Thomas Blankenstein

13.7k total citations · 1 hit paper
209 papers, 10.6k citations indexed

About

Thomas Blankenstein is a scholar working on Immunology, Oncology and Molecular Biology. According to data from OpenAlex, Thomas Blankenstein has authored 209 papers receiving a total of 10.6k indexed citations (citations by other indexed papers that have themselves been cited), including 146 papers in Immunology, 104 papers in Oncology and 56 papers in Molecular Biology. Recurrent topics in Thomas Blankenstein's work include Immunotherapy and Immune Responses (104 papers), Immune Cell Function and Interaction (75 papers) and CAR-T cell therapy research (68 papers). Thomas Blankenstein is often cited by papers focused on Immunotherapy and Immune Responses (104 papers), Immune Cell Function and Interaction (75 papers) and CAR-T cell therapy research (68 papers). Thomas Blankenstein collaborates with scholars based in Germany, United States and Croatia. Thomas Blankenstein's co-authors include Zhihai Qin, Gerald Willimsky, Tibor Diamantstein, Thomas Schüler, Wolfgang Uckert, Thomas Kammertoens, Günther Richter, Mariette Mohaupt, Klaus Überla and Barbara Seliger and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and The Lancet.

In The Last Decade

Thomas Blankenstein

206 papers receiving 10.4k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

CCR7 Governs Skin Dendritic Cell Migration under Inflammatory and Steady-State Conditions

2004 804 citations Thomas Blankenstein et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Thomas Blankenstein Germany	55	6.6k	5.3k	2.8k	1.6k	896	209	10.6k
Paul M. Sondel United States	60	6.7k 1.0×	4.7k 0.9×	3.5k 1.3×	1.1k 0.7×	1.1k 1.2×	334	12.2k
Michael H. Kershaw Australia	50	6.4k 1.0×	7.6k 1.4×	2.8k 1.0×	2.0k 1.2×	452 0.5×	132	11.0k
Zhiya Yu United States	54	8.6k 1.3×	8.8k 1.7×	3.9k 1.4×	2.4k 1.5×	751 0.8×	95	14.2k
Antonella Stoppacciaro Italy	50	4.7k 0.7×	2.9k 0.5×	4.3k 1.5×	970 0.6×	1.3k 1.4×	160	10.8k
Giulio C. Spagnoli Switzerland	56	5.1k 0.8×	3.8k 0.7×	3.2k 1.1×	521 0.3×	680 0.8×	231	9.5k
Walter J. Storkus United States	66	12.9k 1.9×	6.3k 1.2×	5.6k 2.0×	1.9k 1.2×	794 0.9×	249	16.3k
Catia Traversari Italy	46	8.3k 1.2×	5.5k 1.0×	5.5k 1.9×	2.2k 1.4×	377 0.4×	109	12.7k
Per thor Straten Denmark	53	6.1k 0.9×	4.9k 0.9×	3.3k 1.2×	560 0.3×	551 0.6×	211	10.0k
Angel F. López Australia	63	6.9k 1.0×	2.9k 0.5×	3.6k 1.3×	922 0.6×	577 0.6×	275	13.7k
Shannon J. Turley United States	63	11.6k 1.7×	5.3k 1.0×	5.0k 1.8×	1.6k 1.0×	1.3k 1.5×	111	18.6k

Countries citing papers authored by Thomas Blankenstein

Since Specialization

Citations

This map shows the geographic impact of Thomas Blankenstein's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas Blankenstein with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas Blankenstein more than expected).

Fields of papers citing papers by Thomas Blankenstein

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas Blankenstein. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas Blankenstein. The network helps show where Thomas Blankenstein may publish in the future.

Co-authorship network of co-authors of Thomas Blankenstein

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas Blankenstein. A scholar is included among the top collaborators of Thomas Blankenstein based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas Blankenstein. Thomas Blankenstein is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Leisegang, Matthias, Cäcilia Freund, Gerald Willimsky, et al.. (2025). A CD22-specific T-cell receptor enables effective adoptive T-cell therapy for B-cell malignancies. Blood. 147(10). 1058–1069.

