Thérèse J. Resink

9.4k total citations · 1 hit paper
174 papers, 7.7k citations indexed

About

Thérèse J. Resink is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Cell Biology. According to data from OpenAlex, Thérèse J. Resink has authored 174 papers receiving a total of 7.7k indexed citations (citations by other indexed papers that have themselves been cited), including 91 papers in Molecular Biology, 33 papers in Cardiology and Cardiovascular Medicine and 25 papers in Cell Biology. Recurrent topics in Thérèse J. Resink's work include Wnt/β-catenin signaling in development and cancer (30 papers), Protein Kinase Regulation and GTPase Signaling (18 papers) and Cell Adhesion Molecules Research (14 papers). Thérèse J. Resink is often cited by papers focused on Wnt/β-catenin signaling in development and cancer (30 papers), Protein Kinase Regulation and GTPase Signaling (18 papers) and Cell Adhesion Molecules Research (14 papers). Thérèse J. Resink collaborates with scholars based in Switzerland, Russia and United States. Thérèse J. Resink's co-authors include Fritz R. Bühler, Paul Erné, Timothy Scott‐Burden, Maria Philippova, Alfred W.A. Hahn, Josef Flammer, Manjunath B. Joshi, Brian A. Hemmings, Philip Cohen and Tkachuk Va and has published in prestigious journals such as JAMA, Journal of Biological Chemistry and Circulation.

In The Last Decade

Thérèse J. Resink

170 papers receiving 7.5k citations

Hit Papers

align trajectories sort by overperformance

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Smooth muscle cell-driven vascular diseases and molecular mechanisms of VSMC plasticity

2018 295 citations Agne Frismantiene, Maria Philippova et al. Cellular Signalling profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Thérèse J. Resink Switzerland	46	3.6k	1.7k	1.7k	1.1k	891	174	7.7k
Klaus Addicks Germany	50	3.7k 1.0×	645 0.4×	1.1k 0.7×	849 0.8×	966 1.1×	199	8.4k
Lucia Morbidelli Italy	44	3.8k 1.1×	694 0.4×	2.1k 1.2×	643 0.6×	756 0.8×	146	7.8k
Nina Wettschureck Germany	37	3.7k 1.0×	1.1k 0.6×	1.4k 0.8×	771 0.7×	597 0.7×	87	6.7k
Yoh Takuwa Japan	57	7.6k 2.1×	1.3k 0.7×	2.8k 1.7×	1.3k 1.2×	1.1k 1.2×	182	11.7k
Rudolf Kirchmair Austria	43	2.7k 0.7×	832 0.5×	657 0.4×	378 0.4×	1.1k 1.2×	114	6.1k
Ulf Eriksson Sweden	63	9.0k 2.5×	980 0.6×	1.0k 0.6×	1.3k 1.2×	1.3k 1.5×	149	14.3k
Josiah N. Wilcox United States	44	2.5k 0.7×	1.8k 1.1×	1.8k 1.1×	1.1k 1.0×	1.5k 1.7×	77	7.7k
Renhai Cao Sweden	50	5.7k 1.6×	591 0.3×	1.4k 0.8×	915 0.9×	1.1k 1.3×	60	10.1k
Ian Zachary United Kingdom	59	7.6k 2.1×	698 0.4×	667 0.4×	1.1k 1.0×	944 1.1×	138	11.3k
Czeslaw Radziejewski United States	26	6.6k 1.9×	1.1k 0.6×	510 0.3×	840 0.8×	746 0.8×	39	10.5k

Countries citing papers authored by Thérèse J. Resink

Since Specialization

Citations

This map shows the geographic impact of Thérèse J. Resink's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thérèse J. Resink with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thérèse J. Resink more than expected).

Fields of papers citing papers by Thérèse J. Resink

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thérèse J. Resink. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thérèse J. Resink. The network helps show where Thérèse J. Resink may publish in the future.

Co-authorship network of co-authors of Thérèse J. Resink

This figure shows the co-authorship network connecting the top 25 collaborators of Thérèse J. Resink. A scholar is included among the top collaborators of Thérèse J. Resink based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thérèse J. Resink. Thérèse J. Resink is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Schoenenberger, Andreas W., Franco Muggli, Gianfranco Parati, et al.. (2016). Protocol of the Swiss Longitudinal Cohort Study (SWICOS) in rural Switzerland. BMJ Open. 6(11). e013280–e013280. 8 indexed citations

Schoenenberger, Andreas W., Dennis Pfaff, Boris Dasen, et al.. (2015). Gender-Specific Associations between Circulating T-Cadherin and High Molecular Weight-Adiponectin in Patients with Stable Coronary Artery Disease. PLoS ONE. 10(6). e0131140–e0131140. 7 indexed citations

Ellmann, Lisa, Manjunath B. Joshi, Thérèse J. Resink, Anja‐Katrin Bosserhoff, & Silke Kuphal. (2012). BRN2 is a transcriptional repressor of CDH13 (T-cadherin) in melanoma cells. Laboratory Investigation. 92(12). 1788–1800. 24 indexed citations

