Ting Lin

737 total citations
22 papers, 516 citations indexed

About

Ting Lin is a scholar working on Nephrology, Physiology and Surgery. According to data from OpenAlex, Ting Lin has authored 22 papers receiving a total of 516 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Nephrology, 5 papers in Physiology and 4 papers in Surgery. Recurrent topics in Ting Lin's work include Renal Diseases and Glomerulopathies (5 papers), Immune Cell Function and Interaction (3 papers) and Acute Myeloid Leukemia Research (2 papers). Ting Lin is often cited by papers focused on Renal Diseases and Glomerulopathies (5 papers), Immune Cell Function and Interaction (3 papers) and Acute Myeloid Leukemia Research (2 papers). Ting Lin collaborates with scholars based in China, United States and United Kingdom. Ting Lin's co-authors include Kai‐Yuan Fu, Kai Li, Alan R. Light, Xinling Liang, Yuanhan Chen, Ruizhao Li, Shuangxin Liu, Xiaofan Tan, Qianmei Zhang and Xingchen Zhao and has published in prestigious journals such as Kidney International, The Journal of Pathology and Medicine.

In The Last Decade

Ting Lin

21 papers receiving 513 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Ting Lin China 11 142 139 120 97 76 22 516
Judit Hodrea Hungary 16 222 1.6× 45 0.3× 61 0.5× 118 1.2× 57 0.8× 26 558
Tetyana V. Pedchenko United States 14 256 1.8× 156 1.1× 26 0.2× 102 1.1× 37 0.5× 14 560
Laura Bonaccini Italy 9 137 1.0× 123 0.9× 36 0.3× 32 0.3× 38 0.5× 15 407
Anna Hammer Germany 10 188 1.3× 35 0.3× 52 0.4× 81 0.8× 21 0.3× 11 433
Xian Huang China 14 399 2.8× 68 0.5× 32 0.3× 96 1.0× 87 1.1× 19 639
Yuichiro Izumi Japan 13 245 1.7× 63 0.5× 88 0.7× 39 0.4× 28 0.4× 61 564
Martin Schepelmann Austria 13 160 1.1× 90 0.6× 81 0.7× 21 0.2× 14 0.2× 25 408
Sandra M. Soares United States 7 176 1.2× 98 0.7× 59 0.5× 31 0.3× 101 1.3× 8 578
Corry D. Bondi United States 8 189 1.3× 71 0.5× 111 0.9× 74 0.8× 15 0.2× 13 482

Countries citing papers authored by Ting Lin

Since Specialization
Citations

This map shows the geographic impact of Ting Lin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ting Lin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ting Lin more than expected).

Fields of papers citing papers by Ting Lin

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ting Lin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ting Lin. The network helps show where Ting Lin may publish in the future.

