Christian Madry

2.1k total citations · 1 hit paper
27 papers, 1.5k citations indexed

About

Christian Madry is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Neurology. According to data from OpenAlex, Christian Madry has authored 27 papers receiving a total of 1.5k indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Molecular Biology, 15 papers in Cellular and Molecular Neuroscience and 13 papers in Neurology. Recurrent topics in Christian Madry's work include Neuroscience and Neuropharmacology Research (15 papers), Neuroinflammation and Neurodegeneration Mechanisms (13 papers) and Ion channel regulation and function (7 papers). Christian Madry is often cited by papers focused on Neuroscience and Neuropharmacology Research (15 papers), Neuroinflammation and Neurodegeneration Mechanisms (13 papers) and Ion channel regulation and function (7 papers). Christian Madry collaborates with scholars based in Germany, United Kingdom and United States. Christian Madry's co-authors include David Attwell, Vasiliki Kyrargyri, I. Lorena Arancibia‐Cárcamo, Pablo Izquierdo, Bodo Laube, Renaud Jolivet, Robert M. Bryan, Shinichi Kohsaka, Ivana Mesic and Heinrich Betz and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Christian Madry

26 papers receiving 1.5k citations

Hit Papers

Amyloid β oligomers constrict human capillaries in Alzhei... 2019 2026 2021 2023 2019 100 200 300 400

Peers

Christian Madry
Christian Madry
Citations per year, relative to Christian Madry Christian Madry (= 1×) peers Hélène Hirbec

Countries citing papers authored by Christian Madry

Since Specialization
Citations

This map shows the geographic impact of Christian Madry's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Christian Madry with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Christian Madry more than expected).

Fields of papers citing papers by Christian Madry

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Christian Madry. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Christian Madry. The network helps show where Christian Madry may publish in the future.

Co-authorship network of co-authors of Christian Madry

This figure shows the co-authorship network connecting the top 25 collaborators of Christian Madry. A scholar is included among the top collaborators of Christian Madry based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Christian Madry. Christian Madry is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Safaiyan, Shima, et al.. (2026). The Arp2/3 complex controls the development of homeostatic microglia. EMBO Reports. 1 indexed citations
2.
Rödström, Karin E. J., Peter Proks, Sönke Cordeiro, et al.. (2025). Cryo-EM structure of the human THIK-1 K2P K+ channel reveals a lower Y gate regulated by lipids and anesthetics. Nature Structural & Molecular Biology. 32(7). 1167–1174. 3 indexed citations
3.
Kreis, Patricia, Thorsten Trimbuch, Julia Onken, et al.. (2025). The chloride intracellular channel 1 (CLIC1) is essential for microglial morphodynamics and neuroinflammation. Science Advances. 11(43). eads9181–eads9181.
4.
Ossola, Bernardino, Roland W. Bürli, Lee A. Dawson, et al.. (2024). Differential contribution of THIK-1 K+ channels and P2X7 receptors to ATP-mediated neuroinflammation by human microglia. Journal of Neuroinflammation. 21(1). 58–58. 14 indexed citations
5.
Munro, David A. D., et al.. (2024). Lifelong absence of microglia alters hippocampal glutamatergic networks but not synapse and spine density. EMBO Reports. 25(5). 2348–2374. 14 indexed citations
6.
Izquierdo, Pablo, Renaud Jolivet, David Attwell, & Christian Madry. (2023). Amyloid plaques and normal ageing have differential effects on microglial Ca2+ activity in the mouse brain. Pflügers Archiv - European Journal of Physiology. 476(2). 257–270. 3 indexed citations
7.
Nortley, Ross, Nils Korte, Pablo Izquierdo, et al.. (2019). Amyloid β oligomers constrict human capillaries in Alzheimer’s disease via signaling to pericytes. Science. 365(6450). 475 indexed citations breakdown →
8.
Izquierdo, Pablo, David Attwell, & Christian Madry. (2019). Ion Channels and Receptors as Determinants of Microglial Function. Trends in Neurosciences. 42(4). 278–292. 72 indexed citations
9.
Kyrargyri, Vasiliki, David Attwell, Renaud Jolivet, & Christian Madry. (2019). Analysis of Signaling Mechanisms Regulating Microglial Process Movement. Methods in molecular biology. 2034. 191–205. 6 indexed citations
10.
Madry, Christian, Vasiliki Kyrargyri, I. Lorena Arancibia‐Cárcamo, et al.. (2017). Microglial Ramification, Surveillance, and Interleukin-1β Release Are Regulated by the Two-Pore Domain K+ Channel THIK-1. Neuron. 97(2). 299–312.e6. 318 indexed citations
11.
Mesic, Ivana, et al.. (2017). Positive Modulatory Interactions of NMDA Receptor GluN1/2B Ligand Binding Domains Attenuate Antagonists Activity. Frontiers in Pharmacology. 8. 229–229. 7 indexed citations
12.
Mesic, Ivana, et al.. (2016). The N-terminal domain of the GluN3A subunit determines the efficacy of glycine-activated NMDA receptors. Neuropharmacology. 105. 133–141. 14 indexed citations
13.
Madry, Christian & David Attwell. (2015). Receptors, Ion Channels, and Signaling Mechanisms Underlying Microglial Dynamics. Journal of Biological Chemistry. 290(20). 12443–12450. 72 indexed citations
14.
Madry, Christian, et al.. (2010). The role of pannexin hemichannels in the anoxic depolarization of hippocampal pyramidal cells. Brain. 133(12). 3755–3763. 48 indexed citations
15.
Hwang, Ji-Yeon, R. Scott Duncan, Christian Madry, Meharvan Singh, & Peter Koulen. (2008). Progesterone potentiates calcium release through IP3 receptors by an Akt-mediated mechanism in hippocampal neurons. Cell Calcium. 45(3). 233–242. 24 indexed citations
16.
Koulen, Peter, et al.. (2008). Progesterone Potentiates IP<sub>3</sub>-Mediated Calcium Signaling Through Akt/PKB. Cellular Physiology and Biochemistry. 21(1-3). 161–172. 35 indexed citations
17.
Madry, Christian, Ivana Mesic, Heinrich Betz, & Bodo Laube. (2007). The N-Terminal Domains of both NR1 and NR2 Subunits Determine Allosteric Zn2+ Inhibition and Glycine Affinity of N-Methyl-d-aspartate Receptors. Molecular Pharmacology. 72(6). 1535–1544. 46 indexed citations
18.
Schüler, Thomas, Ivana Mesic, Christian Madry, Ingo Bartholomäus, & Bodo Laube. (2007). Formation of NR1/NR2 and NR1/NR3 Heterodimers Constitutes the Initial Step in N-Methyl-D-aspartate Receptor Assembly. Journal of Biological Chemistry. 283(1). 37–46. 76 indexed citations
19.
Madry, Christian, Ivana Mesic, Ingo Bartholomäus, et al.. (2006). Principal role of NR3 subunits in NR1/NR3 excitatory glycine receptor function. Biochemical and Biophysical Research Communications. 354(1). 102–108. 56 indexed citations
20.
Koulen, Peter, et al.. (2004). Differentially Distributed IP3Receptors and Ca2+Signaling in Rod Bipolar Cells. Investigative Ophthalmology & Visual Science. 46(1). 292–292. 16 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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