Kyriakakis, Emmanouil, Maria Philippova, Dennis Pfaff, et al.. (2012). T-Cadherin Is an Auxiliary Negative Regulator of EGFR Pathway Activity in Cutaneous Squamous Cell Carcinoma: Impact on Cell Motility. Journal of Investigative Dermatology. 132(9). 2275–2285. 19 indexed citations

Joshi, Manjunath B., Danila Ivanov, Maria Philippova, Paul Erné, & Thérèse J. Resink. (2007). Integrin‐linked kinase is an essential mediator for T‐cadherin‐dependent signaling via Akt and GSK3β in endothelial cells. The FASEB Journal. 21(12). 3083–3095. 54 indexed citations

Bochkov, Valery N., Yelena Parfyonova, Tkachuk Va, et al.. (2002). Expression of adhesion molecule T-cadherin is increased during neointima formation in experimental restenosis. Histochemistry and Cell Biology. 118(4). 281–290. 64 indexed citations

Stambolsky, Dmitry, et al.. (1999). Identification of 130 kDa cell surface LDL-binding protein from smooth muscle cells as a partially processed T-cadherin precursor. Biochimica et Biophysica Acta (BBA) - Biomembranes. 1416(1-2). 155–160. 19 indexed citations

Zhu, Peili, et al.. (1999). Effect of Ox-LDL on endothelium-dependent response in pig ciliary artery: prevention by an ET(A) antagonist.. PubMed. 40(5). 1015–20. 9 indexed citations

Flammer, Josef, Ivan O. Haefliger, Selim Orgül, & Thérèse J. Resink. (1999). Vascular dysregulation: a principal risk factor for glaucomatous damage?. PubMed. 8(3). 212–9. 223 indexed citations

10.

Drobnik, Wolfgang, et al.. (1995). High-density lipoprotein and low-density lipoprotein-mediated signal transduction in cultured human skin fibroblasts. Cellular Signalling. 7(7). 695–707. 27 indexed citations

11.

Resink, Thérèse J., Fritz R. Bühler, Alfred Hahn, Valery N. Bochkov, & Tkachuk Va. (1994). INTERACTION BETWEEN PLASMA-LIPOPROTEINS AND VASCULAR SMOOTH-MUSCLE CELLS - HOW RELEVANT IS IT TO ARTERIAL-HYPERTENSION. Nutrition Metabolism and Cardiovascular Diseases. 4(3). 163–170. 4 indexed citations

12.

Hahn, Alfred W.A., et al.. (1993). Identification of a Fourth Angiotensin AT1 Receptor Subtype in Rat. Biochemical and Biophysical Research Communications. 192(3). 1260–1265. 21 indexed citations

13.

Resink, Thérèse J., et al.. (1992). Na-K pump and Na-K co-transport in cultured vascular smooth muscle cells from spontaneously hypertensive rats: baseline activity and regulation. Journal of Clinical Hypertension. 10. 733–740. 9 indexed citations

14.

Resink, Thérèse J., et al.. (1990). Stimulation of endothelin mRNA and secretion in rat vascular smooth muscle cells: a novel autocrine function.. PubMed. 1(9). 649–659. 232 indexed citations

15.

Resink, Thérèse J., et al.. (1990). Endothelin messenger RNA and receptors are differentially expressed in cultured human breast epithelial and stromal cells.. Journal of Clinical Investigation. 85(4). 1320–1323. 75 indexed citations

16.

Resink, Thérèse J., et al.. (1989). Differential stimulation of growth related metabolism in cultured smooth muscle cells from SHR and WKY rats by combinations of EGF and LDL. Biochemical and Biophysical Research Communications. 159(2). 624–632. 15 indexed citations

17.

Scott‐Burden, Timothy, Thérèse J. Resink, Ursula Baur, Maria Bürgin, & Fritz R. Bühler. (1988). Activation of S6 kinase in cultured vascular smooth muscle cells by submitogenic levels of thrombospondin. Biochemical and Biophysical Research Communications. 150(1). 278–286. 24 indexed citations

18.

Resink, Thérèse J., Timothy Scott‐Burden, Ursula Baur, Christopher Jones, & Fritz R. Bühler. (1988). Atrial natriuretic peptide induces breakdown of phosphatidylinositol phosphates in cultured vascular smooth‐muscle cells. European Journal of Biochemistry. 172(2). 499–505. 47 indexed citations

19.

Müller, Fabian, Paul Erné, A. E. G. Raine, et al.. (1986). [Secretion of atrial natriuretic peptide: relation to atrial pressure and systemic blood pressure].. PubMed. 116(46). 1610–2. 1 indexed citations

20.

Tung, H.Y. Lim, Thérèse J. Resink, Brian A. Hemmings, Shirish Shenolikar, & Philip Cohen. (1984). The catalytic subunits of protein phosphatase‐1 and protein phosphatase 2A are distinct gene products. European Journal of Biochemistry. 138(3). 635–641. 148 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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