Thomas B. Kuhn

2.4k total citations
37 papers, 2.0k citations indexed

About

Thomas B. Kuhn is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Cell Biology. According to data from OpenAlex, Thomas B. Kuhn has authored 37 papers receiving a total of 2.0k indexed citations (citations by other indexed papers that have themselves been cited), including 19 papers in Molecular Biology, 13 papers in Cellular and Molecular Neuroscience and 12 papers in Cell Biology. Recurrent topics in Thomas B. Kuhn's work include Cellular Mechanics and Interactions (9 papers), Axon Guidance and Neuronal Signaling (8 papers) and Neuroinflammation and Neurodegeneration Mechanisms (7 papers). Thomas B. Kuhn is often cited by papers focused on Cellular Mechanics and Interactions (9 papers), Axon Guidance and Neuronal Signaling (8 papers) and Neuroinflammation and Neurodegeneration Mechanisms (7 papers). Thomas B. Kuhn collaborates with scholars based in United States, Switzerland and Jordan. Thomas B. Kuhn's co-authors include James R. Bamburg, Esther T. Stoeckli, P. Sonderegger, Michael D. Brown, Stanley B. Kater, Markus A. Rüegg, Fritz G. Rathjen, Laurie S. Minamide, Brian M. Barth and Jonathan A. Raper and has published in prestigious journals such as Neuron, Journal of Neuroscience and The Journal of Cell Biology.

In The Last Decade

Thomas B. Kuhn

37 papers receiving 2.0k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Thomas B. Kuhn United States 23 994 896 670 285 254 37 2.0k
Ralf S. Schmid United States 23 1.2k 1.2× 703 0.8× 498 0.7× 582 2.0× 192 0.8× 35 2.4k
Eve‐Ellen Govek United States 16 1.3k 1.4× 606 0.7× 681 1.0× 273 1.0× 153 0.6× 19 2.0k
Kevin C. Flynn United States 17 902 0.9× 1.1k 1.3× 927 1.4× 521 1.8× 117 0.5× 22 2.4k
Eldon E. Geisert United States 29 1.4k 1.4× 779 0.9× 306 0.5× 380 1.3× 210 0.8× 103 2.5k
Fumitoshi Irie Japan 28 1.6k 1.6× 1.3k 1.5× 1.3k 2.0× 362 1.3× 131 0.5× 52 3.1k
Vladislav V. Kiselyov Denmark 22 1.2k 1.2× 509 0.6× 407 0.6× 297 1.0× 113 0.4× 39 1.8k
Babette Fuss United States 27 1.1k 1.1× 751 0.8× 486 0.7× 526 1.8× 248 1.0× 46 2.1k
Michihiro Igarashi Japan 31 1.6k 1.6× 1.1k 1.2× 1.1k 1.7× 398 1.4× 99 0.4× 87 3.0k
Geoffrey C. Owens United States 30 1.9k 1.9× 975 1.1× 275 0.4× 445 1.6× 122 0.5× 62 2.9k
Kazunori Toida Japan 30 911 0.9× 970 1.1× 400 0.6× 481 1.7× 98 0.4× 64 2.9k

Countries citing papers authored by Thomas B. Kuhn

Since Specialization
Citations

This map shows the geographic impact of Thomas B. Kuhn's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas B. Kuhn with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas B. Kuhn more than expected).

Fields of papers citing papers by Thomas B. Kuhn

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas B. Kuhn. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas B. Kuhn. The network helps show where Thomas B. Kuhn may publish in the future.

