Thomas A. Blair

481 total citations
11 papers, 332 citations indexed

About

Thomas A. Blair is a scholar working on Hematology, Molecular Biology and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Thomas A. Blair has authored 11 papers receiving a total of 332 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Hematology, 7 papers in Molecular Biology and 4 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Thomas A. Blair's work include Platelet Disorders and Treatments (8 papers), PI3K/AKT/mTOR signaling in cancer (4 papers) and Blood groups and transfusion (3 papers). Thomas A. Blair is often cited by papers focused on Platelet Disorders and Treatments (8 papers), PI3K/AKT/mTOR signaling in cancer (4 papers) and Blood groups and transfusion (3 papers). Thomas A. Blair collaborates with scholars based in United States and United Kingdom. Thomas A. Blair's co-authors include Andrew L. Frelinger, Steven L. Roberds, R P Hartshorne, Alan D. Michelson, Ingeborg Hers, Samantha Moore, Alastair W. Poole, Sunny S. Po, Paul B. Bennett and Michael M. Tamkun and has published in prestigious journals such as Blood, Scientific Reports and FEBS Letters.

In The Last Decade

Thomas A. Blair

11 papers receiving 329 citations

Peers

Thomas A. Blair

CCM

HEMAT

CMN

PRM

Saori Miwa Japan

Nacéra Saadane Canada

Jen‐Yea Chang United States

Victoria Panagiota Germany

Nipurna Jina United Kingdom

Mélanie Hoch Germany

Toshihiro Okuda Japan

N Beauchamp United Kingdom

Jie‐Yuan Jin China

Emiri Miura‐Yura Japan

Saori Miwa Japan

Thomas A. Blair

265 ×1.5

43 ×0.3

CCM

50 ×0.5

HEMAT

15 ×0.2

CMN

37 ×1.2

PRM

Citations per year, relative to Thomas A. Blair Thomas A. Blair (= 1×) peers Saori Miwa

Countries citing papers authored by Thomas A. Blair

Since Specialization

Citations

This map shows the geographic impact of Thomas A. Blair's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas A. Blair with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas A. Blair more than expected).

Fields of papers citing papers by Thomas A. Blair

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas A. Blair. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas A. Blair. The network helps show where Thomas A. Blair may publish in the future.

Co-authorship network of co-authors of Thomas A. Blair

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas A. Blair. A scholar is included among the top collaborators of Thomas A. Blair based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas A. Blair. Thomas A. Blair is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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11 of 11 papers shown

Moore, Samantha, et al.. (2020). Rapamycin restrains platelet procoagulant responses via FKBP-mediated protection of mitochondrial integrity. Biochemical Pharmacology. 177. 113975–113975. 17 indexed citations

Moore, Samantha, et al.. (2019). Critical roles for the phosphatidylinositide 3-kinase isoforms p110β and p110γ in thrombopoietin-mediated priming of platelet function. Scientific Reports. 9(1). 1468–1468. 12 indexed citations

Blair, Thomas A. & Andrew L. Frelinger. (2019). Platelet surface marker analysis by mass cytometry. Platelets. 31(5). 633–640. 33 indexed citations

Blair, Thomas A., Samantha Moore, Tony G. Walsh, et al.. (2018). Phosphoinositide 3-kinase p110α negatively regulates thrombopoietin-mediated platelet activation and thrombus formation. Cellular Signalling. 50. 111–120. 12 indexed citations

Blair, Thomas A., Alan D. Michelson, & Andrew L. Frelinger. (2018). Mass Cytometry Reveals Distinct Platelet Subtypes in Healthy Subjects and Novel Alterations in Surface Glycoproteins in Glanzmann Thrombasthenia. Scientific Reports. 8(1). 10300–10300. 55 indexed citations

Blair, Thomas A., Alan D. Michelson, & Andrew L. Frelinger. (2017). Mass Cytometry of Platelets: Simultaneous Functional and Phenotypic Analysis of Multiple Surface Markers in Healthy Subjects and Patients with Inherited Platelet Disorders Reveals Novel Platelet Sub-Populations. Blood. 130. 2311. 1 indexed citations

Moore, Samantha, Christopher Williams, Edward M. Brown, et al.. (2015). Loss of the insulin receptor in murine megakaryocytes/platelets causes thrombocytosis and alterations in IGF signalling. Cardiovascular Research. 107(1). 9–19. 43 indexed citations

Blair, Thomas A., Samantha Moore, Christopher M. Williams, et al.. (2014). Phosphoinositide 3-Kinases p110α and p110β Have Differential Roles in Insulin-Like Growth Factor-1–Mediated Akt Phosphorylation and Platelet Priming. Arteriosclerosis Thrombosis and Vascular Biology. 34(8). 1681–1688. 35 indexed citations

Roberds, Steven L., Sunny S. Po, Thomas A. Blair, et al.. (1993). Molecular Biology of the Voltage‐Gated Potassium Channels of the Cardiovascular System. Journal of Cardiovascular Electrophysiology. 4(1). 68–80. 56 indexed citations

10.

Blair, Thomas A., et al.. (1991). Functional characterization of RK5, a voltage‐gated K⁺ channel cloned from the rat cardiovascular system. FEBS Letters. 295(1-3). 211–213. 54 indexed citations

11.

Blair, Thomas A., Marco Parenti, & Thomas F. Murray. (1989). Development of pharmacological sensitivity to adenosine analogs in embryonic chick heart: role of A1 adenosine receptors and adenylyl cyclase inhibition.. Molecular Pharmacology. 35(5). 661–670. 14 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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