Tiewei Lv

797 total citations
33 papers, 567 citations indexed

About

Tiewei Lv is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Epidemiology. According to data from OpenAlex, Tiewei Lv has authored 33 papers receiving a total of 567 indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Molecular Biology, 14 papers in Cardiology and Cardiovascular Medicine and 11 papers in Epidemiology. Recurrent topics in Tiewei Lv's work include Congenital Heart Disease Studies (10 papers), Congenital heart defects research (5 papers) and Cardiomyopathy and Myosin Studies (5 papers). Tiewei Lv is often cited by papers focused on Congenital Heart Disease Studies (10 papers), Congenital heart defects research (5 papers) and Cardiomyopathy and Myosin Studies (5 papers). Tiewei Lv collaborates with scholars based in China, United States and Russia. Tiewei Lv's co-authors include Jing Zhu, Jie Tian, Xupei Huang, Bo Pan, Huichao Sun, Guozhen Chen, Lingjuan Liu, Lingjuan Liu, Yuhao Wu and Chun Wu and has published in prestigious journals such as SHILAP Revista de lepidopterología, PLoS ONE and Life Sciences.

In The Last Decade

Tiewei Lv

30 papers receiving 562 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Tiewei Lv China 12 284 130 100 90 84 33 567
Qihong Wu China 14 205 0.7× 82 0.6× 93 0.9× 49 0.5× 54 0.6× 34 503
Xiaohong Zhu China 16 409 1.4× 50 0.4× 57 0.6× 81 0.9× 53 0.6× 41 739
Wen Du China 16 283 1.0× 90 0.7× 34 0.3× 89 1.0× 38 0.5× 44 652
Rulin Zhuang China 13 385 1.4× 57 0.4× 92 0.9× 101 1.1× 50 0.6× 26 609
Jihwa Chung South Korea 15 200 0.7× 77 0.6× 50 0.5× 73 0.8× 39 0.5× 21 469
Ilaria Floris France 12 402 1.4× 82 0.6× 63 0.6× 305 3.4× 26 0.3× 23 709
Anning Yang China 18 415 1.5× 77 0.6× 40 0.4× 185 2.1× 66 0.8× 41 741
Weijing Feng China 15 316 1.1× 92 0.7× 102 1.0× 150 1.7× 68 0.8× 34 687
Kyung Hee Hong South Korea 10 211 0.7× 101 0.8× 46 0.5× 67 0.7× 55 0.7× 27 690
Hui Yuan China 11 215 0.8× 74 0.6× 99 1.0× 71 0.8× 67 0.8× 40 599

Countries citing papers authored by Tiewei Lv

Since Specialization
Citations

This map shows the geographic impact of Tiewei Lv's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tiewei Lv with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tiewei Lv more than expected).

Fields of papers citing papers by Tiewei Lv

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tiewei Lv. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tiewei Lv. The network helps show where Tiewei Lv may publish in the future.