Co-authorship network of co-authors of Thomas B. Kuhn

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas B. Kuhn. A scholar is included among the top collaborators of Thomas B. Kuhn based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas B. Kuhn. Thomas B. Kuhn is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Kuhn, Thomas B., Laurie S. Minamide, Lubna H. Tahtamouni, et al.. (2024). Chemokine Receptor Antagonists Prevent and Reverse Cofilin-Actin Rod Pathology and Protect Synapses in Cultured Rodent and Human iPSC-Derived Neurons. Biomedicines. 12(1). 93–93. 1 indexed citations
2.
Tahtamouni, Lubna H., Thomas B. Kuhn, Laurie S. Minamide, et al.. (2023). Characterization of a Human Neuronal Culture System for the Study of Cofilin–Actin Rod Pathology. Biomedicines. 11(11). 2942–2942. 2 indexed citations
3.
4.
Castellini, J. Margaret, Thomas B. Kuhn, Lorrie D. Rea, et al.. (2020). Assessing oxidative stress in Steller sea lions (Eumetopias jubatus): Associations with mercury and selenium concentrations. Comparative Biochemistry and Physiology Part C Toxicology & Pharmacology. 235. 108786–108786. 13 indexed citations
5.
Castellini, J. Margaret, et al.. (2018). Enhanced quantification of serum immunoglobulin G from a non-model wildlife species, the Steller sea lion (Eumetopias jubatus), using a protein A ELISA. Journal of Immunological Methods. 462. 42–47. 1 indexed citations
6.
7.
Minamide, Laurie S., Alisa E. Shaw, David R. Brown, et al.. (2014). Amyloid-β and Proinflammatory Cytokines Utilize a Prion Protein-Dependent Pathway to Activate NADPH Oxidase and Induce Cofilin-Actin Rods in Hippocampal Neurons. PLoS ONE. 9(4). e95995–e95995. 57 indexed citations
8.
Shaw, Alisa E., Chi W. Pak, Laurie S. Minamide, et al.. (2013). A Genetically Encoded Reporter for Real-Time Imaging of Cofilin-Actin Rods in Living Neurons. PLoS ONE. 8(12). e83609–e83609. 18 indexed citations
9.
Dunlap, Kriya L., et al.. (2012). A Nonpolar Blueberry Fraction Blunts NADPH Oxidase Activation in Neuronal Cells Exposed to Tumor Necrosis Factor-α. Oxidative Medicine and Cellular Longevity. 2012. 1–12. 22 indexed citations
10.
Barth, Brian M., et al.. (2011). Neutral sphingomyelinase activation precedes NADPH oxidase‐dependent damage in neurons exposed to the proinflammatory cytokine tumor necrosis factor‐α. Journal of Neuroscience Research. 90(1). 229–242. 44 indexed citations
11.
Barth, Brian M., et al.. (2011). Ceramide kinase regulates TNFα-stimulated NADPH oxidase activity and eicosanoid biosynthesis in neuroblastoma cells. Cellular Signalling. 24(6). 1126–1133. 22 indexed citations
12.
Barth, Brian M., Megan M. Young, Todd E. Fox, et al.. (2010). Inhibition of NADPH oxidase by glucosylceramide confers chemoresistance. Cancer Biology & Therapy. 10(11). 1126–1136. 25 indexed citations
13.
Barth, Brian M., et al.. (2009). Proinflammatory cytokines provoke oxidative damage to actin in neuronal cells mediated by Rac1 and NADPH oxidase. Molecular and Cellular Neuroscience. 41(2). 274–285. 52 indexed citations
14.
Kuhn, Thomas B. & James R. Bamburg. (2008). Tropomyosin and ADF/Cofilin as Collaborators and Competitors. Advances in experimental medicine and biology. 644. 232–249. 48 indexed citations
15.
Kuhn, Thomas B.. (2003). Growing and Working with Spinal Motor Neurons. Methods in cell biology. 71. 67–87. 25 indexed citations
16.
Kuhn, Thomas B., et al.. (2002). CNS neurons express two distinct plasma membrane electron transport systems implicated in neuronal viability. Journal of Neurochemistry. 83(3). 655–664. 20 indexed citations
17.
Drew, Kelly L., Margaret E. Rice, Thomas B. Kuhn, & Mark A. Smith. (2001). Neuroprotective adaptations in hibernation: therapeutic implications for ischemia-reperfusion, traumatic brain injury and neurodegenerative diseases. Free Radical Biology and Medicine. 31(5). 563–573. 120 indexed citations
18.
Kuhn, Thomas B., Michael D. Brown, & James R. Bamburg. (1998). Rac1-dependent actin filament organization in growth cones is necessary for ?1-integrin-mediated advance but not for growth on poly-D-lysine. Journal of Neurobiology. 37(4). 524–540. 77 indexed citations
19.
Kuhn, Thomas B., et al.. (1995). Laminin and fibronectin guideposts signal sustained but opposite effects to passing growth cones. Neuron. 14(2). 275–285. 107 indexed citations
20.
Stoeckli, Esther T., et al.. (1989). Identification of proteins secreted from axons of embryonic dorsal‐root‐ganglia neurons. European Journal of Biochemistry. 180(2). 249–258. 73 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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