Tina Wiest

1.2k total citations
11 papers, 563 citations indexed

About

Tina Wiest is a scholar working on Molecular Biology, Genetics and Epidemiology. According to data from OpenAlex, Tina Wiest has authored 11 papers receiving a total of 563 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 4 papers in Genetics and 3 papers in Epidemiology. Recurrent topics in Tina Wiest's work include Connective tissue disorders research (3 papers), Congenital heart defects research (3 papers) and Neuroinflammation and Neurodegeneration Mechanisms (3 papers). Tina Wiest is often cited by papers focused on Connective tissue disorders research (3 papers), Congenital heart defects research (3 papers) and Neuroinflammation and Neurodegeneration Mechanisms (3 papers). Tina Wiest collaborates with scholars based in Germany, United States and Switzerland. Tina Wiest's co-authors include Elisabeth Schwarz, Christa Flechtenmacher, Franz X. Bosch, Christel Enders, Caspar Grond‐Ginsbach, Armin Grau, Ulrich Mansmann, Gerd U. Auffarth, Markus Schwaninger and Marius Hartmann and has published in prestigious journals such as Stroke, Oncogene and Cell and Tissue Research.

In The Last Decade

Tina Wiest

11 papers receiving 550 citations

Peers

Countries citing papers authored by Tina Wiest

Since Specialization

Citations

This map shows the geographic impact of Tina Wiest's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tina Wiest with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tina Wiest more than expected).

Fields of papers citing papers by Tina Wiest

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tina Wiest. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tina Wiest. The network helps show where Tina Wiest may publish in the future.

Co-authorship network of co-authors of Tina Wiest

This figure shows the co-authorship network connecting the top 25 collaborators of Tina Wiest. A scholar is included among the top collaborators of Tina Wiest based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tina Wiest. Tina Wiest is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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11 of 11 papers shown

#	Work	Indexed citations
1	Copy number variation in patients with cervical artery dissection 2012 ·European Journal of Human Genetics ·Caspar Grond‐Ginsbach, Bowang Chen, Rastislav Pjontek, Tina Wiest, (unknown), Barbara Burwinkel, Sandrine Tchatchou, Michael Krawczak, Stefan Schreiber, Tobias Brandt, Manja Kloß, (unknown), Kari Hemminki, Christoph Lichy, Philippe Lyrer, Ingrid Haußer, Stefan T. Engelter	23
2	Increased Expression of Cell–Cell Signaling Genes by Stimulated Mononuclear Leukocytes in Patients with Previous Atherothrombotic Stroke 2009 ·European Neurology ·Caspar Grond‐Ginsbach, Solveig Horstmann, Manuela Hummel, Tina Wiest, (unknown), (unknown), Manfred Hergenhahn, (unknown), Annette Weninger, (unknown), Ulrich Mansmann, Simone Wagner, Armin Grau	2
3	Gene expression in human peripheral blood mononuclear cells upon acute ischemic stroke 2008 ·Journal of Neurology ·Caspar Grond‐Ginsbach, (unknown), Tina Wiest, S. Horstmann, (unknown), Manfred Hergenhahn, Monica Hollstein, Ulrich Mansmann, Armin Grau, Simone Wagner	47
4	Organotypic co-cultures allow for immortalized human gingival keratinocytes to reconstitute a gingival epithelial phenotype in vitro 2006 ·Differentiation ·Mariana Roesch‐Ely, Thorsten Steinberg, (unknown), Eva Müssig, Noel J. Whitaker, Tina Wiest, Annette Kohl, G. Komposch, Pascal Tomakidi	40
5	Genetic Analysis of Familial Connective Tissue Alterations Associated With Cervical Artery Dissections Suggests Locus Heterogeneity 2006 ·Stroke ·Tina Wiest, (unknown), (unknown), (unknown), Alessandro Pezzini, Ingrid Haußer, Marie-Luise Arnold, Juan José Martin, Stefan T. Engelter, Philippe Lyrer, O. Busse, Tobias Brandt, Caspar Grond‐Ginsbach	20
6	MTHFR 677TT genotype increases the risk for cervical artery dissections 2006 ·Aktuelle Neurologie ·M. Kloss, Tina Wiest, (unknown), (unknown), (unknown), Christoph Lichy, Caspar Grond‐Ginsbach	2
7	Transcriptome analysis of peripheral blood mononuclear cells from stroke survivors 2006 ·Aktuelle Neurologie ·Caspar Grond‐Ginsbach, S. Horstmann, Tina Wiest, (unknown), Ulrich Mansmann, (unknown), Manfred Hergenhahn, Annette Weninger, Simone Wagner, Armin Grau	1
8	Expression of mRNAs encoding for growth factors, ECM molecules, and MMP13 in mono-cultures and co-cultures of human periodontal ligament fibroblasts and alveolar bone cells 2005 ·Cell and Tissue Research ·Stephan Winter, Annette Kohl, Andrea Huppertz, (unknown), Tina Wiest, G. Komposch, Pascal Tomakidi	9
9	Suggestive linkage of a putative genetic risk factor for cervical artery dissections to chromosome region 15q2 2004 ·Aktuelle Neurologie ·(unknown), (unknown), Tina Wiest, (unknown), Alessandro Pezzini, (unknown), Ingrid Haußer, Tobias Brandt, Caspar Grond‐Ginsbach	1
10	Multiple Levels of Regulation of the Interleukin-6 System in Stroke 2003 ·Stroke ·(unknown), Tina Wiest, Marius Hartmann, (unknown), Ulrich Mansmann, Gerd U. Auffarth, Armin Grau, (unknown), Caspar Grond‐Ginsbach, Markus Schwaninger	117
11	Involvement of intact HPV16 E6/E7 gene expression in head and neck cancers with unaltered p53 status and perturbed pRb cell cycle control 2002 ·Oncogene ·Tina Wiest, Elisabeth Schwarz, Christel Enders, Christa Flechtenmacher, Franz X. Bosch	301

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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