This map shows the geographic impact of Tamra Goodrow's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tamra Goodrow with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tamra Goodrow more than expected).
This network shows the impact of papers produced by Tamra Goodrow. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tamra Goodrow. The network helps show where Tamra Goodrow may publish in the future.
Co-authorship network of co-authors of Tamra Goodrow
This figure shows the co-authorship network connecting the top 25 collaborators of Tamra Goodrow.
A scholar is included among the top collaborators of Tamra Goodrow based on the total number of
citations received by their joint publications. Widths of edges
represent the number of papers authors have co-authored together.
Node borders
signify the number of papers an author published with Tamra Goodrow. Tamra Goodrow is excluded from
the visualization to improve readability, since they are connected to all nodes in the network.
Mahloogi, Haleh, Anatilde M. González-Guerrico, Ricardo López de Cicco, et al.. (2003). Overexpression of the calcium sensor visinin-like protein-1 leads to a cAMP-mediated decrease of in vivo and in vitro growth and invasiveness of squamous cell carcinoma cells.. PubMed. 63(16). 4997–5004.50 indexed citations
Jin, Fuzi, et al.. (1997). The FHIT gene product is highly expressed in the cytoplasm of renal tubular epithelium and is down-regulated in kidney cancers.. PubMed. 151(6). 1541–7.20 indexed citations
5.
Zhang, Shi Yu, et al.. (1997). Increased expression of G1 cyclins and cyclin-dependent kinases during tumor progression of chemically induced mouse skin neoplasms.. PubMed. 18(3). 142–52.33 indexed citations
Goodrow, Tamra, et al.. (1997). Expression of PACE4 in chemically induced carcinomas is associated with spindle cell tumor conversion and increased invasive ability.. PubMed. 57(23). 5226–31.43 indexed citations
Huang, Lingyi, et al.. (1996). Deletion and mutation analyses of the P16/MTS-1 tumor suppressor gene in human ductal pancreatic cancer reveals a higher frequency of abnormalities in tumor-derived cell lines than in primary ductal adenocarcinomas.. PubMed. 56(5). 1137–41.69 indexed citations
Takagi, Hitoshi, Richard Sharp, Tamra Goodrow, et al.. (1992). Molecular and genetic analysis of liver oncogenesis in transforming growth factor alpha transgenic mice.. PubMed. 52(19). 5171–7.71 indexed citations
16.
Conti, C J, A. Bianchi, Nora M. Navone, et al.. (1992). Tumor suppressor genes in squamous cell carcinoma.. PubMed. 376. 85–101.3 indexed citations
17.
Ruggeri, Bruce, Jorge Caamaño, Tamra Goodrow, et al.. (1991). Alterations of the p53 tumor suppressor gene during mouse skin tumor progression.. PubMed. 51(24). 6615–21.103 indexed citations
18.
Candrian, Urs, Ming You, Tamra Goodrow, et al.. (1991). Activation of protooncogenes in spontaneously occurring non-liver tumors from C57BL/6 x C3H F1 mice.. PubMed. 51(4). 1148–53.22 indexed citations
19.
Goodrow, Tamra, Steven H. Reynolds, R Maronpot, & M W Anderson. (1990). Activation of K-ras by codon 13 mutations in C57BL/6 X C3H F1 mouse tumors induced by exposure to 1,3-butadiene.. PubMed. 50(15). 4818–23.58 indexed citations
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive
bibliographic database. While OpenAlex provides broad and valuable coverage of the global
research landscape, it—like all bibliographic datasets—has inherent limitations. These include
incomplete records, variations in author disambiguation, differences in journal indexing, and
delays in data updates. As a result, some metrics and network relationships displayed in
Rankless may not fully capture the entirety of a scholar's output or impact.