Thomas Weide

8.8k citations

56 papers · 2.1k indexed · h-index 25

Impact in

Nephrology top 1%
- Renal Diseases and Glomerulopathies
- Chronic Kidney Disease and Diabetes
Cell Biology top 1%
- Cellular transport and secretion
- Hippo pathway signaling and YAP/TAZ
- Microtubule and mitosis dynamics

Papers in ⓘ

Nephrology 23
- Renal Diseases and Glomerulopathies 22
- Chronic Kidney Disease and Diabetes 8
Cell Biology 24
- Hippo pathway signaling and YAP/TAZ 14
- Cellular transport and secretion 10

Co-authors: Angelika Barnekow (6 shared papers)Hermann Pavenstädt (16 shared papers)Hermann Pavenstädt (20 shared papers)Michael Bayer (4 shared papers)Beate Vollenbröker (10 shared papers)Joachim Kremerskothen (10 shared papers)Tobias B. Huber (3 shared papers)Ulf Schulze (10 shared papers)
Journals: Journal of the American Society of Nephrology (6 papers)The FASEB Journal (4 papers)American Journal of Physiology-Renal Physiology (4 papers)BMC Biotechnology (3 papers)Kidney International (3 papers)
Partner nations: Germany France United States

In The Last Decade

Thomas Weide

55 papers receiving 2.1k citations

Peers

Countries citing papers authored by Thomas Weide

Since Specialization

Citations

This map shows the geographic impact of Thomas Weide's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas Weide with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas Weide more than expected).

Fields of papers citing papers by Thomas Weide

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas Weide. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas Weide. The network helps show where Thomas Weide may publish in the future.

Co-authors

The 25 scholars most cited alongside Thomas Weide, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Thomas Weide Line = papers co-authored together Thomas Weide links everyone, so they are left out of the graph.

All Works

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20 of 20 papers shown

Showing the 20 most-cited of 56 papers — load more, or switch the sort, to bring in the rest.

