Thomas Weide

8.8k total citations
56 papers, 2.1k citations indexed

About

Thomas Weide is a scholar working on Molecular Biology, Cell Biology and Nephrology. According to data from OpenAlex, Thomas Weide has authored 56 papers receiving a total of 2.1k indexed citations (citations by other indexed papers that have themselves been cited), including 32 papers in Molecular Biology, 24 papers in Cell Biology and 23 papers in Nephrology. Recurrent topics in Thomas Weide's work include Renal Diseases and Glomerulopathies (22 papers), Hippo pathway signaling and YAP/TAZ (14 papers) and Renal and related cancers (11 papers). Thomas Weide is often cited by papers focused on Renal Diseases and Glomerulopathies (22 papers), Hippo pathway signaling and YAP/TAZ (14 papers) and Renal and related cancers (11 papers). Thomas Weide collaborates with scholars based in Germany, France and United States. Thomas Weide's co-authors include Angelika Barnekow, Hermann Pavenstädt, Hermann Pavenstädt, Michael Bayer, Beate Vollenbröker, Joachim Kremerskothen, Tobias B. Huber, Ulf Schulze, Elaine Del Nery and Т. Ф. Степанова and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Nature Communications.

In The Last Decade

Thomas Weide

55 papers receiving 2.1k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Thomas Weide Germany 25 1.1k 1.0k 511 232 186 56 2.1k
Yasuaki Shirayoshi Japan 26 2.6k 2.3× 493 0.5× 106 0.2× 659 2.8× 167 0.9× 87 3.6k
Timothy A. Fields United States 25 1.6k 1.4× 318 0.3× 272 0.5× 434 1.9× 120 0.6× 40 2.2k
Jan Domin United Kingdom 29 1.6k 1.4× 904 0.9× 62 0.1× 116 0.5× 250 1.3× 43 2.6k
Sharon F. Suchy United States 17 946 0.8× 797 0.8× 61 0.1× 270 1.2× 222 1.2× 31 1.5k
Diana Escalante‐Alcalde Mexico 21 2.3k 2.0× 454 0.4× 66 0.1× 263 1.1× 236 1.3× 38 2.8k
Bernd Kinzel Switzerland 23 1.4k 1.2× 295 0.3× 99 0.2× 183 0.8× 207 1.1× 30 2.1k
Taroh Iiri Japan 30 2.1k 1.8× 354 0.3× 80 0.2× 335 1.4× 269 1.4× 72 3.2k
Gwenn M. Hansen United States 25 1.0k 0.9× 247 0.2× 55 0.1× 337 1.5× 165 0.9× 48 1.7k
Hagar Kalinski Israel 12 1.1k 0.9× 229 0.2× 79 0.2× 117 0.5× 108 0.6× 15 1.6k
Susumu Sekine Japan 18 844 0.7× 100 0.1× 349 0.7× 526 2.3× 95 0.5× 31 1.6k

Countries citing papers authored by Thomas Weide

Since Specialization
Citations

This map shows the geographic impact of Thomas Weide's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas Weide with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas Weide more than expected).

Fields of papers citing papers by Thomas Weide

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas Weide. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas Weide. The network helps show where Thomas Weide may publish in the future.

