Teruhiko Inoue
- Molecular Biology top 10%
- Oncology top 10%
- Cancer Research top 5%
- Cell Biology top 5%
- Cardiology and Cardiovascular Medicine top 10%
- Co-authors
- Kazuki NabeshimaYoshiya ShimaoMasashi KoonoTetsuro SameshimaHiroaki KataokaFumitoshi AsaiTaketoshi OgawaHiroyuki Koike
- Topics
- Protease and Inhibitor Mechanisms (8 papers)Wnt/β-catenin signaling in development and cancer (6 papers)Cellular Mechanics and Interactions (5 papers)
- Partner nations
- JapanUnited StatesIndia
In The Last Decade
Teruhiko Inoue
55 papers receiving 1.8k citations
Peers
Comparison fields: 5 of 127
- Molecular Biology 776
- Oncology 442
- Cancer Research 310
- Cell Biology 272
- Cardiology and Cardiovascular Medicine 178
Countries citing papers authored by Teruhiko Inoue
This map shows the geographic impact of Teruhiko Inoue's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Teruhiko Inoue with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Teruhiko Inoue more than expected).
Fields of papers citing papers by Teruhiko Inoue
This network shows the impact of papers produced by Teruhiko Inoue. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Teruhiko Inoue. The network helps show where Teruhiko Inoue may publish in the future.
Co-authorship network of co-authors of Teruhiko Inoue
This figure shows the co-authorship network connecting the top 25 collaborators of Teruhiko Inoue. A scholar is included among the top collaborators of Teruhiko Inoue based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Teruhiko Inoue. Teruhiko Inoue is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 5 | |
| 2 | 65 | |
| 3 | 8 | |
| 4 | 22 | |
| 5 | 19 | |
| 6 | 22 | |
| 7 | 106 | |
| 8 | 7 | |
| 9 | 17 | |
| 10 | 24 | |
| 11 | 71 | |
| 12 | 13 | |
| 13 | 49 | |
| 14 | 153 | |
| 15 | 10 | |
| 16 | 7 | |
| 17 | 18 | |
| 18 | 照射を受けたレシピエント内に静止して、二次レシピエント内に24日脾臓コロニーを産生したHY-110WGA+LIN-C-KIT骨髄細胞 | 2 |
| 19 | 2 | |
| 20 | [Pathological and clinical study on Kienboeck's disease]. | 1 |
About Teruhiko Inoue
Teruhiko Inoue is a scholar working on Immunology and Allergy, Cancer Research and Hepatology, having authored 56 papers that have together received 1.8k indexed citations. Recurring topics across this work include Protease and Inhibitor Mechanisms (8 papers), Wnt/β-catenin signaling in development and cancer (6 papers) and Cellular Mechanics and Interactions (5 papers). The work is most often cited by research in Immunology and Allergy (165 citations), Cancer Research (310 citations) and Cell Biology (272 citations). Teruhiko Inoue has collaborated with scholars based in Japan, United States and India. Frequent co-authors include Kazuki Nabeshima, Yoshiya Shimao, Masashi Koono, Tetsuro Sameshima, Hiroaki Kataoka, Fumitoshi Asai, Taketoshi Ogawa, Hiroyuki Koike, Atsuhiro Sugidachi and Takahiko Ishiguro. Their work appears in journals such as Nucleic Acids Research, The Journal of Physical Chemistry and FEBS Letters.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.