Thomas Hentrich

1.6k total citations
33 papers, 1.1k citations indexed

About

Thomas Hentrich is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Neurology. According to data from OpenAlex, Thomas Hentrich has authored 33 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Molecular Biology, 12 papers in Cellular and Molecular Neuroscience and 12 papers in Neurology. Recurrent topics in Thomas Hentrich's work include Parkinson's Disease Mechanisms and Treatments (10 papers), Nuclear Receptors and Signaling (8 papers) and Epigenetics and DNA Methylation (5 papers). Thomas Hentrich is often cited by papers focused on Parkinson's Disease Mechanisms and Treatments (10 papers), Nuclear Receptors and Signaling (8 papers) and Epigenetics and DNA Methylation (5 papers). Thomas Hentrich collaborates with scholars based in Germany, Canada and United States. Thomas Hentrich's co-authors include Julia M. Schulze‐Hentrich, Michael S. Kobor, Ali Shilatifard, Mark D. Wilkinson, Benjamin M. Good, Shima Nakanishi, Sue L. Jaspersen, Jessica Jackson, Olaf Rieß and Eldon Emberly and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Genes & Development and Blood.

In The Last Decade

Thomas Hentrich

32 papers receiving 1.1k citations

Peers

Thomas Hentrich

NEURO

CMN

Pei-I Tsai Taiwan

Jinsong Cai China

David Schubert United States

Kui Duan China

Anita Bandrowski United States

Patrick J. Gannon United States

Kenneth Bryan Ireland

Douglas Hoffman United States

Ji He China

Esti Yeger‐Lotem Israel

Pei-I Tsai Taiwan

Thomas Hentrich

230 ×0.3

125 ×0.9

NEURO

109 ×0.9

CMN

33 ×0.3

73 ×1.3

Citations per year, relative to Thomas Hentrich Thomas Hentrich (= 1×) peers Pei-I Tsai

Countries citing papers authored by Thomas Hentrich

Since Specialization

Citations

This map shows the geographic impact of Thomas Hentrich's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas Hentrich with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas Hentrich more than expected).

Fields of papers citing papers by Thomas Hentrich

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas Hentrich. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas Hentrich. The network helps show where Thomas Hentrich may publish in the future.

Co-authorship network of co-authors of Thomas Hentrich

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas Hentrich. A scholar is included among the top collaborators of Thomas Hentrich based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas Hentrich. Thomas Hentrich is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Hentrich, Thomas, Julia M. Schulze‐Hentrich, Matthias H. Munk, et al.. (2023). Blood transcriptome analysis suggests an indirect molecular association of early life adversities and adult social anxiety disorder by immune-related signal transduction. Frontiers in Psychiatry. 14. 1125553–1125553. 6 indexed citations

Fitzel, Rahel, Hildegard Keppeler, Rebekka Schairer, et al.. (2023). Targeting MYC in combination with epigenetic regulators induces synergistic anti-leukemic effects in MLLr leukemia and simultaneously improves immunity. Neoplasia. 41. 100902–100902. 4 indexed citations

Hentrich, Thomas, Rebekka Schairer, Hildegard Keppeler, et al.. (2023). The RORɣ/SREBP2 pathway is a master regulator of cholesterol metabolism and serves as potential therapeutic target in t(4;11) leukemia. Oncogene. 43(4). 281–293. 7 indexed citations

Brucker, Sara Y., et al.. (2022). Endometrial organoids derived from Mayer–Rokitansky–Küster–Hauser syndrome patients provide insights into disease-causing pathways. Disease Models & Mechanisms. 15(5). 12 indexed citations

Buchert, Rebecca, Thomas Hentrich, Nico Weber, et al.. (2022). Genome Sequencing and Transcriptome Profiling in Twins Discordant for Mayer-Rokitansky-Küster-Hauser Syndrome. Journal of Clinical Medicine. 11(19). 5598–5598. 9 indexed citations

Hentrich, Thomas, Zinah Wassouf, James D. Mills, et al.. (2020). Increased expression of myelin-associated genes in frontal cortex of <i>SNCA</i> overexpressing rats and Parkinson’s disease patients. Aging. 12(19). 18889–18906. 13 indexed citations

