Daniel W. Meechan

1.1k total citations
17 papers, 761 citations indexed

About

Daniel W. Meechan is a scholar working on Molecular Biology, Genetics and Cellular and Molecular Neuroscience. According to data from OpenAlex, Daniel W. Meechan has authored 17 papers receiving a total of 761 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 9 papers in Genetics and 4 papers in Cellular and Molecular Neuroscience. Recurrent topics in Daniel W. Meechan's work include Congenital heart defects research (13 papers), Genetics and Neurodevelopmental Disorders (4 papers) and Genomic variations and chromosomal abnormalities (4 papers). Daniel W. Meechan is often cited by papers focused on Congenital heart defects research (13 papers), Genetics and Neurodevelopmental Disorders (4 papers) and Genomic variations and chromosomal abnormalities (4 papers). Daniel W. Meechan collaborates with scholars based in United States, United Kingdom and Bulgaria. Daniel W. Meechan's co-authors include Thomas M. Maynard, Anthony‐Samuel LaMantia, Eric S. Tucker, Beverly A. Karpinski, Yifei Wu, Lawrence A. Rothblat, Jeffrey A. Lieberman, Tomoko Sugimoto, Anastas Popratiloff and Erin R. Bonner and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Neuron and Journal of Neuroscience.

In The Last Decade

Daniel W. Meechan

16 papers receiving 757 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Daniel W. Meechan United States 13 550 277 138 122 115 17 761
Takeshi Hiramoto Japan 13 372 0.7× 187 0.7× 166 1.2× 132 1.1× 56 0.5× 21 735
Mari Kondo Japan 15 351 0.6× 169 0.6× 140 1.0× 126 1.0× 23 0.2× 41 726
David McNay United States 10 367 0.7× 176 0.6× 101 0.7× 61 0.5× 78 0.7× 12 1.0k
Zhilian Xia United States 17 589 1.1× 445 1.6× 179 1.3× 122 1.0× 21 0.2× 22 1.0k
Christina Chatzi United States 13 539 1.0× 190 0.7× 218 1.6× 67 0.5× 18 0.2× 20 768
David Chun Cheong Tsui United States 7 290 0.5× 76 0.3× 104 0.8× 43 0.4× 26 0.2× 14 579
Liam P. Tuffy Ireland 12 474 0.9× 92 0.3× 295 2.1× 90 0.7× 107 0.9× 13 798
Nicola Romanò United Kingdom 15 271 0.5× 145 0.5× 98 0.7× 38 0.3× 38 0.3× 34 804
Beatrice Paradiso Italy 17 321 0.6× 157 0.6× 394 2.9× 111 0.9× 48 0.4× 36 966
Miriam Fernández Spain 16 310 0.6× 64 0.2× 225 1.6× 58 0.5× 34 0.3× 29 726

Countries citing papers authored by Daniel W. Meechan

Since Specialization
Citations

This map shows the geographic impact of Daniel W. Meechan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Daniel W. Meechan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Daniel W. Meechan more than expected).

Fields of papers citing papers by Daniel W. Meechan

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Daniel W. Meechan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Daniel W. Meechan. The network helps show where Daniel W. Meechan may publish in the future.

