Tetsuya Yamagata
- Molecular Biology top 10%
- Immunology top 2%
- Hematology top 2%
- Oncology top 5%
- Genetics top 10%
- Co-authors
- Kinuko MitaniHisamaru HiraiChristophe BenoıstDiane MathisJill SkepnerJianfei YangYoshio YazakiMark S. Sundrud
- Topics
- Acute Myeloid Leukemia Research (11 papers)T-cell and B-cell Immunology (9 papers)Immune Cell Function and Interaction (7 papers)
- Cited by
- HematologyImmunologyGenetics
- Journals
- Proceedings of the National Academy of SciencesJournal of Biological ChemistryNature Medicine
- Partner nations
- JapanUnited StatesUnited Kingdom
In The Last Decade
Tetsuya Yamagata
57 papers receiving 2.6k citations
Peers
Comparison fields: 5 of 103
- Molecular Biology 1.2k
- Immunology 1.1k
- Hematology 618
- Oncology 499
- Genetics 266
Countries citing papers authored by Tetsuya Yamagata
This map shows the geographic impact of Tetsuya Yamagata's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tetsuya Yamagata with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tetsuya Yamagata more than expected).
Fields of papers citing papers by Tetsuya Yamagata
This network shows the impact of papers produced by Tetsuya Yamagata. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tetsuya Yamagata. The network helps show where Tetsuya Yamagata may publish in the future.
Co-authorship network of co-authors of Tetsuya Yamagata
This figure shows the co-authorship network connecting the top 25 collaborators of Tetsuya Yamagata. A scholar is included among the top collaborators of Tetsuya Yamagata based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tetsuya Yamagata. Tetsuya Yamagata is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 281 | |
| 2 | 1 | |
| 3 | 2 | |
| 4 | 2 | |
| 5 | 9 | |
| 6 | 12 | |
| 7 | 33 | |
| 8 | 49 | |
| 9 | 7 | |
| 10 | 134 | |
| 11 | 464 | |
| 12 | 89 | |
| 13 | 22 | |
| 14 | Activated caspase-9 and-3 were involved in apoptosis of myogenenic cell lines of MELAS. | 1 |
| 15 | 114 | |
| 16 | 22 | |
| 17 | 103 | |
| 18 | 16 | |
| 19 | 7 | |
| 20 | 2 |
About Tetsuya Yamagata
Tetsuya Yamagata is a scholar working on Hematology, Immunology and Oncology, having authored 58 papers that have together received 2.7k indexed citations. Recurring topics across this work include Acute Myeloid Leukemia Research (11 papers), T-cell and B-cell Immunology (9 papers) and Immune Cell Function and Interaction (7 papers). The work is most often cited by research in Hematology (618 citations), Immunology (1.1k citations) and Genetics (228 citations). Tetsuya Yamagata has collaborated with scholars based in Japan, United States and United Kingdom. Frequent co-authors include Kinuko Mitani, Hisamaru Hirai, Christophe Benoıst, Diane Mathis, Jill Skepner, Jianfei Yang, Yoshio Yazaki, Mark S. Sundrud, Takashi Saito and Takashi Asai. Their work appears in journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Nature Medicine.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.