Co-authorship network of co-authors of Thomas Weide

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas Weide. A scholar is included among the top collaborators of Thomas Weide based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas Weide. Thomas Weide is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Nedvetsky, Pavel I., Uwe Hansen, Michael P. Krahn, et al.. (2024). PALS1 is a key regulator of the lateral distribution of tight junction proteins in renal epithelial cells. Journal of Cell Science. 137(5). 2 indexed citations
2.
Grampp, Steffen, et al.. (2023). Hypoxia hits APOL1 in the kidney. Kidney International. 104(1). 53–60. 11 indexed citations
3.
George, Britta, et al.. (2022). Loss of surface transport is a main cellular pathomechanism of CRB2 variants causing podocytopathies. Life Science Alliance. 6(3). e202201649–e202201649. 4 indexed citations
4.
Breljak, Davorka, et al.. (2022). Impact of Pals1 on Expression and Localization of Transporters Belonging to the Solute Carrier Family. Frontiers in Molecular Biosciences. 9. 792829–792829. 2 indexed citations
5.
Jehn, U., Veerle Van Marck, Thomas Weide, et al.. (2021). α-Galactosidase a Deficiency in Fabry Disease Leads to Extensive Dysregulated Cellular Signaling Pathways in Human Podocytes. International Journal of Molecular Sciences. 22(21). 11339–11339. 19 indexed citations
6.
Schmitz, Jürgen, et al.. (2021). Evolution of Renal-Disease Factor APOL1 Results in Cis and Trans Orientations at the Endoplasmic Reticulum That Both Show Cytotoxic Effects. Molecular Biology and Evolution. 38(11). 4962–4976. 14 indexed citations
7.
Wennmann, Dirk Oliver, et al.. (2018). Nuclear YAP localization as a key regulator of podocyte function. Cell Death and Disease. 9(9). 850–850. 35 indexed citations
8.
Wennmann, Dirk Oliver, Veerle Van Marck, Marius Sudol, et al.. (2017). WW and C2 domain–containing proteins regulate hepatic cell differentiation and tumorigenesis through the hippo signaling pathway. Hepatology. 67(4). 1546–1559. 36 indexed citations
9.
Siebrasse, Jan Peter, Ulf Schulze, Marc A. Schlüter, et al.. (2016). The C-terminal domain controls the mobility of Crumbs 3 isoforms. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1863(6). 1208–1217. 13 indexed citations
10.
Siebrasse, Jan Peter, Ulf Schulze, Marc A. Schlüter, et al.. (2016). Trajectories and single-particle tracking data of intracellular vesicles loaded with either SNAP-Crb3A or SNAP-Crb3B. Data in Brief. 7. 1665–1669. 7 indexed citations
11.
Wennmann, Dirk Oliver, Beate Vollenbröker, Frank Erdmann, et al.. (2014). The Hippo pathway is controlled by Angiotensin II signaling and its reactivation induces apoptosis in podocytes. Cell Death and Disease. 5(11). e1519–e1519. 82 indexed citations
12.
Schulze, Ulf, Beate Vollenbröker, Daniela A. Braun, et al.. (2014). The Vac14-interaction Network Is Linked to Regulators of the Endolysosomal and Autophagic Pathway. Molecular & Cellular Proteomics. 13(6). 1397–1411. 46 indexed citations
13.
Wennmann, Dirk Oliver, Jürgen Schmitz, Michael C. Wehr, et al.. (2014). Evolutionary and Molecular Facts Link the WWC Protein Family to Hippo Signaling. Molecular Biology and Evolution. 31(7). 1710–1723. 55 indexed citations
14.
Duning, Kerstin, Marc A. Schlüter, Yuemin Tian, et al.. (2010). Polycystin-2 Activity Is Controlled by Transcriptional Coactivator with PDZ Binding Motif and PALS1-associated Tight Junction Protein. Journal of Biological Chemistry. 285(44). 33584–33588. 17 indexed citations
15.
Duning, Kerstin, Thomas Weide, Truc Le, et al.. (2009). Hypertension in mice lacking the CXCR3 chemokine receptor. American Journal of Physiology-Renal Physiology. 296(4). F780–F789. 11 indexed citations
16.
Duning, Kerstin, Michael Bayer, Albrecht Schwab, et al.. (2008). KIBRA Modulates Directional Migration of Podocytes. Journal of the American Society of Nephrology. 19(10). 1891–1903. 100 indexed citations
17.
18.
Bayer, Michael, Julia Fischer, Joachim Kremerskothen, et al.. (2005). Identification and characterization of Iporin as a novel interaction partner for rab1. BMC Cell Biology. 6(1). 15–15. 25 indexed citations
19.
Fischer, Julia, Thomas Weide, & Angelika Barnekow. (2005). The MICAL proteins and rab1: a possible link to the cytoskeleton?. Biochemical and Biophysical Research Communications. 328(2). 415–423. 60 indexed citations
20.
Akhmanova, Anna, Phebe S. Wulf, Elaine Del Nery, et al.. (2002). Bicaudal-D regulates COPI-independent Golgi–ER transport by recruiting the dynein–dynactin motor complex. Nature Cell Biology. 4(12). 986–992. 314 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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