Thomas M. Maynard

4.6k total citations
57 papers, 3.3k citations indexed

About

Thomas M. Maynard is a scholar working on Molecular Biology, Genetics and Surgery. According to data from OpenAlex, Thomas M. Maynard has authored 57 papers receiving a total of 3.3k indexed citations (citations by other indexed papers that have themselves been cited), including 48 papers in Molecular Biology, 16 papers in Genetics and 10 papers in Surgery. Recurrent topics in Thomas M. Maynard's work include Congenital heart defects research (31 papers), Developmental Biology and Gene Regulation (11 papers) and Congenital Heart Disease Studies (7 papers). Thomas M. Maynard is often cited by papers focused on Congenital heart defects research (31 papers), Developmental Biology and Gene Regulation (11 papers) and Congenital Heart Disease Studies (7 papers). Thomas M. Maynard collaborates with scholars based in United States, Denmark and United Kingdom. Thomas M. Maynard's co-authors include Anthony‐Samuel LaMantia, James A. Weston, Yoshio Wakamatsu, Daniel W. Meechan, Linmarie Sikich, Eric S. Tucker, J.A. Lieberman, Maria K. Lehtinen, Gloria Haskell and Sally A. Moody and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nature Communications and Neuron.

In The Last Decade

Thomas M. Maynard

53 papers receiving 3.2k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Thomas M. Maynard United States 30 2.1k 787 729 576 281 57 3.3k
Susan M. Dymecki United States 40 3.4k 1.6× 922 1.2× 1.7k 2.4× 595 1.0× 700 2.5× 69 6.2k
Gerhard Schratt Germany 38 4.9k 2.4× 824 1.0× 1.1k 1.5× 774 1.3× 288 1.0× 72 6.7k
Boris P. Sokolov United States 24 1.5k 0.7× 599 0.8× 666 0.9× 216 0.4× 249 0.9× 40 3.0k
Jean‐Michel Revest France 19 1.5k 0.7× 413 0.5× 730 1.0× 734 1.3× 317 1.1× 31 2.8k
Vivi M. Heine Netherlands 26 1.6k 0.8× 382 0.5× 683 0.9× 840 1.5× 267 1.0× 66 3.3k
Nicole A. Datson Netherlands 35 1.9k 0.9× 822 1.0× 596 0.8× 186 0.3× 138 0.5× 72 4.1k
Benjamin R. Arenkiel United States 35 1.5k 0.7× 344 0.4× 1.7k 2.3× 366 0.6× 942 3.4× 97 4.4k
Liya Shen United States 28 3.7k 1.8× 1.1k 1.5× 1.5k 2.1× 912 1.6× 209 0.7× 38 5.8k
Chengji J. Zhou United States 35 1.8k 0.9× 795 1.0× 894 1.2× 455 0.8× 120 0.4× 77 3.1k
K. Dengke United States 21 2.2k 1.0× 854 1.1× 744 1.0× 923 1.6× 226 0.8× 45 3.4k

Countries citing papers authored by Thomas M. Maynard

Since Specialization
Citations

This map shows the geographic impact of Thomas M. Maynard's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas M. Maynard with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas M. Maynard more than expected).

Fields of papers citing papers by Thomas M. Maynard

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas M. Maynard. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas M. Maynard. The network helps show where Thomas M. Maynard may publish in the future.

