201 1.1× 452 2.7× 11 0.1× 56 0.8× 34 850
Liesbeth M. Bleeker-Wagemakers Netherlands 10 415 0.9× 63 0.4× 205 1.2× 15 0.2× 74 1.1× 12 557
Sophie Châtelin France 7 512 1.2× 316 1.8× 87 0.5× 12 0.2× 66 1.0× 8 585
Shoichi Irie Japan 7 395 0.9× 48 0.3× 80 0.5× 18 0.2× 36 0.5× 11 474

Countries citing papers authored by Tobias Eisenberger

Since Specialization
Citations

This map shows the geographic impact of Tobias Eisenberger's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tobias Eisenberger with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tobias Eisenberger more than expected).

Fields of papers citing papers by Tobias Eisenberger

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tobias Eisenberger. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tobias Eisenberger. The network helps show where Tobias Eisenberger may publish in the future.

Co-authorship network of co-authors of Tobias Eisenberger

This figure shows the co-authorship network connecting the top 25 collaborators of Tobias Eisenberger. A scholar is included among the top collaborators of Tobias Eisenberger based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tobias Eisenberger. Tobias Eisenberger is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

14 of 14 papers shown
1.
Epting, Daniel, Eva L. Decker, Elisabeth Ott, et al.. (2022). The ciliary transition zone protein TMEM218 synergistically interacts with the NPHP module and its reduced dosage leads to a wide range of syndromic ciliopathies. Human Molecular Genetics. 31(14). 2295–2306. 5 indexed citations
2.
Birtel, Johannes, Martin Gliem, Elisabeth Mangold, et al.. (2018). Next-generation sequencing identifies unexpected genotype-phenotype correlations in patients with retinitis pigmentosa. PLoS ONE. 13(12). e0207958–e0207958. 76 indexed citations
3.
Birtel, Johannes, Tobias Eisenberger, Martin Gliem, et al.. (2018). Clinical and genetic characteristics of 251 consecutive patients with macular and cone/cone-rod dystrophy. Scientific Reports. 8(1). 4824–4824. 147 indexed citations
4.
Eisenberger, Tobias, Nataliya Di Donato, Christian Decker, et al.. (2017). A C-terminal nonsense mutation links PTPRQ with autosomal-dominant hearing loss, DFNA73. Genetics in Medicine. 20(6). 614–621. 24 indexed citations
5.
Thoenes, Michaela, Ulrike Zimmermann, Inga Ebermann, et al.. (2015). OSBPL2 encodes a protein of inner and outer hair cell stereocilia and is mutated in autosomal dominant hearing loss (DFNA67). Orphanet Journal of Rare Diseases. 10(1). 15–15. 46 indexed citations
6.
Khan, Arif O., Tobias Eisenberger, Kerstin Nagel‐Wolfrum, Uwe Wolfrum, & Hanno J. Bolz. (2015). C21orf2is mutated in recessive early-onset retinal dystrophy with macular staphyloma and encodes a protein that localises to the photoreceptor primary cilium. British Journal of Ophthalmology. 99(12). 1725–1731. 35 indexed citations
7.
Eisenberger, Tobias, Christian Decker, Milan Hiersche, et al.. (2015). An Efficient and Comprehensive Strategy for Genetic Diagnostics of Polycystic Kidney Disease. PLoS ONE. 10(2). e0116680–e0116680. 47 indexed citations
8.
Eisenberger, Tobias, Nataliya Di Donato, Shahid Mahmood Baig, et al.. (2014). Targeted and Genomewide NGS Data Disqualify Mutations inMYO1A, the “DFNA48Gene”, as a Cause of Deafness. Human Mutation. 35(5). 565–570. 26 indexed citations
9.
Khan, Arif O., Carsten Bergmann, Tobias Eisenberger, & Hanno J. Bolz. (2014). ATULP1founder mutation, p.Gln301*, underlies a recognisable congenital rod–cone dystrophy phenotype on the Arabian Peninsula. British Journal of Ophthalmology. 99(4). 488–492. 15 indexed citations
10.
Fehrenbach, Henry, Christian Decker, Tobias Eisenberger, et al.. (2014). Mutations in WDR19 encoding the intraflagellar transport component IFT144 cause a broad spectrum of ciliopathies. Pediatric Nephrology. 29(8). 1451–1456. 30 indexed citations
11.
Neuhaus, Christine, Markus N. Preising, Arif O. Khan, et al.. (2013). High-coverage next-generation sequencing (NGS) for retinal dystrophies and Usher syndrome: High diagnostic yield, CNV detection, novel disease mechanisms and therapy targets. Investigative Ophthalmology & Visual Science. 54(15). 3373–3373. 1 indexed citations
12.
Khan, Arif O., Leen Abu‐Safieh, Tobias Eisenberger, Hanno J. Bolz, & Fowzan S. Alkuraya. (2013). TheRPGRIP1-related retinal phenotype in children. British Journal of Ophthalmology. 97(6). 760–764. 18 indexed citations
13.
Bergmann, Carsten, Saeed Reza Ghaffari, Manfred Schürmann, et al.. (2012). Next-generation Sequencing Of All Known Genes For RP And Allied Diseases Identifies The Causative Mutations In The Majority Of Patients. Investigative Ophthalmology & Visual Science. 53(14). 4525–4525. 1 indexed citations
14.
Eisenberger, Tobias, Rima Slim, Ahmad M. Mansour, et al.. (2012). Targeted next-generation sequencing identifies a homozygous nonsense mutation in ABHD12, the gene underlying PHARC, in a family clinically diagnosed with Usher syndrome type 3. Orphanet Journal of Rare Diseases. 7(1). 59–59. 60 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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2026