Thomas K. Baker
- Molecular Biology
- Pharmacology top 5%
- Oncology
- Biomedical Engineering
- Health, Toxicology and Mutagenesis top 10%
- Co-authors
- James StevensTimothy P. RyanJames E. KlaunigGeorge H. SearfossHong GaoMark A. CarfagnaA KwiatkowskiErnst R. Dow
- Topics
- Computational Drug Discovery Methods (4 papers)Drug Transport and Resistance Mechanisms (3 papers)Connexins and lens biology (3 papers)
- Partner nations
- United StatesUnited KingdomSouth Korea
In The Last Decade
Thomas K. Baker
27 papers receiving 781 citations
Peers
Comparison fields: 5 of 101
- Molecular Biology 335
- Pharmacology 130
- Oncology 109
- Biomedical Engineering 100
- Health, Toxicology and Mutagenesis 95
Countries citing papers authored by Thomas K. Baker
This map shows the geographic impact of Thomas K. Baker's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas K. Baker with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas K. Baker more than expected).
Fields of papers citing papers by Thomas K. Baker
This network shows the impact of papers produced by Thomas K. Baker. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas K. Baker. The network helps show where Thomas K. Baker may publish in the future.
Co-authorship network of co-authors of Thomas K. Baker
This figure shows the co-authorship network connecting the top 25 collaborators of Thomas K. Baker. A scholar is included among the top collaborators of Thomas K. Baker based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas K. Baker. Thomas K. Baker is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 4 | |
| 2 | 1 | |
| 3 | 10 | |
| 4 | 13 | |
| 5 | 32 | |
| 6 | 4 | |
| 7 | 17 | |
| 8 | 17 | |
| 9 | 26 | |
| 10 | 208 | |
| 11 | 31 | |
| 12 | 20 | |
| 13 | 48 | |
| 14 | 30 | |
| 15 | 118 | |
| 16 | Modulation of gap junctional intercellular communication in rodent, monkey and human hepatocyte by nongenotoxic compounds. | 15 |
| 17 | 42 | |
| 18 | 50 | |
| 19 | 25 | |
| 20 | 26 |
About Thomas K. Baker
Thomas K. Baker is a scholar working on Health, Toxicology and Mutagenesis, Small Animals and Geriatrics and Gerontology, having authored 28 papers that have together received 818 indexed citations. Recurring topics across this work include Computational Drug Discovery Methods (4 papers), Drug Transport and Resistance Mechanisms (3 papers) and Connexins and lens biology (3 papers). The work is most often cited by research in Pharmacology (130 citations), Hepatology (78 citations) and Health, Toxicology and Mutagenesis (95 citations). Thomas K. Baker has collaborated with scholars based in United States, United Kingdom and South Korea. Frequent co-authors include James Stevens, Timothy P. Ryan, James E. Klaunig, George H. Searfoss, Hong Gao, Mark A. Carfagna, A Kwiatkowski, Ernst R. Dow, Qingqin S. Li and Thomas F. Peeper. Their work appears in journals such as Cancer Research, Journal of Medicinal Chemistry and Lab on a Chip.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.