Tatsuya Ishizuka

677 total citations
17 papers, 564 citations indexed

About

Tatsuya Ishizuka is a scholar working on Endocrinology, Diabetes and Metabolism, Endocrine and Autonomic Systems and Physiology. According to data from OpenAlex, Tatsuya Ishizuka has authored 17 papers receiving a total of 564 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Endocrinology, Diabetes and Metabolism, 6 papers in Endocrine and Autonomic Systems and 5 papers in Physiology. Recurrent topics in Tatsuya Ishizuka's work include Regulation of Appetite and Obesity (6 papers), Adipose Tissue and Metabolism (5 papers) and Pituitary Gland Disorders and Treatments (3 papers). Tatsuya Ishizuka is often cited by papers focused on Regulation of Appetite and Obesity (6 papers), Adipose Tissue and Metabolism (5 papers) and Pituitary Gland Disorders and Treatments (3 papers). Tatsuya Ishizuka collaborates with scholars based in Japan, United States and China. Tatsuya Ishizuka's co-authors include Jacob E. Friedman, Richard J. Koletsky, Paul Ernsberger, Miyao Matsubara, Jianhua Shao, Koji Nakagawa, Sha Liu, Hiroshi Yamashita, Mariko Mita and Minoru Tomita and has published in prestigious journals such as Journal of Biological Chemistry, Endocrinology and Journal of Nutrition.

In The Last Decade

Tatsuya Ishizuka

17 papers receiving 555 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Tatsuya Ishizuka Japan 11 215 171 124 117 112 17 564
Akinori Kogure Japan 14 105 0.5× 341 2.0× 86 0.7× 128 1.1× 117 1.0× 26 589
Eishi Miki Japan 10 370 1.7× 131 0.8× 71 0.6× 119 1.0× 64 0.6× 16 620
Hiroaki Kakizawa Japan 7 86 0.4× 120 0.7× 64 0.5× 73 0.6× 123 1.1× 11 389
Tsunekazu Umekawa Japan 15 79 0.4× 393 2.3× 84 0.7× 132 1.1× 127 1.1× 25 600
Olga Szabó United States 13 142 0.7× 201 1.2× 127 1.0× 152 1.3× 26 0.2× 31 520
Anja Böhm Germany 16 174 0.8× 350 2.0× 50 0.4× 292 2.5× 192 1.7× 31 763
Behzad Varamini United States 6 49 0.2× 119 0.7× 45 0.4× 140 1.2× 117 1.0× 8 699
C. Di Filippo Italy 11 43 0.2× 74 0.4× 25 0.2× 105 0.9× 37 0.3× 14 406
Todd M. Suter United States 9 270 1.3× 283 1.7× 242 2.0× 258 2.2× 187 1.7× 12 740
Fiona Hanner United States 7 86 0.4× 128 0.7× 59 0.5× 433 3.7× 21 0.2× 8 625

Countries citing papers authored by Tatsuya Ishizuka

Since Specialization
Citations

This map shows the geographic impact of Tatsuya Ishizuka's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tatsuya Ishizuka with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tatsuya Ishizuka more than expected).

Fields of papers citing papers by Tatsuya Ishizuka

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tatsuya Ishizuka. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tatsuya Ishizuka. The network helps show where Tatsuya Ishizuka may publish in the future.

