Thomas M. Piers

2.3k total citations
34 papers, 1.5k citations indexed

About

Thomas M. Piers is a scholar working on Neurology, Cellular and Molecular Neuroscience and Immunology. According to data from OpenAlex, Thomas M. Piers has authored 34 papers receiving a total of 1.5k indexed citations (citations by other indexed papers that have themselves been cited), including 23 papers in Neurology, 15 papers in Cellular and Molecular Neuroscience and 12 papers in Immunology. Recurrent topics in Thomas M. Piers's work include Neuroinflammation and Neurodegeneration Mechanisms (23 papers), Neuroscience and Neuropharmacology Research (13 papers) and Inflammation biomarkers and pathways (10 papers). Thomas M. Piers is often cited by papers focused on Neuroinflammation and Neurodegeneration Mechanisms (23 papers), Neuroscience and Neuropharmacology Research (13 papers) and Inflammation biomarkers and pathways (10 papers). Thomas M. Piers collaborates with scholars based in United Kingdom, United States and South Korea. Thomas M. Piers's co-authors include Jennifer M. Pocock, Daniel J. Whitcomb, Kwangwook Cho, Philip Regan, John Hardy, Anna Mallach, Jihoon Jo, Jee Hyun Yi, Eunjoon Kim and Graham L. Collingridge and has published in prestigious journals such as Journal of Neuroscience, Nature reviews. Neuroscience and Brain.

In The Last Decade

Thomas M. Piers

34 papers receiving 1.5k citations

Peers

Thomas M. Piers
Juan Piña-Crespo United States
Raquel Sánchez‐Varo Spain
Mariana Vargas‐Caballero United Kingdom
Harun N. Noristani France
Christian Madry Germany
Sneha Narasimhan United States
Claudia Prada United States
Shu-Ming Huang China
Jorge Montesinos Spain
Hélène Hirbec France
Juan Piña-Crespo United States
Thomas M. Piers
Citations per year, relative to Thomas M. Piers Thomas M. Piers (= 1×) peers Juan Piña-Crespo

Countries citing papers authored by Thomas M. Piers

Since Specialization
Citations

This map shows the geographic impact of Thomas M. Piers's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas M. Piers with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas M. Piers more than expected).

Fields of papers citing papers by Thomas M. Piers

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas M. Piers. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas M. Piers. The network helps show where Thomas M. Piers may publish in the future.

Co-authorship network of co-authors of Thomas M. Piers

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas M. Piers. A scholar is included among the top collaborators of Thomas M. Piers based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas M. Piers. Thomas M. Piers is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Pishva, Ehsan, et al.. (2025). Amyloid-β can activate JNK signalling via WNT5A-ROR2 to reduce synapse formation in Alzheimer's disease. Journal of Cell Science. 138(3). 2 indexed citations
2.
Piers, Thomas M., Seema C. Namboori, Corin Liddle, et al.. (2024). WNT7A-positive dendritic cytonemes control synaptogenesis in cortical neurons. Development. 151(23). 2 indexed citations
3.
Piers, Thomas M., et al.. (2024). Amelioration of signaling deficits underlying metabolic shortfall in TREM2R47H human iPSC‐derived microglia. FEBS Journal. 292(7). 1743–1762. 2 indexed citations
4.
Arber, Charles, Jackie M. Casey, Sarah Wiethoff, et al.. (2024). Microglia contribute to the production of the amyloidogenic ABri peptide in familial British dementia. Acta Neuropathologica. 148(1). 65–65. 3 indexed citations
5.
Graham, Andrew, Thomas M. Piers, Maryam Shoai, et al.. (2021). A genetic link between risk for Alzheimer's disease and severe COVID-19 outcomes via the OAS1 gene. Brain. 144(12). 3727–3741. 75 indexed citations
6.
Cosker, Katharina, Anna Mallach, Thomas M. Piers, et al.. (2021). Microglial signalling pathway deficits associated with the patient derived R47H TREM2 variants linked to AD indicate inability to activate inflammasome. Scientific Reports. 11(1). 13316–13316. 32 indexed citations
7.
Liu, Wenfei, Rui Wang, Sevinç Bayram, et al.. (2020). Trem2 promotes anti-inflammatory responses in microglia and is suppressed under pro-inflammatory conditions. Human Molecular Genetics. 29(19). 3224–3248. 122 indexed citations
8.
Piers, Thomas M., Emma East, Claudio Villegas-Llerena, et al.. (2018). Soluble Fibrinogen Triggers Non-cell Autonomous ER Stress-Mediated Microglial-Induced Neurotoxicity. Frontiers in Cellular Neuroscience. 12. 404–404. 14 indexed citations
9.
Pocock, Jennifer M. & Thomas M. Piers. (2018). Modelling microglial function with induced pluripotent stem cells: an update. Nature reviews. Neuroscience. 19(8). 445–452. 40 indexed citations
10.
Garcia-Reitboeck, Pablo, Alexandra Phillips, Thomas M. Piers, et al.. (2018). Human Induced Pluripotent Stem Cell-Derived Microglia-Like Cells Harboring TREM2 Missense Mutations Show Specific Deficits in Phagocytosis. Cell Reports. 24(9). 2300–2311. 116 indexed citations
11.
Yi, Jee Hyun, Dong Hyun Kim, Thomas M. Piers, et al.. (2018). Postsynaptic p47phox regulates long-term depression in the hippocampus. Cell Discovery. 4(1). 44–44. 12 indexed citations
12.
Piers, Thomas M., et al.. (2015). Amyloid-β oligomers unveil a novel primate model of sporadic Alzheimer's disease. Frontiers in Neuroscience. 9. 47–47. 6 indexed citations
13.
Piers, Thomas M., et al.. (2015). Selective Depletion of Microglia from Cerebellar Granule Cell Cultures Using L-leucine Methyl Ester. Journal of Visualized Experiments. e52983–e52983. 10 indexed citations
14.
Piers, Thomas M., Jee Hyun Yi, Seong‐Min Choi, et al.. (2015). Activation of a synapse weakening pathway by human Val66 but not Met66 pro-brain-derived neurotrophic factor (proBDNF). Pharmacological Research. 104. 97–107. 29 indexed citations
15.
Regan, Philip, Thomas M. Piers, Jee Hyun Yi, et al.. (2015). Tau Phosphorylation at Serine 396 Residue Is Required for Hippocampal LTD. Journal of Neuroscience. 35(12). 4804–4812. 155 indexed citations
16.
Whitehead, Garry, Jihoon Jo, Thomas M. Piers, et al.. (2013). Acute stress causes rapid synaptic insertion of Ca2+-permeable AMPA receptors to facilitate long-term potentiation in the hippocampus. Brain. 136(12). 3753–3765. 77 indexed citations
17.
Bechtold, David A., Woojin Lee, Daniel M. Snell, et al.. (2013). Safinamide and flecainide protect axons and reduce microglial activation in models of multiple sclerosis. Brain. 136(4). 1067–1082. 65 indexed citations
18.
Piers, Thomas M., Dong Hyun Kim, Byeong C. Kim, et al.. (2012). Translational Concepts of mGluR5 in Synaptic Diseases of the Brain. Frontiers in Pharmacology. 3. 199–199. 63 indexed citations
19.
Piers, Thomas M., Emma East, & Jennifer M. Pocock. (2009). FIBRIN AND FIBRINOGEN CAUSE NEURON NON-CELL AUTONOMOUS DEGENERATION. Open Research Online (The Open University). 1 indexed citations
20.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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