Thomas G. Koch

2.1k total citations
60 papers, 1.6k citations indexed

About

Thomas G. Koch is a scholar working on Genetics, Surgery and Molecular Biology. According to data from OpenAlex, Thomas G. Koch has authored 60 papers receiving a total of 1.6k indexed citations (citations by other indexed papers that have themselves been cited), including 29 papers in Genetics, 21 papers in Surgery and 15 papers in Molecular Biology. Recurrent topics in Thomas G. Koch's work include Mesenchymal stem cell research (29 papers), Tissue Engineering and Regenerative Medicine (18 papers) and Osteoarthritis Treatment and Mechanisms (11 papers). Thomas G. Koch is often cited by papers focused on Mesenchymal stem cell research (29 papers), Tissue Engineering and Regenerative Medicine (18 papers) and Osteoarthritis Treatment and Mechanisms (11 papers). Thomas G. Koch collaborates with scholars based in Canada, Denmark and United States. Thomas G. Koch's co-authors include Dean H. Betts, John R. Sodeau, Keith A. Russell, Preben D. Thomsen, Andrew B. Horn, Lise Charlotte Berg, Dorothee Bienzle, Thomas W. G. Gibson, Jonathan LaMarre and Judith Koenig and has published in prestigious journals such as SHILAP Revista de lepidopterología, Journal of Geophysical Research Atmospheres and PLoS ONE.

In The Last Decade

Thomas G. Koch

56 papers receiving 1.5k citations

Peers

Thomas G. Koch
Locksley E. McGann Canada
Erik J. Woods United States
Christer Lindqvist Finland
L.E. McGann Canada
Takahiro Okabe Japan
Stefan A. Tschanz Switzerland
Pietro Gobbi Italy
Timothy A. Steele United States
Daniel Haddad Germany
T. Ono Japan
Locksley E. McGann Canada
Thomas G. Koch
Citations per year, relative to Thomas G. Koch Thomas G. Koch (= 1×) peers Locksley E. McGann

Countries citing papers authored by Thomas G. Koch

Since Specialization
Citations

This map shows the geographic impact of Thomas G. Koch's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas G. Koch with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas G. Koch more than expected).

Fields of papers citing papers by Thomas G. Koch

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas G. Koch. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas G. Koch. The network helps show where Thomas G. Koch may publish in the future.

Co-authorship network of co-authors of Thomas G. Koch

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas G. Koch. A scholar is included among the top collaborators of Thomas G. Koch based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas G. Koch. Thomas G. Koch is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Lively, Starlee, Pratibha Potla, Nathalie Côté, et al.. (2024). microRNAs are differentially expressed in equine plasma of horses with osteoarthritis and osteochondritis dissecans versus control horses. PLoS ONE. 19(2). e0297303–e0297303. 2 indexed citations
2.
Roberts, Eugene L., et al.. (2023). Computer controlled expansion of equine cord blood mesenchymal stromal cells on microcarriers in 3 L vertical-wheel® bioreactors. Frontiers in Bioengineering and Biotechnology. 11. 1250077–1250077. 1 indexed citations
3.
Koch, Thomas G., et al.. (2023). Overview of Equine Stem Cells. Veterinary Clinics of North America Equine Practice. 39(3). 461–474.
4.
Lively, Starlee, et al.. (2023). MicroRNAs as Prognostic Markers for Chondrogenic Differentiation Potential of Equine Mesenchymal Stromal Cells. Stem Cells and Development. 32(21-22). 693–702.
5.
Russell, Keith A., et al.. (2023). Opening the “Black Box” Underlying Barriers to the Use of Canine Induced Pluripotent Stem Cells: A Narrative Review. Stem Cells and Development. 32(11-12). 271–291. 2 indexed citations
6.
7.
Arzi, Boaz, Tracy L. Webb, Thomas G. Koch, et al.. (2021). Cell Therapy in Veterinary Medicine as a Proof-of-Concept for Human Therapies: Perspectives From the North American Veterinary Regenerative Medicine Association. Frontiers in Veterinary Science. 8. 779109–779109. 13 indexed citations
8.
Koenig, Judith, et al.. (2020). Extracorporeal Shock Wave Therapy Enhances the In Vitro Metabolic Activity and Differentiation of Equine Umbilical Cord Blood Mesenchymal Stromal Cells. Frontiers in Veterinary Science. 7. 554306–554306. 8 indexed citations
9.
Kamesan, Vashine, et al.. (2019). Beyond Cartilage Repair: The Role of the Osteochondral Unit in Joint Health and Disease. Tissue Engineering Part B Reviews. 25(2). 114–125. 75 indexed citations
10.
Luk, Franka, Sander S. Korevaar, Thomas G. Koch, et al.. (2019). The Importance of Dosing, Timing, and (in)Activation of Adipose Tissue-Derived Mesenchymal Stromal Cells on Their Immunomodulatory Effects. Stem Cells and Development. 29(1). 38–48. 12 indexed citations
11.
Koch, Thomas G., et al.. (2019). On the road to biomarkers: developing a robust system for miRNA evaluation in equine blood and synovial fluid. Osteoarthritis and Cartilage. 27. S110–S111. 2 indexed citations
12.
Villagómez, D.A.F., et al.. (2018). Cell Identity, Proliferation, and Cytogenetic Assessment of Equine Umbilical Cord Blood Mesenchymal Stromal Cells. Stem Cells and Development. 27(24). 1729–1738. 5 indexed citations
13.
Nagy, Kristina Vintersten, et al.. (2015). Generation, Characterization, and Multilineage Potency of Mesenchymal-Like Progenitors Derived from Equine Induced Pluripotent Stem Cells. Stem Cells and Development. 25(1). 80–89. 20 indexed citations
14.
Bienzle, Dorothee, et al.. (2015). Phenotypic and Immunomodulatory Properties of Equine Cord Blood-Derived Mesenchymal Stromal Cells. PLoS ONE. 10(4). e0122954–e0122954. 38 indexed citations
15.
Foldager, Casper Bindzus, et al.. (2013). Hypoxia enhances chondrogenic differentiation of human adipose tissue-derived stromal cells in scaffold-free and scaffold systems. Cell and Tissue Research. 355(1). 89–102. 26 indexed citations
16.
LaMarre, Jonathan, et al.. (2012). MicroRNA-140 Expression During Chondrogenic Differentiation of Equine Cord Blood-Derived Mesenchymal Stromal Cells. Stem Cells and Development. 22(8). 1288–1296. 41 indexed citations
17.
Koch, Thomas G., Preben D. Thomsen, & Dean H. Betts. (2009). Improved isolation protocol for equine cord blood-derived mesenchymal stromal cells. Cytotherapy. 11(4). 443–447. 39 indexed citations
18.
Rho, Gyu‐Jin, Gianfranco Coppola, R. Kasimanickam, et al.. (2007). Use of Somatic Cell Nuclear Transfer to Study Meiosis in Female Cattle Carrying A Sex-Dependent Fertility-Impairing X-Chromosome Abnormality. Cloning and Stem Cells. 9(1). 118–129. 10 indexed citations
19.
Koch, Thomas G., Xin Wen, & Dorothee Bienzle. (2006). Lymphoma, Erythrocytosis, and Tumor Erythropoietin Gene Expression in a Horse. Journal of Veterinary Internal Medicine. 20(5). 1251–1255. 11 indexed citations
20.
Koch, Thomas G., Xin Wen, & Dorothee Bienzle. (2006). Lymphoma, Erythrocytosis, and Tumor Erythropoietin Gene Expression in a Horse. Journal of Veterinary Internal Medicine. 20(5). 1251–1251. 10 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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