Co-authorship network of co-authors of Tatsuya Ishizuka

This figure shows the co-authorship network connecting the top 25 collaborators of Tatsuya Ishizuka. A scholar is included among the top collaborators of Tatsuya Ishizuka based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tatsuya Ishizuka. Tatsuya Ishizuka is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
Huseynova, Tukezban, et al.. (2014). Comparative Study of 2 Types of Implantable Collamer Lenses, 1 With and 1 Without a Central Artificial Hole. American Journal of Ophthalmology. 157(6). 1136–1143. 64 indexed citations
2.
Friedman, Jacob E., et al.. (2009). Metabolic consequences of a nonsense mutation in the leptin receptor gene (fa k) in the obeses spontaneously hypertensive Koletsky rat (SHROB). Experimental and Clinical Endocrinology & Diabetes. 105(S 03). 82–84. 1 indexed citations
3.
Yamashita, Hiroshi, Jianhua Shao, Tatsuya Ishizuka, et al.. (2001). Leptin Administration Prevents Spontaneous Gestational Diabetes in Heterozygous Leprdb/+Mice: Effects on Placental Leptin and Fetal Growth1. Endocrinology. 142(7). 2888–2897. 92 indexed citations
4.
Shao, Jianhua, et al.. (2000). Vanadate enhances but does not normalize glucose transport and insulin receptor phosphorylation in skeletal muscle from obese women with gestational diabetes mellitus. American Journal of Obstetrics and Gynecology. 183(5). 1263–1270. 12 indexed citations
5.
Kubo, Mitsumasa, Chikara Shimizu, Hiromichi Kijima, et al.. (2000). Alternate Exon in the 5'-Untranslated Region of the Human ACTH Receptor Gene.. Endocrine Journal. 47(5). 543–548. 3 indexed citations
6.
Ernsberger, Paul, et al.. (1999). Mechanisms of Antihyperglycemic Effects of Moxonidine in the Obese Spontaneously Hypertensive Koletsky Rat (SHROB). Journal of Pharmacology and Experimental Therapeutics. 288(1). 139–147. 45 indexed citations
7.
Ishizuka, Tatsuya, et al.. (1998). Phenotypic Consequences of a Nonsense Mutation in the Leptin Receptor Gene (fak) in Obese Spontaneously Hypertensive Koletsky Rats (SHROB). Journal of Nutrition. 128(12). 2299–2306. 48 indexed citations
8.
Shimizu, Chikara, Mitsumasa Kubo, Tsuyoshi Matsumura, et al.. (1997). Genomic organization of the mouse adrenocorticotropin receptor. Gene. 188(1). 17–21. 12 indexed citations
9.
Friedman, Jacob E., et al.. (1997). Reduced insulin receptor signaling in the obese spontaneously hypertensive Koletsky rat. American Journal of Physiology-Endocrinology and Metabolism. 273(5). E1014–E1023. 64 indexed citations
10.
Friedman, Jacob E., Yang Sun, Tatsuya Ishizuka, et al.. (1997). Phosphoenolpyruvate Carboxykinase (GTP) Gene Transcription and Hyperglycemia Are Regulated by Glucocorticoids in Genetically Obesedb/db Transgenic Mice. Journal of Biological Chemistry. 272(50). 31475–31481. 94 indexed citations
11.
Kijima, Hiromichi, Mitsumasa Kubo, Tatsuya Ishizuka, Mitsuaki Kakinuma, & Takao Koike. (1996). A novel missense mutation in the thyroid hormone receptor β gene in a kindred with resistance to thyroid hormone. Human Genetics. 97(3). 407–408. 4 indexed citations
12.
13.
Kubo, Mitsumasa, Tatsuya Ishizuka, Hiromichi Kijima, Mitsuaki Kakinuma, & Takao Koike. (1995). Cloning of a mouse adrenocorticotropin receptor-encoding gene. Gene. 153(2). 279–280. 2 indexed citations
14.
Nakagawa, Koji, Tatsuya Ishizuka, Chikara Shimizu, Yoshito Ito, & Ichiji Wakabayashi. (1992). Increased hypothalamic somatostatin mRNA following dexamethasone administration in rats. European Journal of Endocrinology. 127(5). 416–419. 23 indexed citations
15.
Nakagawa, Koji, et al.. (1987). Dichotomic action of glucocorticoids on growth hormone secretion. European Journal of Endocrinology. 116(2). 165–171. 63 indexed citations
16.
Nakagawa, Koji, et al.. (1987). Biphasic Effects of Dexamethsone on Growth Hormone Release in vitro.. Endocrinologia Japonica. 34(6). 947–953. 3 indexed citations
17.
Kubo, Mitsumasa, et al.. (1985). In vivo and in vitro ACTH response to ovine corticotropin-releasing factor in a bronchial carcinoid from a patient with ectopic ACTH syndrome.. Endocrinologia Japonica. 32(4). 577–581. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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