Co-authorship network of co-authors of Tiewei Lv

This figure shows the co-authorship network connecting the top 25 collaborators of Tiewei Lv. A scholar is included among the top collaborators of Tiewei Lv based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tiewei Lv. Tiewei Lv is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Xu, Xiaohui, Xin Wang, Xin Zeng, et al.. (2024). Mitochondrial GRIM19 Loss Induces Liver Fibrosis through NLRP3/IL33 Activation via Reactive Oxygen Species/NF-кB Signaling. Journal of Clinical and Translational Hepatology. 12(6). 539–550. 3 indexed citations
2.
Wan, Kun, et al.. (2023). Establishment and identification of cardiomyocyte arhGEF18 gene conditional knockout mice. SHILAP Revista de lepidopterología. 1(2). e20–e20.
3.
Zhang, Qin, Ai Zhang, Yanqin Wang, et al.. (2023). Feasibility of the Omaha system for the care of children with dilated cardiomyopathy. Frontiers in Pediatrics. 11. 1136663–1136663. 1 indexed citations
4.
5.
Pan, Bo, et al.. (2022). Pediatric Diastolic Heart Failure: Clinical Features Description of 421 Cases. Frontiers in Pediatrics. 10. 846408–846408. 5 indexed citations
6.
Liu, Guolin, Xin Xu, Qijian Yi, & Tiewei Lv. (2021). The efficacy of catheter ablation versus ICD for prevention of ventricular tachycardia in patients with ischemic heart disease: a systematic review and meta-analysis. Journal of Interventional Cardiac Electrophysiology. 61(3). 435–443. 2 indexed citations
7.
Lu, Qian, Bo Pan, Weian Zhao, et al.. (2021). Intranuclear cardiac troponin I plays a functional role in regulating Atp2a2 expression in cardiomyocytes. Genes & Diseases. 9(6). 1689–1700. 11 indexed citations
8.
9.
10.
Huang, Zhilin, et al.. (2020). A New Diagnostic Model to Distinguish Kawasaki Disease From Other Febrile Illnesses in Chongqing: A Retrospective Study on 10,367 Patients. Frontiers in Pediatrics. 8. 533759–533759. 6 indexed citations
11.
Zhao, Jianquan, Tiewei Lv, Weian Zhao, et al.. (2018). Identification of target genes in cardiomyopathy with fibrosis and cardiac remodeling. Journal of Biomedical Science. 25(1). 63–63. 50 indexed citations
12.
Pan, Bo, Tiewei Lv, Lingjuan Liu, et al.. (2017). Inhibition of histone acetylation by curcumin reduces alcohol-induced fetal cardiac apoptosis. Journal of Biomedical Science. 24(1). 1–1. 63 indexed citations
13.
Shen, Xing, Bo Pan, Huiming Zhou, et al.. (2017). Differentiation of mesenchymal stem cells into cardiomyocytes is regulated by miRNA-1-2 via WNT signaling pathway. Journal of Biomedical Science. 24(1). 29–29. 59 indexed citations
14.
Zhang, Yuting, Ling He, Jinhua Cai, et al.. (2015). Measurements in Pediatric Patients with Cardiomyopathies: Comparison of Cardiac Magnetic Resonance Imaging and Echocardiography. Cardiology. 131(4). 245–250. 10 indexed citations
15.
Zhang, Weihua, Min Zheng, Jing Zhu, et al.. (2014). Prenatal alcohol exposure causes the over-expression of DHAND and EHAND by increasing histone H3K14 acetylation in C57 BL/6 mice. Toxicology Letters. 228(3). 140–146. 20 indexed citations
16.
Zheng, Min, Jing Zhu, Tiewei Lv, et al.. (2013). Bone morphogenetic protein-2 enhances the expression of cardiac transcription factors by increasing histone H3 acetylation in H9c2 cells. Molecular Medicine Reports. 7(3). 953–958. 7 indexed citations
17.
Pan, Bo, Huichao Sun, Tiewei Lv, et al.. (2013). Islet-1 May Function as an Assistant Factor for Histone Acetylation and Regulation of Cardiac Development-Related Transcription Factor Mef2c Expression. PLoS ONE. 8(10). e77690–e77690. 9 indexed citations
18.
Zhu, Jing, Tiewei Lv, Guozhen Chen, et al.. (2010). Ethanol and Its Metabolites Induce Histone Lysine 9 Acetylation and an Alteration of the Expression of Heart Development-Related Genes in Cardiac Progenitor Cells. Cardiovascular Toxicology. 10(4). 268–274. 41 indexed citations
19.
Sun, Huichao, Jing Zhu, Tiewei Lv, et al.. (2010). Inhibition of p300-HAT results in a reduced histone acetylation and down-regulation of gene expression in cardiac myocytes. Life Sciences. 87(23-26). 707–714. 44 indexed citations
20.
Chen, Guozhen, Jing Zhu, Tiewei Lv, et al.. (2009). Spatiotemporal expression of histone acetyltransferases, p300 and CBP, in developing embryonic hearts. Journal of Biomedical Science. 16(1). 24–24. 25 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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