Thomas M. Ashton

1.8k total citations · 1 hit paper
11 papers, 1.3k citations indexed

About

Thomas M. Ashton is a scholar working on Molecular Biology, Cancer Research and Oncology. According to data from OpenAlex, Thomas M. Ashton has authored 11 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 5 papers in Cancer Research and 4 papers in Oncology. Recurrent topics in Thomas M. Ashton's work include DNA Repair Mechanisms (3 papers), Mitochondrial Function and Pathology (3 papers) and Cancer, Hypoxia, and Metabolism (3 papers). Thomas M. Ashton is often cited by papers focused on DNA Repair Mechanisms (3 papers), Mitochondrial Function and Pathology (3 papers) and Cancer, Hypoxia, and Metabolism (3 papers). Thomas M. Ashton collaborates with scholars based in United Kingdom, Denmark and Germany. Thomas M. Ashton's co-authors include Geoff S. Higgins, W. Gillies McKenna, Leoni A. Kunz‐Schughart, Ian D. Hickson, Giacomo Pirovano, Hocine W Mankouri, Emmanouil Fokas, Matt Kelly, Michael R.L. Stratford and Ruth J. Muschel and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nature Communications and Molecular and Cellular Biology.

In The Last Decade

Thomas M. Ashton

11 papers receiving 1.3k citations

Hit Papers

Oxidative Phosphorylation as an Emerging Target in Cancer... 2018 2026 2020 2023 2018 250 500 750
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. Oxidative Phosphorylation as an Emerging Target in Cancer Therapy
    2018 797 citations Thomas M. Ashton, W. Gillies McKenna et al. Clinical Cancer Research profile →

Peers

Thomas M. Ashton
Min Kyung Ju South Korea
Pin Yi China
Flonné Wildes United States
H. Charles Manning United States
Yuxi Zhu China
Mark Esposito United States
Jingli Hao Australia
Hye Gyeong Park South Korea
Aki Pui‐Wah Tse Hong Kong
Jérôme Durivault France
Min Kyung Ju South Korea
Thomas M. Ashton
Citations per year, relative to Thomas M. Ashton Thomas M. Ashton (= 1×) peers Min Kyung Ju

Countries citing papers authored by Thomas M. Ashton

Since Specialization
Citations

This map shows the geographic impact of Thomas M. Ashton's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas M. Ashton with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas M. Ashton more than expected).

Fields of papers citing papers by Thomas M. Ashton

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas M. Ashton. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas M. Ashton. The network helps show where Thomas M. Ashton may publish in the future.

Co-authorship network of co-authors of Thomas M. Ashton

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas M. Ashton. A scholar is included among the top collaborators of Thomas M. Ashton based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas M. Ashton. Thomas M. Ashton is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

11 of 11 papers shown
1.
Coates, James T., Gonzalo Rodríguez‐Berriguete, Rathi Puliyadi, et al.. (2020). The anti-malarial drug atovaquone potentiates platinum-mediated cancer cell death by increasing oxidative stress. Cell Death Discovery. 6(1). 110–110. 18 indexed citations
2.
Anderson, Victoria E., Anika M. Weber, Tina Ahmed, et al.. (2019). Abstract 2313: Enhanced activity of second-generation MAGE-A4 SPEAR T-cells through co-expression of a CD8α homodimer. Immunology. 2313–2313. 1 indexed citations
3.
Anderson, Victoria E., Anika M. Weber, Tina Ahmed, et al.. (2019). Abstract 2313: Enhanced activity of second-generation MAGE-A4 SPEAR T-cells through co-expression of a CD8α homodimer. Cancer Research. 79(13_Supplement). 2313–2313. 4 indexed citations
4.
Herbert, Katharine, Thomas M. Ashton, Remko Prevo, Giacomo Pirovano, & Geoff S. Higgins. (2018). T-LAK cell-originated protein kinase (TOPK): an emerging target for cancer-specific therapeutics. Cell Death and Disease. 9(11). 1089–1089. 60 indexed citations
5.
Ashton, Thomas M., W. Gillies McKenna, Leoni A. Kunz‐Schughart, & Geoff S. Higgins. (2018). Oxidative Phosphorylation as an Emerging Target in Cancer Therapy. Clinical Cancer Research. 24(11). 2482–2490. 797 indexed citations breakdown →
6.
Pirovano, Giacomo, Thomas M. Ashton, Katharine Herbert, et al.. (2017). TOPK modulates tumour-specific radiosensitivity and correlates with recurrence after prostate radiotherapy. British Journal of Cancer. 117(4). 503–512. 22 indexed citations
7.
Ashton, Thomas M., Emmanouil Fokas, Leoni A. Kunz‐Schughart, et al.. (2016). The anti-malarial atovaquone increases radiosensitivity by alleviating tumour hypoxia. Nature Communications. 7(1). 12308–12308. 195 indexed citations
8.
Kelly, Matt, Emmanouil Fokas, Pavitra Kannan, et al.. (2014). Regulation of O2 consumption by the PI3K and mTOR pathways contributes to tumor hypoxia. Radiotherapy and Oncology. 111(1). 72–80. 36 indexed citations
9.
Mankouri, Hocine W, Thomas M. Ashton, & Ian D. Hickson. (2011). Holliday junction-containing DNA structures persist in cells lacking Sgs1 or Top3 following exposure to DNA damage. Proceedings of the National Academy of Sciences. 108(12). 4944–4949. 46 indexed citations
10.
Ashton, Thomas M., et al.. (2011). Pathways for Holliday Junction Processing during Homologous Recombination in Saccharomyces cerevisiae. Molecular and Cellular Biology. 31(9). 1921–1933. 54 indexed citations
11.
Ashton, Thomas M. & Ian D. Hickson. (2010). Yeast as a model system to study RecQ helicase function. DNA repair. 9(3). 303–314. 46 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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