Co-authorship network of co-authors of Ting Lin

This figure shows the co-authorship network connecting the top 25 collaborators of Ting Lin. A scholar is included among the top collaborators of Ting Lin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ting Lin. Ting Lin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Wang, Wenjing, et al.. (2025). Distribution, Drug Resistance, and Risk Factors of Infection in Liver Transplant Recipients With Liver Failure. Transplantation Proceedings. 57(2). 305–311.
2.
Zhang, Qi, Ting Huang, Xiaomin Li, et al.. (2024). Prognostic impact of enhanced CD96 expression on NK cells by TGF-β1 in AML. International Immunopharmacology. 141. 112958–112958. 3 indexed citations
3.
Liu, Junru, et al.. (2024). Analysis of the Haematological Phenotype and Molecular Characteristics of Rare Abnormal Haemoglobin. Molecular Genetics & Genomic Medicine. 12(9). e70012–e70012. 1 indexed citations
4.
He, Wenfang, Chenguang Ding, Ting Lin, et al.. (2024). An enzyme-mimicking reactive oxygen species scavenger targeting oxidative stress-inflammation cycle ameliorates IR-AKI by inhibiting pyruvate dehydrogenase kinase 4. Theranostics. 14(19). 7534–7553. 5 indexed citations
5.
Lin, Ting, et al.. (2023). Simulation and experimental study of buried natural gas pipeline leak detection based on sound source characteristics. Thermal Science. 28(1 Part A). 101–113. 4 indexed citations
6.
Yang, Chuan, Sijia Li, Zhuo Li, et al.. (2022). Effects of predilution hemodiafiltration-induced intradialytic left ventricular systolic dysfunction compared to high-flux hemodialysis. International Urology and Nephrology. 54(10). 2653–2662. 1 indexed citations
7.
Zhang, Qi, Jingying Yang, Xiaomin Li, et al.. (2021). Increased TIGIT expressing NK cells with dysfunctional phenotype in AML patients correlated with poor prognosis. Cancer Immunology Immunotherapy. 71(2). 277–287. 52 indexed citations
8.
9.
Liang, Shun, Hong Zhang, Yue Du, et al.. (2018). RANK Deficiency Ameliorates Podocyte Injury by Suppressing Calcium/Calcineurin/ NFATc1 Signaling. Kidney & Blood Pressure Research. 43(4). 1149–1159. 7 indexed citations
10.
Zhao, Xingchen, Yuanhan Chen, Xiaofan Tan, et al.. (2018). Advanced glycation end‐products suppress autophagic flux in podocytes by activating mammalian target of rapamycin and inhibiting nuclear translocation of transcription factor EB. The Journal of Pathology. 245(2). 235–248. 75 indexed citations
11.
Lin, Ting, Li Zhang, Shuangxin Liu, et al.. (2017). WWC1 promotes podocyte survival via stabilizing slit diaphragm protein dendrin. Molecular Medicine Reports. 16(6). 8685–8690. 9 indexed citations
12.
Zhang, Li, Qianmei Zhang, Shuangxin Liu, et al.. (2017). DNA methyltransferase 1 may be a therapy target for attenuating diabetic nephropathy and podocyte injury. Kidney International. 92(1). 140–153. 82 indexed citations
13.
Liu, Shuangxin, Lixia Xu, Jianchao Ma, et al.. (2017). High‐volume continuous venovenous hemodiafiltration plus resin hemoperfusion improves severe metformin‐associated toxicity. Journal of Diabetes Investigation. 9(4). 975–978. 8 indexed citations
14.
Xie, Jingdun, et al.. (2017). Microglia-Synapse Pathways: Promising Therapeutic Strategy for Alzheimer’s Disease. BioMed Research International. 2017. 1–11. 18 indexed citations
15.
Tan, Xiaofan, Yuanhan Chen, Xinling Liang, et al.. (2016). Lipopolysaccharide-induced podocyte injury is mediated by suppression of autophagy. Molecular Medicine Reports. 14(1). 811–818. 12 indexed citations
16.
Li, R, Juan Ma, Changjiang Yu, et al.. (2016). Cdc42 deficiency induces podocyte apoptosis by inhibiting the Nwasp/stress fibers/YAP pathway. Cell Death and Disease. 7(3). e2142–e2142. 51 indexed citations
17.
18.
Lin, Huan-Bing, Yufang Cheng, Heng Zhou, et al.. (2011). Promotion of β-amyloid production by C-reactive protein and its implications in the early pathogenesis of Alzheimer’s disease. Neurochemistry International. 60(3). 257–266. 35 indexed citations
19.
Li, Kai, Ting Lin, Ye Cao, Alan R. Light, & Kai‐Yuan Fu. (2010). Peripheral Formalin Injury Induces 2 Stages of Microglial Activation in the Spinal Cord. Journal of Pain. 11(11). 1056–1065. 36 indexed citations
20.
Lin, Ting, et al.. (2007). Dissociation of spinal microglia morphological activation and peripheral inflammation in inflammatory pain models. Journal of Neuroimmunology. 192(1-2). 40–48. 57 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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