Takayuki Oka
- Molecular Biology
- Surgery
- Cellular and Molecular Neuroscience top 10%
- Cell Biology top 10%
- Ophthalmology top 5%
- Co-authors
- Mitsuyoshi AzumaThomas R. ShearerMichihiro SuganoTakemasa TsujiYukuo YoshidaKazuo KobayashiYoshiyuki TamadaTatsuya Takagi
- Topics
- Ion channel regulation and function (8 papers)Cardiac electrophysiology and arrhythmias (7 papers)Calpain Protease Function and Regulation (6 papers)
- Journals
- Applied and Environmental MicrobiologyThe Journal of Physical Chemistry BThe Journal of Physical Chemistry C
- Partner nations
- JapanUnited StatesHong Kong
In The Last Decade
Takayuki Oka
40 papers receiving 849 citations
Peers
Comparison fields: 5 of 101
- Molecular Biology 444
- Surgery 170
- Cellular and Molecular Neuroscience 159
- Cell Biology 152
- Ophthalmology 148
Countries citing papers authored by Takayuki Oka
This map shows the geographic impact of Takayuki Oka's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Takayuki Oka with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Takayuki Oka more than expected).
Fields of papers citing papers by Takayuki Oka
This network shows the impact of papers produced by Takayuki Oka. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Takayuki Oka. The network helps show where Takayuki Oka may publish in the future.
Co-authorship network of co-authors of Takayuki Oka
This figure shows the co-authorship network connecting the top 25 collaborators of Takayuki Oka. A scholar is included among the top collaborators of Takayuki Oka based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Takayuki Oka. Takayuki Oka is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 0 | |
| 3 | 0 | |
| 4 | 3 | |
| 5 | 9 | |
| 6 | 1 | |
| 7 | 1 | |
| 8 | 3 | |
| 9 | 5 | |
| 10 | 40 | |
| 11 | 9 | |
| 12 | 16 | |
| 13 | 40 | |
| 14 | 57 | |
| 15 | 10 | |
| 16 | 50 | |
| 17 | 33 | |
| 18 | 93 | |
| 19 | 2 | |
| 20 | 103 |
About Takayuki Oka
Takayuki Oka is a scholar working on Ophthalmology, Cellular and Molecular Neuroscience and Cell Biology, having authored 42 papers that have together received 878 indexed citations. Recurring topics across this work include Ion channel regulation and function (8 papers), Cardiac electrophysiology and arrhythmias (7 papers) and Calpain Protease Function and Regulation (6 papers). The work is most often cited by research in Ophthalmology (148 citations), Cell Biology (152 citations) and Cellular and Molecular Neuroscience (159 citations). Takayuki Oka has collaborated with scholars based in Japan, United States and Hong Kong. Frequent co-authors include Mitsuyoshi Azuma, Thomas R. Shearer, Michihiro Sugano, Takemasa Tsuji, Yukuo Yoshida, Kazuo Kobayashi, Yoshiyuki Tamada, Tatsuya Takagi, Fumiaki Ito and Mitsunori Waki. Their work appears in journals such as Applied and Environmental Microbiology, The Journal of Physical Chemistry B and The Journal of Physical Chemistry C.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.