Co-authorship network of co-authors of Daniel W. Meechan

This figure shows the co-authorship network connecting the top 25 collaborators of Daniel W. Meechan. A scholar is included among the top collaborators of Daniel W. Meechan based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Daniel W. Meechan. Daniel W. Meechan is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
Meechan, Daniel W., et al.. (2023). Out of Line or Altered States? Neural Progenitors as a Target in a Polygenic Neurodevelopmental Disorder. Developmental Neuroscience. 46(1). 1–21.
2.
Meechan, Daniel W., et al.. (2023). Ranbp1modulates morphogenesis of the craniofacial midline in mouse models of 22q11.2 deletion syndrome. Human Molecular Genetics. 32(12). 1959–1974. 3 indexed citations
3.
Meechan, Daniel W., Beverly A. Karpinski, Erin R. Bonner, et al.. (2019). Mitochondrial Dysfunction Leads to Cortical Under-Connectivity and Cognitive Impairment. Neuron. 102(6). 1127–1142.e3. 106 indexed citations
4.
Meechan, Daniel W., Thomas M. Maynard, Eric S. Tucker, et al.. (2015). Modeling a model: Mouse genetics, 22q11.2 Deletion Syndrome, and disorders of cortical circuit development. Progress in Neurobiology. 130. 1–28. 71 indexed citations
5.
Myers, Alyson K., et al.. (2014). Cortical Interneurons Require Jnk1 to Enter and Navigate the Developing Cerebral Cortex. Journal of Neuroscience. 34(23). 7787–7801. 26 indexed citations
6.
7.
Meechan, Daniel W., et al.. (2013). Cognitive Ability is Associated with Altered Medial Frontal Cortical Circuits in the LgDel Mouse Model of 22q11.2DS. Cerebral Cortex. 25(5). 1143–1151. 36 indexed citations
8.
Meechan, Daniel W., Eric S. Tucker, Thomas M. Maynard, & Anthony‐Samuel LaMantia. (2012). Cxcr4 regulation of interneuron migration is disrupted in 22q11.2 deletion syndrome. Proceedings of the National Academy of Sciences. 109(45). 18601–18606. 80 indexed citations
9.
Rawson, Nancy E., Fritz W. Lischka, Karen K. Yee, et al.. (2010). Specific mesenchymal/epithelial induction of olfactory receptor, vomeronasal, and gonadotropin‐releasing hormone (GnRH) neurons. Developmental Dynamics. 239(6). 1723–1738. 15 indexed citations
10.
Meechan, Daniel W., et al.. (2010). Three phases of DiGeorge/22q11 deletion syndrome pathogenesis during brain development: Patterning, proliferation, and mitochondrial functions of 22q11 genes. International Journal of Developmental Neuroscience. 29(3). 283–294. 48 indexed citations
11.
Meechan, Daniel W., Eric S. Tucker, Thomas M. Maynard, & Anthony‐Samuel LaMantia. (2009). Diminished dosage of 22q11 genes disrupts neurogenesis and cortical development in a mouse model of 22q11 deletion/DiGeorge syndrome. Proceedings of the National Academy of Sciences. 106(38). 16434–16445. 131 indexed citations
12.
Maynard, Thomas M., Daniel W. Meechan, Tomoko Sugimoto, et al.. (2008). Mitochondrial localization and function of a subset of 22q11 deletion syndrome candidate genes. Molecular and Cellular Neuroscience. 39(3). 439–451. 90 indexed citations
13.
Maynard, Thomas M., Daniel W. Meechan, Clifford Heindel, et al.. (2006). No evidence for parental imprinting of mouse 22q11 gene orthologs. Mammalian Genome. 17(8). 822–832. 7 indexed citations
14.
Meechan, Daniel W., et al.. (2006). When Half Is Not Enough: Gene Expression and Dosage in the 22q11 Deletion Syndrome. Gene Expression. 13(6). 299–310. 42 indexed citations
15.
Tucker, Eric S., Gloria Haskell, Thomas M. Maynard, et al.. (2006). Limited influence of olanzapine on adult forebrain neural precursors in vitro. Neuroscience. 140(1). 111–122. 11 indexed citations
16.
Meechan, Daniel W., et al.. (2006). Gene dosage in the developing and adult brain in a mouse model of 22q11 deletion syndrome. Molecular and Cellular Neuroscience. 33(4). 412–428. 37 indexed citations
17.
Quinton, Naomi, et al.. (2004). Single nucleotide polymorphisms in the leptin receptor gene: studies in anorexia nervosa. Psychiatric Genetics. 14(4). 191–194. 18 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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