#	Work
1	Bicaudal-D regulates COPI-independent Golgi–ER transport by recruiting the dynein–dynactin motor complex Nature Cell Biology ·Theodoros Matanis, Anna Akhmanova, Phebe S. Wulf, Elaine Del Nery, Thomas Weide, Т. Ф. Степанова, Niels Galjart, Frank Grosveld, Bruno Goud, Chris I. De Zeeuw, Angelika Barnekow, Casper C. Hoogenraad	2002	314
2	In vivo, Pikfyve generates PI(3,5)P ₂ , which serves as both a signaling lipid and the major precursor for PI5P Proceedings of the National Academy of Sciences ·Sergey N. Zolov, Dave Bridges, Yanling Zhang, Wei-Wei Lee, Ellen Riehle, Rakesh Kumar Verma, Guy M. Lenk, Kimber Converso‐Baran, Thomas Weide, Roger L. Albin, Alan R. Saltiel, Miriam H. Meisler, Mark W. Russell, Lois S. Weisman	2012	172
3	Mechanisms of angiotensin II signaling on cytoskeleton of podocytes Journal of Molecular Medicine ·Hsiang‐Hao Hsu, Sigrid Hoffmann, Nicole Endlich, Ana Velić, Albrecht Schwab, Thomas Weide, Eberhard Schlatter, Hermann Pavenstädt	2008	129
4	The Amot/Patj/Syx signaling complex spatially controls RhoA GTPase activity in migrating endothelial cells Blood ·Mira Ernkvist, Nathalie Luna Persson, Stéphane Audebert, Patrick Lécine, Indranil Sinha, Miaoliang Liu, Marc Schlueter, Arie Horowitz, Karin Aase, Thomas Weide, Jean‐Paul Borg, Årindam Majumdar, Lars Holmgren	2008	116
5	The Golgi matrix protein GM130: a specific interacting partner of the small GTPase rab1b EMBO Reports ·Thomas Weide, Michael Bayer, M. Köster, Jan Peter Siebrasse, Reiner Peters, Angelika Barnekow	2001	109
6	KIBRA Modulates Directional Migration of Podocytes Journal of the American Society of Nephrology ·Kerstin Duning, Eva-Maria Schurek, Marc SchluCombining Diaeresister, Michael Bayer, Hans-Christian Reinhardt, Albrecht Schwab, Liliana Schaefer, Thomas Benzing, Bernhard Schermer, Moin A. Saleem, Tobias B. Huber, Sebastian Bachmann, Joachim Kremerskothen, Thomas Weide, Hermann PavenstaCombining Diaeresisdt	2008	100
7	mTOR regulates expression of slit diaphragm proteins and cytoskeleton structure in podocytes American Journal of Physiology-Renal Physiology ·Beate Vollenbröker, Britta George, Maria Wolfgart, Moin A. Saleem, Hermann Pavenstädt, Thomas Weide	2008	95
8	The Hippo pathway is controlled by Angiotensin II signaling and its reactivation induces apoptosis in podocytes Cell Death and Disease ·Dirk Oliver Wennmann, Beate Vollenbröker, A K Eckart, Jakob Bonse, Frank Erdmann, Dirk Wolters, Laura K. Schenk, Ulf Schulze, Joachim Kremerskothen, Thomas Weide, Hermann Pavenstädt	2014	82
9	Intracellular APOL1 Risk Variants Cause Cytotoxicity Accompanied by Energy Depletion Journal of the American Society of Nephrology ·Daniel Granado, Daria Müller, Vanessa Krausel, Etty Kruzel-Davila, Christian Schuberth, Melanie Eschborn, Roland Wedlich‐Söldner, Karl Skorecki, Hermann Pavenstädt, Ulf Michgehl, Thomas Weide	2017	78
10	The MICAL proteins and rab1: a possible link to the cytoskeleton? Biochemical and Biophysical Research Communications ·Julia Fischer, Thomas Weide, Angelika Barnekow	2005	60
11	Evolutionary and Molecular Facts Link the WWC Protein Family to Hippo Signaling Molecular Biology and Evolution ·Dirk Oliver Wennmann, Jürgen Schmitz, Michael C. Wehr, Michael P. Krahn, Nora Koschmal, Sascha Gromnitza, Ulf Schulze, Thomas Weide, Anil Chekuri, Boris V. Skryabin, Volker Gerke, Hermann Pavenstädt, Kerstin Duning, Joachim Kremerskothen	2014	55
12	MICAL-1 isoforms, novel rab1 interacting proteins Biochemical and Biophysical Research Communications ·Thomas Weide, Julia Teuber, Michael Bayer, Angelika Barnekow	2003	54
13	KIBRA (KIdney/BRAin protein) regulates learning and memory and stabilizes Protein kinase Mζ Journal of Neurochemistry ·Angela K. Vogt-Eisele, Carola Krüger, Kerstin Duning, Daniela Weber, Robert Spoelgen, Claudia Pitzer, Christian Plaas, Gisela Eisenhardt, Annette Meyer, Gerhard Vogt, Markus Krieger, Eva Handwerker, Dirk Oliver Wennmann, Thomas Weide, Boris V. Skryabin, Matthias Klugmann, Hermann Pavenstädt, Matthew J. Huentelmann, Joachim Kremerskothen, Armin Schneider	2013	53
14	AmotL2 disrupts apical–basal cell polarity and promotes tumour invasion Nature Communications ·Mahdi Mojallal, Yujuan Zheng, Sara Hultin, Stéphane Audebert, Tanja van Harn, Per Johnsson, Claes Lenander, Nicolas Fritz, Christin Mieth, Martin Corcoran, Frédérique Lembo, Marja Hallström, Johan Hartman, Nathalie M. Mazure, Thomas Weide, Dan Grandér, Jean‐Paul Borg, Per Uhlén, Lars Holmgren	2014	51
15	The Vac14-interaction Network Is Linked to Regulators of the Endolysosomal and Autophagic Pathway Molecular & Cellular Proteomics ·Ulf Schulze, Beate Vollenbröker, Daniela A. Braun, Truc Le, Daniel Granado, Joachim Kremerskothen, Benjamin Fränzel, Rafael Klosowski, Johannes Barth, Christian Fufezan, Dirk Wolters, Hermann Pavenstädt, Thomas Weide	2014	46
16	Implications of autophagy for glomerular aging and disease Cell and Tissue Research ·Thomas Weide, Tobias B. Huber	2011	41
17	WW and C2 domain–containing proteins regulate hepatic cell differentiation and tumorigenesis through the hippo signaling pathway Hepatology ·Anke Hermann, Dirk Oliver Wennmann, Sascha Gromnitza, Maria Edeling, Veerle Van Marck, Marius Sudol, Liliana Schaefer, Kerstin Duning, Thomas Weide, Hermann Pavenstädt, Joachim Kremerskothen	2017	36
18	Downregulation of the antioxidant protein peroxiredoxin 2 contributes to angiotensin II–mediated podocyte apoptosis Kidney International ·Hsiang‐Hao Hsu, Sigrid Hoffmann, Giovana Seno Di Marco, Nicole Endlich, Jasna Peter‐Katalinić, Thomas Weide, Hermann Pavenstädt	2011	36
19	Nuclear YAP localization as a key regulator of podocyte function Cell Death and Disease ·Jakob Bonse, Dirk Oliver Wennmann, Joachim Kremerskothen, Thomas Weide, Ulf Michgehl, Hermann Pavenstädt, Beate Vollenbröker	2018	35
20	Distinct functions of Crumbs regulating slit diaphragms and endocytosis in Drosophila nephrocytes Cellular and Molecular Life Sciences ·Florian Hochapfel, Lucia Denk, Gudrun Mendl, Ulf Schulze, Christine Maaßen, Yulia Zaytseva, Hermann Pavenstädt, Thomas Weide, Reinhard Rachel, Ralph Witzgall, Michael P. Krahn	2017	32

About Thomas Weide

Thomas Weide is a scholar working on Nephrology, Cell Biology, Physiology, Genetics and Molecular Biology, having authored 56 papers that have together received 2.1k indexed citations. Recurring topics across this work include Renal Diseases and Glomerulopathies (22 papers), Hippo pathway signaling and YAP/TAZ (14 papers), Renal and related cancers (11 papers), Cellular transport and secretion (10 papers), Genetic and Kidney Cyst Diseases (9 papers), Chronic Kidney Disease and Diabetes (8 papers), Calcium signaling and nucleotide metabolism (6 papers) and Ion Transport and Channel Regulation (4 papers). The work is most often cited by research in Nephrology (511 citations), Cell Biology (1.0k citations), Physiology (160 citations), Molecular Biology (1.1k citations) and Genetics (116 citations). Thomas Weide has collaborated with scholars based in Germany, France and United States. Frequent co-authors include Angelika Barnekow, Hermann Pavenstädt, Hermann Pavenstädt, Michael Bayer, Beate Vollenbröker, Joachim Kremerskothen, Tobias B. Huber, Ulf Schulze, Casper C. Hoogenraad and Phebe S. Wulf. Their work appears in journals such as Journal of the American Society of Nephrology, The FASEB Journal, American Journal of Physiology-Renal Physiology, BMC Biotechnology and Kidney International.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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