Thomas Wossning
Impact in
- Genetics top 2%
- Chronic Lymphocytic Leukemia Research
- Immunology top 5%
- T-cell and B-cell Immunology
- Immune Cell Function and Interaction
- Immunodeficiency and Autoimmune Disorders
- Immunotherapy and Immune Responses
Papers in
-
- T-cell and B-cell Immunology 5
- Immune Cell Function and Interaction 3
- Mast cells and histamine 2
- Co-authors
- Hassan JumaaMichael RethFabian KöhlerMichael BachRudolf ÜbelhartAlexandra FlemmingMaike GallwitzWolfgang W. Schamel
In The Last Decade
Thomas Wossning
13 papers receiving 1.1k citations
Hit Papers
Peers
Comparison fields: 5 of 61
- Genetics 447
- Immunology 677
- Pathology and Forensic Medicine 314
- Hematology 131
- Radiology, Nuclear Medicine and Imaging 197
Countries citing papers authored by Thomas Wossning
This map shows the geographic impact of Thomas Wossning's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas Wossning with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas Wossning more than expected).
Fields of papers citing papers by Thomas Wossning
This network shows the impact of papers produced by Thomas Wossning. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas Wossning. The network helps show where Thomas Wossning may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Thomas Wossning, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2017 | 39 | |
| 2 | 2015 | 4 | |
| 3 | 2015 | 92 | |
| 4 | Chronic lymphocytic leukaemia is driven by antigen-independent cell-autonomous signalling Hit paper breakdown → | 2012 | 379 |
| 5 | 2012 | 48 | |
| 6 | 2010 | 70 | |
| 7 | 2007 | 67 | |
| 8 | 2006 | 63 | |
| 9 | 2006 | 1 | |
| 10 | 2005 | 1 | |
| 11 | 2004 | 21 | |
| 12 | 2004 | 0 | |
| 13 | 2003 | 41 | |
| 14 | 2002 | 252 |
About Thomas Wossning
Thomas Wossning is a scholar working on Immunology, Toxicology, Radiology, Nuclear Medicine and Imaging, Genetics and Hematology, having authored 14 papers that have together received 1.1k indexed citations. Recurring topics across this work include Monoclonal and Polyclonal Antibodies Research (6 papers), T-cell and B-cell Immunology (5 papers), Immune Cell Function and Interaction (3 papers), Mast cells and histamine (2 papers), Glycosylation and Glycoproteins Research (2 papers), Chronic Lymphocytic Leukemia Research (2 papers), CAR-T cell therapy research (2 papers) and Lymphoma Diagnosis and Treatment (2 papers). The work is most often cited by research in Genetics (447 citations), Immunology (677 citations), Pathology and Forensic Medicine (314 citations), Hematology (131 citations) and Radiology, Nuclear Medicine and Imaging (197 citations). Thomas Wossning has collaborated with scholars based in Germany, Japan and Australia. Frequent co-authors include Hassan Jumaa, Michael Reth, Fabian Köhler, Michael Bach, Rudolf Übelhart, Alexandra Flemming, Maike Gallwitz, Wolfgang W. Schamel, Véronique Rolli and Marcus Dühren-von Minden. Their work appears in journals such as The Journal of Experimental Medicine, Nature Immunology, Blood, The Journal of Cell Biology and Immunity.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.