Papafotiou, George, et al.. (2023). Targeting the recurrent Rac1P29S neoepitope in melanoma with heterologous high-affinity T cell receptors. Frontiers in Immunology. 14. 1119498–1119498. 3 indexed citations

Willimsky, Gerald, Leo Hansmann, Thomas Blankenstein, et al.. (2022). Isolation of Neoantigen-Specific Human T Cell Receptors from Different Human and Murine Repertoires. Cancers. 14(7). 1842–1842. 2 indexed citations

Blankenstein, Thomas, et al.. (2021). The role of CD4 T cells in rejection of solid tumors. Current Opinion in Immunology. 74. 18–24. 83 indexed citations

Keller, Christin, Anja A. Kühl, Ana Textor, et al.. (2018). ERAP1-Dependent Antigen Cross-Presentation Determines Efficacy of Adoptive T-cell Therapy in Mice. Cancer Research. 78(12). 3243–3254. 11 indexed citations

Gavvovidis, Ioannis, Matthias Leisegang, Gerald Willimsky, et al.. (2018). Targeting Merkel Cell Carcinoma by Engineered T Cells Specific to T-Antigens of Merkel Cell Polyomavirus. Clinical Cancer Research. 24(15). 3644–3655. 24 indexed citations

Herrmann, Andreas, Christoph Lahtz, Toshikage Nagao, et al.. (2017). CTLA4 Promotes Tyk2-STAT3–Dependent B-cell Oncogenicity. Cancer Research. 77(18). 5118–5128. 38 indexed citations

Szyska, Martin, et al.. (2017). A Transgenic Dual-Luciferase Reporter Mouse for Longitudinal and Functional Monitoring of T Cells In Vivo. Cancer Immunology Research. 6(1). 110–120. 13 indexed citations

Miller, Natalie, Candice D. Church, Lichun Dong, et al.. (2017). Tumor-Infiltrating Merkel Cell Polyomavirus-Specific T Cells Are Diverse and Associated with Improved Patient Survival. Cancer Immunology Research. 5(2). 137–147. 71 indexed citations

10.

Textor, Ana, Cynthia Pérez, Anna Kruschinski, et al.. (2014). Efficacy of CAR T-cell Therapy in Large Tumors Relies upon Stromal Targeting by IFNγ. Cancer Research. 74(23). 6796–6805. 58 indexed citations

11.

Michelini, Rodrigo Hess, Teresa Manzo, Veronica Basso, et al.. (2013). Vaccine-Instructed Intratumoral IFN-γ Enables Regression of Autochthonous Mouse Prostate Cancer in Allogeneic T-Cell Transplantation. Cancer Research. 73(15). 4641–4652. 15 indexed citations

12.

Zhang, Jinhua, Lin Chen, Xiaoman Liu, et al.. (2013). Fibroblast-Specific Protein 1/S100A4–Positive Cells Prevent Carcinoma through Collagen Production and Encapsulation of Carcinogens. Cancer Research. 73(9). 2770–2781. 68 indexed citations

13.

Leisegang, Matthias, Adriana Turqueti‐Neves, Boris Engels, et al.. (2010). T-Cell Receptor Gene–Modified T Cells with Shared Renal Cell Carcinoma Specificity for Adoptive T-Cell Therapy. Clinical Cancer Research. 16(8). 2333–2343. 26 indexed citations

14.

Kruschinski, Anna, Andreas Moosmann, Isabel Poschke, et al.. (2008). Engineering antigen-specific primary human NK cells against HER-2 positive carcinomas. Proceedings of the National Academy of Sciences. 105(45). 17481–17486. 158 indexed citations

15.

Willimsky, Gerald, Melinda Czéh, Christoph Loddenkemper, et al.. (2008). Immunogenicity of premalignant lesions is the primary cause of general cytotoxic T lymphocyte unresponsiveness. The Journal of Experimental Medicine. 205(7). 1687–1700. 88 indexed citations

16.

Schmidt‐Kittler, Oleg, Thomas Ragg, Martin Granzow, et al.. (2003). From latent disseminated cells to overt metastasis: Genetic analysis of systemic breast cancer progression. Proceedings of the National Academy of Sciences. 100(13). 7737–7742. 493 indexed citations

17.

Uckert, Wolfgang, Thomas Blankenstein, Roger Nadrowitz, et al.. (2003). Overexpression of NPM–ALK induces different types of malignant lymphomas in IL-9 transgenic mice. Oncogene. 22(4). 517–527. 56 indexed citations

18.

Blankenstein, Thomas. (1999). Gene therapy : principles and applications. Birkhäuser-Verlag eBooks. 11 indexed citations

19.

Noffz, Gabriele, Zhihai Qin, Manfred Köpf, & Thomas Blankenstein. (1998). Neutrophils but Not Eosinophils Are Involved in Growth Suppression of IL-4-Secreting Tumors. The Journal of Immunology. 160(1). 345–350. 62 indexed citations

20.

Blankenstein, Thomas, Weiqun Li, Werner Müller, & Tibor Diamantstein. (1990). Retroviral interleukin 4 gene transfer into an interleukin 4‐dependent cell line results in autocrine growth but not in tumorigenicity. European Journal of Immunology. 20(4). 935–938. 26 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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