Taha Rashid

1.0k citations
27 papers · 682 indexed · h-index 15

Taha Rashid

27 papers receiving 638 citations

Peers

Taha Rashid
Comparison fields: 5 of 95
  • Rheumatology 291
  • Immunology 184
  • Gastroenterology 39
  • Molecular Medicine 33
  • Endocrinology 33
Replace Astrid Konrad with:
Astrid Konrad Germany
Scott A. Siegel United States
Frederico R. C. Costa Brazil
A. Lombardi Italy
Ingrid A. Schrijver Netherlands
Sylvain Normand France
Anja Schirbel Germany
Kenneth J. Gorelick United States
E. L. Krawitt United States
Marie‐Françoise Odou France
Taha Rashid relative to Astrid Konrad Germany Astrid Konrad's profile →
Citations per field
00.5×3.5×
Astrid Konrad · 1×
Citations per year

Countries citing papers authored by Taha Rashid

Since Specialization
Citations

This map shows the geographic impact of Taha Rashid's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Taha Rashid with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Taha Rashid more than expected).

Fields of papers citing papers by Taha Rashid

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Taha Rashid. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Taha Rashid. The network helps show where Taha Rashid may publish in the future.

Co-authorship network

The 10 scholars most cited alongside Taha Rashid, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Taha Rashid Line = papers co-authored together Taha Rashid links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 20167
2 20149
3 201349
4 201334
5 20125
6
Gut-mediated and HLA-B27-associated arthritis: an emphasis on ankylosing spondylitis and Crohn's disease with a proposal for the use of new treatment.
201119
7 200961
8 200915
9 20084
10 200712
11 20067
12 200634
13 20063
14 200653
15 200676
16 200619
17 200513
18 200514
19 20045
20 200323

About Taha Rashid

Taha Rashid is a scholar working on Rheumatology, Immunology and Complementary and Manual Therapy, having authored 27 papers that have together received 682 indexed citations. Recurring topics across this work include Spondyloarthritis Studies and Treatments (11 papers), Rheumatoid Arthritis Research and Therapies (10 papers), Systemic Lupus Erythematosus Research (7 papers), Inflammatory Bowel Disease (6 papers), Psoriasis: Treatment and Pathogenesis (6 papers), Prion Diseases and Protein Misfolding (3 papers), Multiple Sclerosis Research Studies (3 papers) and Dermatology and Skin Diseases (3 papers). The work is most often cited by research in Rheumatology (291 citations), Immunology (184 citations) and Gastroenterology (39 citations). Taha Rashid has collaborated with scholars based in United Kingdom, Australia and United States. Frequent co-authors include Alan Ebringer, Clyde Wilson, Harmale Tiwana, Mark D. Fielder, Sukhvinder S. Bansal, Edward J. Thompson, Rich Boden, Camille Ettelaie, Allan Binder and Leena Paimela. Their work appears in journals such as SHILAP Revista de lepidopterología, Annals of the New York Academy of Sciences and Autoimmunity Reviews.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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