Cheng, Fubo, Michael Walter, Zinah Wassouf, et al.. (2020). Unraveling Molecular Mechanisms of THAP1 Missense Mutations in DYT6 Dystonia. Journal of Molecular Neuroscience. 70(7). 999–1008. 14 indexed citations

Hentrich, Thomas, André Koch, Nico Weber, et al.. (2020). The Endometrial Transcription Landscape of MRKH Syndrome. Frontiers in Cell and Developmental Biology. 8. 572281–572281. 17 indexed citations

Keppeler, Hildegard, Hannes Schmid, Dominik Schneidawind, et al.. (2019). Inhibition of DOT1L and PRMT5 promote synergistic anti-tumor activity in a human MLL leukemia model induced by CRISPR/Cas9. Oncogene. 38(46). 7181–7195. 26 indexed citations

10.

Hentrich, Thomas, et al.. (2019). The Challenge and Opportunity to Diagnose Parkinson's Disease in Midlife. Frontiers in Neurology. 10. 1328–1328. 25 indexed citations

11.

Novati, Arianna, Thomas Hentrich, Zinah Wassouf, et al.. (2018). Environment-dependent striatal gene expression in the BACHD rat model for Huntington disease. Scientific Reports. 8(1). 5803–5803. 10 indexed citations

12.

Wassouf, Zinah, Thomas Hentrich, Carola Rotermund, et al.. (2018). Environmental Enrichment Prevents Transcriptional Disturbances Induced by Alpha-Synuclein Overexpression. Frontiers in Cellular Neuroscience. 12. 112–112. 29 indexed citations

13.

Paiva, Isabel, Gaurav Jain, Diana F. Lázaro, et al.. (2018). Alpha-synuclein deregulates the expression of COL4A2 and impairs ER-Golgi function. Neurobiology of Disease. 119. 121–135. 46 indexed citations

14.

Bonzheim, Irina, Claudia Dufke, Thomas Hentrich, et al.. (2017). Next generation sequencing of the clonal IGH rearrangement detects ongoing mutations and interfollicular trafficking in in situ follicular neoplasia. PLoS ONE. 12(6). e0178503–e0178503. 14 indexed citations

15.

Häbig, Karina, Sandra Gellhaar, Florian Giesert, et al.. (2013). LRRK2 guides the actin cytoskeleton at growth cones together with ARHGEF7 and Tropomyosin 4. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1832(12). 2352–2367. 49 indexed citations

16.

Lenstra, Tineke L., Joris J. Benschop, Tae Soo Kim, et al.. (2011). The Specificity and Topology of Chromatin Interaction Pathways in Yeast. Molecular Cell. 42(4). 536–549. 181 indexed citations

17.

Takahashi, Yoh-hei, Julia M. Schulze‐Hentrich, Jessica Jackson, et al.. (2011). Dot1 and Histone H3K79 Methylation in Natural Telomeric and HM Silencing. Molecular Cell. 42(1). 118–126. 93 indexed citations

18.

Schulze‐Hentrich, Julia M., Thomas Hentrich, Shima Nakanishi, et al.. (2011). Splitting the task: Ubp8 and Ubp10 deubiquitinate different cellular pools of H2BK123. Genes & Development. 25(21). 2242–2247. 86 indexed citations

19.

Schulze‐Hentrich, Julia M., Jessica Jackson, Shima Nakanishi, et al.. (2009). Linking Cell Cycle to Histone Modifications: SBF and H2B Monoubiquitination Machinery and Cell-Cycle Regulation of H3K79 Dimethylation. Molecular Cell. 35(5). 626–641. 140 indexed citations

20.

Bergmann, Antje, M. Reimann, Thomas Hentrich, et al.. (2008). Polymorphisms in Perilipin Gene (PLIN) are not Associated with Obesity and Weight Variation in People with High Risk of Type 2 Diabetes. Experimental and Clinical Endocrinology & Diabetes. 116(S 01). S56–S58. 9 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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