Takehiro Kamo

748 total citations
8 papers, 501 citations indexed

About

Takehiro Kamo is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Infectious Diseases. According to data from OpenAlex, Takehiro Kamo has authored 8 papers receiving a total of 501 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Molecular Biology, 4 papers in Cardiology and Cardiovascular Medicine and 3 papers in Infectious Diseases. Recurrent topics in Takehiro Kamo's work include Clostridium difficile and Clostridium perfringens research (3 papers), Gut microbiota and health (3 papers) and Genetics, Aging, and Longevity in Model Organisms (2 papers). Takehiro Kamo is often cited by papers focused on Clostridium difficile and Clostridium perfringens research (3 papers), Gut microbiota and health (3 papers) and Genetics, Aging, and Longevity in Model Organisms (2 papers). Takehiro Kamo collaborates with scholars based in Japan. Takehiro Kamo's co-authors include Issei Komuro, Hiroshi Akazawa, Jun‐ichi Suzuki, Yu Shimizu, Hiroki Yagi, Atsuhiko T. Naito, Jun-ichi Suzuki, Hidetoshi Morita, Masahira Hattori and Qing Liu and has published in prestigious journals such as Circulation, PLoS ONE and Circulation Research.

In The Last Decade

Takehiro Kamo

8 papers receiving 486 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Takehiro Kamo Japan 7 323 178 167 66 54 8 501
Minghui Bao China 10 378 1.2× 157 0.9× 140 0.8× 56 0.8× 82 1.5× 22 581
Chaowei Hu China 10 253 0.8× 149 0.8× 107 0.6× 69 1.0× 35 0.6× 30 442
Xinyu Ge China 9 388 1.2× 170 1.0× 95 0.6× 25 0.4× 52 1.0× 26 601
Leticia Coín‐Aragüez Spain 14 277 0.9× 223 1.3× 60 0.4× 41 0.6× 142 2.6× 20 603
Dillon K. Jarrell United States 9 249 0.8× 90 0.5× 66 0.4× 22 0.3× 107 2.0× 20 439
Rikeish R. Muralitharan Australia 10 260 0.8× 190 1.1× 38 0.2× 17 0.3× 68 1.3× 20 474
Ting‐Wei Lee Taiwan 14 223 0.7× 116 0.7× 180 1.1× 28 0.4× 80 1.5× 29 552
M Eder Austria 11 180 0.6× 135 0.8× 56 0.3× 13 0.2× 48 0.9× 19 438
Arnela Saljic Denmark 12 207 0.6× 58 0.3× 370 2.2× 21 0.3× 51 0.9× 43 553
Adriana Ortega‐Hernández Spain 10 133 0.4× 62 0.3× 76 0.5× 21 0.3× 46 0.9× 22 300

Countries citing papers authored by Takehiro Kamo

Since Specialization
Citations

This map shows the geographic impact of Takehiro Kamo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Takehiro Kamo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Takehiro Kamo more than expected).

Fields of papers citing papers by Takehiro Kamo

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Takehiro Kamo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Takehiro Kamo. The network helps show where Takehiro Kamo may publish in the future.

Co-authorship network of co-authors of Takehiro Kamo

This figure shows the co-authorship network connecting the top 25 collaborators of Takehiro Kamo. A scholar is included among the top collaborators of Takehiro Kamo based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Takehiro Kamo. Takehiro Kamo is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

8 of 8 papers shown
1.
Kamo, Takehiro. (2020). Abstract 12596: Manipulation of Gut Microbiota Influences Myocardial Mass in Mice. Circulation. 142(Suppl_3). 1 indexed citations
2.
Kamo, Takehiro, Hiroshi Akazawa, Wataru Suda, et al.. (2017). Dysbiosis and compositional alterations with aging in the gut microbiota of patients with heart failure. PLoS ONE. 12(3). e0174099–e0174099. 197 indexed citations
3.
Kamo, Takehiro, Hiroshi Akazawa, Jun-ichi Suzuki, & Issei Komuro. (2017). Novel Concept of a Heart-Gut Axis in the Pathophysiology of Heart Failure. Korean Circulation Journal. 47(5). 663–663. 51 indexed citations
4.
Kamo, Takehiro, Hiroshi Akazawa, Jun‐ichi Suzuki, & Issei Komuro. (2016). Roles of renin-angiotensin system and Wnt pathway in aging-related phenotypes. Inflammation and Regeneration. 36(1). 12–12. 14 indexed citations
5.
Yano, Masamichi, Hiroshi Akazawa, Toru Oka, et al.. (2015). Monocyte-derived extracellular Nampt-dependent biosynthesis of NAD+ protects the heart against pressure overload. Scientific Reports. 5(1). 15857–15857. 26 indexed citations
6.
Akazawa, Hiroshi, Rie Yamamoto, Masamichi Yano, et al.. (2015). Angiotensin II receptor blockade promotes repair of skeletal muscle through down-regulation of aging-promoting C1q expression. Scientific Reports. 5(1). 14453–14453. 43 indexed citations
7.
Kamo, Takehiro, Hiroshi Akazawa, & Issei Komuro. (2015). Pleiotropic Effects of Angiotensin II Receptor Signaling in Cardiovascular Homeostasis and Aging. International Heart Journal. 56(3). 249–254. 39 indexed citations
8.
Kamo, Takehiro, Hiroshi Akazawa, & Issei Komuro. (2015). Cardiac Nonmyocytes in the Hub of Cardiac Hypertrophy. Circulation Research. 117(1). 89–98. 130 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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