Thomas A. Brodie

904 total citations
13 papers, 635 citations indexed

About

Thomas A. Brodie is a scholar working on Molecular Biology, Oncology and Immunology. According to data from OpenAlex, Thomas A. Brodie has authored 13 papers receiving a total of 635 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Molecular Biology, 3 papers in Oncology and 3 papers in Immunology. Recurrent topics in Thomas A. Brodie's work include Signaling Pathways in Disease (2 papers), Immune Response and Inflammation (2 papers) and Peroxisome Proliferator-Activated Receptors (2 papers). Thomas A. Brodie is often cited by papers focused on Signaling Pathways in Disease (2 papers), Immune Response and Inflammation (2 papers) and Peroxisome Proliferator-Activated Receptors (2 papers). Thomas A. Brodie collaborates with scholars based in United States, Germany and United Kingdom. Thomas A. Brodie's co-authors include Carl Blobel, Gisela Weskamp, Katia Manova, Hui Cai, Thomas Ludwig, Riccardo Chiusaroli, Lawrence Lum, Hans-Peter Hammes, Stephen A. Stimpson and Roland Baron and has published in prestigious journals such as Science, Molecular and Cellular Biology and Infection and Immunity.

In The Last Decade

Thomas A. Brodie

13 papers receiving 612 citations

Peers

Thomas A. Brodie
Krzysztof Darłak Poland
P.K. Chiang United States
Tamotsu Goto Japan
Kathleen Clancy United States
Joy L. Little United States
Satoko Kobayashi Japan
David Powers United States
Т. А. Смирнова Russia
Julia M. Ayala United States
Stephen P. Holly United States
Krzysztof Darłak Poland
Thomas A. Brodie
Citations per year, relative to Thomas A. Brodie Thomas A. Brodie (= 1×) peers Krzysztof Darłak

Countries citing papers authored by Thomas A. Brodie

Since Specialization
Citations

This map shows the geographic impact of Thomas A. Brodie's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas A. Brodie with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas A. Brodie more than expected).

Fields of papers citing papers by Thomas A. Brodie

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas A. Brodie. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas A. Brodie. The network helps show where Thomas A. Brodie may publish in the future.

Co-authorship network of co-authors of Thomas A. Brodie

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas A. Brodie. A scholar is included among the top collaborators of Thomas A. Brodie based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas A. Brodie. Thomas A. Brodie is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

13 of 13 papers shown
1.
Haggerty, Helen G., Jean G. Sathish, Carol Gleason, et al.. (2021). Thymic lymphomas in a 6-Month rasH2-Tg mouse carcinogenicity study with the RORγt Inverse Agonist, BMS-986251. Toxicological Sciences. 183(1). 93–104. 6 indexed citations
2.
Fletcher, Anthony M., Pierre Tellier, Julie Douville, et al.. (2019). Adverse vacuolation in multiple tissues in cynomolgus monkeys following repeat-dose administration of a PEGylated protein. Toxicology Letters. 317. 120–129. 17 indexed citations
3.
Shoieb, Ahmed, et al.. (2010). Renal Dysplasia in Beagle Dogs. Toxicologic Pathology. 38(7). 1051–1057. 8 indexed citations
4.
Conway, James G., Sarva M. Tadepalli, Peiyuan Lin, et al.. (2008). Effects of the cFMS Kinase Inhibitor 5-(3-Methoxy-4-((4-methoxybenzyl)oxy)benzyl)pyrimidine-2,4-diamine (GW2580) in Normal and Arthritic Rats. Journal of Pharmacology and Experimental Therapeutics. 326(1). 41–50. 48 indexed citations
5.
Faiola, Brenda, Richard A. Peterson, Thomas A. Brodie, et al.. (2008). PPAR alpha, more than PPAR delta, Mediates the Hepatic and Skeletal Muscle Alterations Induced by the PPAR Agonist GW0742. Toxicological Sciences. 105(2). 384–394. 29 indexed citations
6.
Böhm, Beate, Thomas Aigner, Barbara A. Roy, et al.. (2005). Homeostatic effects of the metalloproteinase disintegrin ADAM15 in degenerative cartilage remodeling. Arthritis & Rheumatism. 52(4). 1100–1109. 52 indexed citations
7.
8.
Horiuchi, Keisuke, Gisela Weskamp, Lawrence Lum, et al.. (2003). Potential Role for ADAM15 in Pathological Neovascularization in Mice. Molecular and Cellular Biology. 23(16). 5614–5624. 150 indexed citations
9.
Weskamp, Gisela, Hui Cai, Thomas A. Brodie, et al.. (2002). Mice Lacking the Metalloprotease-Disintegrin MDC9 (ADAM9) Have No Evident Major Abnormalities during Development or Adult Life. Molecular and Cellular Biology. 22(5). 1537–1544. 162 indexed citations
10.
Elangbam, Chandikumar S., Thomas A. Brodie, H. Roger Brown, et al.. (2002). Vascular Effects of GI262570X (PPAR-γ agonist) in the Brown Adipose Tissue of Han Wistar Rats: A Review of 1-month, 13-week, 27-week and 2-year Oral Toxicity Studies. Toxicologic Pathology. 30(4). 420–426. 8 indexed citations
11.
Li, Xiangli, Blair U. Bradford, Michael D. Wheeler, et al.. (2001). Dietary Glycine Prevents Peptidoglycan Polysaccharide-Induced Reactive Arthritis in the Rat: Role for Glycine-Gated Chloride Channel. Infection and Immunity. 69(9). 5883–5891. 75 indexed citations
12.
Zheng, Biao, Sara Brett, J P Tite, et al.. (1992). Galactose Oxidation in the Design of Immunogenic Vaccines. Science. 256(5063). 1560–1563. 45 indexed citations
13.
Brodie, Thomas A.. (1964). Attitude Toward School and Academic Achievement. The Personnel and Guidance Journal. 43(4). 375–378. 27 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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2026