Thomas Krag

3.7k total citations · 1 hit paper
83 papers, 2.7k citations indexed

About

Thomas Krag is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Thomas Krag has authored 83 papers receiving a total of 2.7k indexed citations (citations by other indexed papers that have themselves been cited), including 61 papers in Molecular Biology, 26 papers in Cellular and Molecular Neuroscience and 16 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Thomas Krag's work include Muscle Physiology and Disorders (38 papers), Genetic Neurodegenerative Diseases (17 papers) and Mitochondrial Function and Pathology (14 papers). Thomas Krag is often cited by papers focused on Muscle Physiology and Disorders (38 papers), Genetic Neurodegenerative Diseases (17 papers) and Mitochondrial Function and Pathology (14 papers). Thomas Krag collaborates with scholars based in Denmark, United States and Spain. Thomas Krag's co-authors include Tejvir S. Khurana, John Vissing, Sasha Bogdanovich, Elisabeth R. Barton, Rexford S. Ahima, Simon Hauerslev, Tina D. Jeppesen, Morten Dunø, Kelly J. Perkins and Steen Gammeltoft and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Thomas Krag

79 papers receiving 2.7k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Functional improvement of dystrophic muscle by myostatin blockade

2002 651 citations Sasha Bogdanovich, Thomas Krag et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Thomas Krag Denmark	24	2.2k	629	452	406	376	83	2.7k
Anne Picard Italy	15	2.9k 1.3×	1.1k 1.8×	630 1.4×	415 1.0×	257 0.7×	32	3.6k
Giulia Milan Italy	11	2.6k 1.2×	1.2k 2.0×	660 1.5×	311 0.8×	207 0.6×	14	3.4k
Erqian Na United States	15	3.1k 1.4×	1.3k 2.1×	935 2.1×	489 1.2×	492 1.3×	20	4.1k
Lionel Tintignac Switzerland	24	2.1k 1.0×	715 1.1×	588 1.3×	332 0.8×	146 0.4×	37	2.7k
Venus Lai United States	2	2.4k 1.1×	981 1.6×	724 1.6×	406 1.0×	291 0.8×	4	2.9k
Shinji Hatakeyama Japan	23	1.6k 0.7×	857 1.4×	404 0.9×	352 0.9×	117 0.3×	45	2.6k
Martin Brockington United Kingdom	34	3.7k 1.7×	362 0.6×	518 1.1×	814 2.0×	761 2.0×	55	4.1k
Claudio Bruno Italy	37	3.3k 1.5×	588 0.9×	697 1.5×	483 1.2×	482 1.3×	146	4.6k
Leslie M. Baehr United States	23	1.9k 0.8×	978 1.6×	725 1.6×	294 0.7×	174 0.5×	33	2.5k
Jill A. Rafael‐Fortney United States	23	1.7k 0.8×	525 0.8×	214 0.5×	227 0.6×	653 1.7×	56	2.1k

Countries citing papers authored by Thomas Krag

Since Specialization

Citations

This map shows the geographic impact of Thomas Krag's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas Krag with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas Krag more than expected).

Fields of papers citing papers by Thomas Krag

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas Krag. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas Krag. The network helps show where Thomas Krag may publish in the future.

Co-authorship network of co-authors of Thomas Krag

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas Krag. A scholar is included among the top collaborators of Thomas Krag based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas Krag. Thomas Krag is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Straub, Volker, et al.. (2025). A homozygous variant in the beta-1,3-N-acetylglucosaminyltransferase 4 gene causes progressive brain atrophy and muscular dystrophy. European Journal of Human Genetics. 34(2). 288–292.

Krag, Thomas, Lauren Brady, France Leturcq, et al.. (2025). Variants in CAPN3 Causing Autosomal Dominant Limb–Girdle Muscular Dystrophy Combined With Calpain‐3 Deficiency. Human Mutation. 2025(1). 9301465–9301465. 1 indexed citations

Krag, Thomas, et al.. (2024). Quantitative Muscle MRI to Monitor Disease Progression in Hypokalemic Periodic Paralysis. Neurology Genetics. 10(6). e200211–e200211. 1 indexed citations

Krag, Thomas, et al.. (2023). Skeletal Muscle Involvement in Patients With Truncations of Titin and Familial Dilated Cardiomyopathy. JACC Heart Failure. 12(4). 740–753. 7 indexed citations

Dunø, Morten, Ulf Birkedal, John Vissing, et al.. (2023). Homozygous splice variant (c.1741-6G>A) of the COL6A1 gene in three patients with Ullrich congenital muscular dystrophy. Neuromuscular Disorders. 33(7). 539–545.