Co-authorship network of co-authors of Thomas M. Maynard

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas M. Maynard. A scholar is included among the top collaborators of Thomas M. Maynard based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas M. Maynard. Thomas M. Maynard is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Meechan, Daniel W., et al.. (2023). Out of Line or Altered States? Neural Progenitors as a Target in a Polygenic Neurodevelopmental Disorder. Developmental Neuroscience. 46(1). 1–21.
2.
Maynard, Thomas M., et al.. (2023). Identity, lineage and fates of a temporally distinct progenitor population in the embryonic olfactory epithelium. Developmental Biology. 495. 76–91.
3.
Muñoz‐Ballester, Carmen, Allison N. Tegge, Katherine L. Brown, et al.. (2021). Development and implementation of a scalable and versatile test for COVID-19 diagnostics in rural communities. Nature Communications. 12(1). 4400–4400. 13 indexed citations
4.
Karpinski, Beverly A., et al.. (2020). Variations in maternal vitamin A intake modifies phenotypes in a mouse model of 22q11.2 deletion syndrome. Birth Defects Research. 112(16). 1194–1208. 7 indexed citations
5.
Bunyak, Filiz, et al.. (2020). Persistent Feeding and Swallowing Deficits in a Mouse Model of 22q11.2 Deletion Syndrome. Frontiers in Neurology. 11. 4–4. 18 indexed citations
6.
Steullet, Pascal, Joseph T. Coyle, Michael Didriksen, et al.. (2017). Oxidative stress-driven parvalbumin interneuron impairment as a common mechanism in models of schizophrenia. Molecular Psychiatry. 22(7). 936–943. 273 indexed citations
7.
Maynard, Thomas M. & M. Chiara Manzini. (2017). Balancing Act: Maintaining Amino Acid Levels in the Autistic Brain. Neuron. 93(3). 476–479. 17 indexed citations
8.
Karpinski, Beverly A., Jennifer L. Baker, Anélia Horvath, et al.. (2016). A cellular and molecular mosaic establishes growth and differentiation states for cranial sensory neurons. Developmental Biology. 415(2). 228–241. 21 indexed citations
9.
Meechan, Daniel W., Thomas M. Maynard, Eric S. Tucker, et al.. (2015). Modeling a model: Mouse genetics, 22q11.2 Deletion Syndrome, and disorders of cortical circuit development. Progress in Neurobiology. 130. 1–28. 71 indexed citations
10.
Meechan, Daniel W., et al.. (2013). Cognitive Ability is Associated with Altered Medial Frontal Cortical Circuits in the LgDel Mouse Model of 22q11.2DS. Cerebral Cortex. 25(5). 1143–1151. 36 indexed citations
11.
Maynard, Thomas M., et al.. (2012). 22q11 Gene dosage establishes an adaptive range for sonic hedgehog and retinoic acid signaling during early development. Human Molecular Genetics. 22(2). 300–312. 36 indexed citations
12.
Rawson, Nancy E., Fritz W. Lischka, Karen K. Yee, et al.. (2010). Specific mesenchymal/epithelial induction of olfactory receptor, vomeronasal, and gonadotropin‐releasing hormone (GnRH) neurons. Developmental Dynamics. 239(6). 1723–1738. 15 indexed citations
13.
Manzini, M. Chiara, Anna Rajab, Thomas M. Maynard, et al.. (2009). Developmental and degenerative features in a complicated spastic paraplegia. Annals of Neurology. 67(4). 516–525. 27 indexed citations
14.
Maynard, Thomas M., Daniel W. Meechan, Tomoko Sugimoto, et al.. (2008). Mitochondrial localization and function of a subset of 22q11 deletion syndrome candidate genes. Molecular and Cellular Neuroscience. 39(3). 439–451. 90 indexed citations
15.
Maynard, Thomas M., Daniel W. Meechan, Clifford Heindel, et al.. (2006). No evidence for parental imprinting of mouse 22q11 gene orthologs. Mammalian Genome. 17(8). 822–832. 7 indexed citations
16.
Maynard, Thomas M., et al.. (2003). Mesenchymal/epithelial regulation of retinoic acid signaling in the olfactory placode. Developmental Biology. 261(1). 82–98. 49 indexed citations
17.
Haskell, Gloria, et al.. (2002). Retinoic acid signaling at sites of plasticity in the mature central nervous system. The Journal of Comparative Neurology. 452(3). 228–241. 67 indexed citations
18.
Maynard, Thomas M., et al.. (2002). High-resolution mapping of the Gli3 mutation Extra-toesJ reveals a 51.5-kb deletion. Mammalian Genome. 13(1). 58–61. 77 indexed citations
19.
Maynard, Thomas M., Linmarie Sikich, J.A. Lieberman, & Anthony‐Samuel LaMantia. (2001). Neural Development, Cell-Cell Signaling, and the "Two-Hit" Hypothesis of Schizophrenia. Schizophrenia Bulletin. 27(3). 457–476. 285 indexed citations
20.
Henion, Paul D., et al.. (2000). Avian transitin expression mirrors glial cell fate restrictions during neural crest development. Developmental Dynamics. 218(1). 150–159. 23 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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