Santalla, Alfredo, Gisela Nogales‐Gadea, Pedro L. Valenzuela, et al.. (2022). Low aerobic capacity in McArdle disease: A role for mitochondrial network impairment?. Molecular Metabolism. 66. 101648–101648. 8 indexed citations

Dunø, Morten, et al.. (2021). Myopathy can be a key phenotype of membrin (GOSR2) deficiency. Human Mutation. 42(9). 1101–1106. 2 indexed citations

Brull, Astrid, Gisela Nogales‐Gadea, Antoni L. Andreu, et al.. (2021). Preclinical Research in McArdle Disease: A Review of Research Models and Therapeutic Strategies. Genes. 13(1). 74–74. 5 indexed citations

Vissing, John, et al.. (2021). Antimyostatin Treatment in Health and Disease: The Story of Great Expectations and Limited Success. Cells. 10(3). 533–533. 32 indexed citations

10.

Krag, Thomas, et al.. (2021). Autophagy is affected in patients with hypokalemic periodic paralysis: an involvement in vacuolar myopathy?. Acta Neuropathologica Communications. 9(1). 109–109. 4 indexed citations

11.

Fritzen, Andreas M., et al.. (2020). Preserved Capacity for Adaptations in Strength and Muscle Regulatory Factors in Elderly in Response to Resistance Exercise Training and Deconditioning. Journal of Clinical Medicine. 9(7). 2188–2188. 10 indexed citations

12.

Fritzen, Andreas M., Søren Andersen, Thomas Krag, et al.. (2020). Effect of Aerobic Exercise Training and Deconditioning on Oxidative Capacity and Muscle Mitochondrial Enzyme Machinery in Young and Elderly Individuals. Journal of Clinical Medicine. 9(10). 3113–3113. 25 indexed citations

13.

Luna, Noemí de, Gisela Nogales‐Gadea, Antoni L. Andreu, et al.. (2020). Preclinical Research in Glycogen Storage Diseases: A Comprehensive Review of Current Animal Models. International Journal of Molecular Sciences. 21(24). 9621–9621. 16 indexed citations

14.

Vissing, John, Julia R. Dahlqvist, Carinne Roudaut, et al.. (2020). A single c.1715G>C calpain 3 gene variant causes dominant calpainopathy with loss of calpain 3 expression and activity. Human Mutation. 41(9). 1507–1513. 10 indexed citations

15.

Fritzen, Andreas M., Kasper Højgaard Thybo, Christoffer Rasmus Vissing, et al.. (2019). Adaptations in Mitochondrial Enzymatic Activity Occurs Independent of Genomic Dosage in Response to Aerobic Exercise Training and Deconditioning in Human Skeletal Muscle. Cells. 8(3). 237–237. 27 indexed citations

16.

Luna, Noemí de, Astrid Brull, Miguel A. Martı́n, et al.. (2019). Absence of p.R50X Pygm read-through in McArdle disease cellular models. Disease Models & Mechanisms. 13(1). 10 indexed citations

17.

Krag, Thomas, et al.. (2018). Expanding the phenotype of filamin-C-related myofibrillar myopathy. Clinical Neurology and Neurosurgery. 176. 30–33. 4 indexed citations

18.

Andersen, Annarita Ghosh, Henrik Daa Schrøder, Thomas Krag, et al.. (2018). BAG3 myopathy is not always associated with cardiomyopathy. Neuromuscular Disorders. 28(9). 798–801. 9 indexed citations

19.

Pinós, Tomàs, et al.. (2017). Exercising with blocked muscle glycogenolysis: Adaptation in the McArdle mouse. Molecular Genetics and Metabolism. 123(1). 21–27. 8 indexed citations

20.

Bogdanovich, Sasha, Kelly J. Perkins, Thomas Krag, & Tejvir S. Khurana. (2004). Therapeutics for Duchenne muscular dystrophy: current approaches and future directions. Journal of Molecular Medicine. 82(2). 102–115. 78 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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