Tadashi Kagimoto
- Immunology top 10%
- Molecular Biology
- Hematology top 5%
- Nephrology top 5%
- Genetics top 10%
- Co-authors
- K. UyedaHideki NakakumaKiyoshi TakatsukiShoichi NagakuraKentaro HorikawaNorihiro IwamotoTatsuya KawaguchiKosaku Uyeda
- Topics
- Complement system in diseases (13 papers)Adenosine and Purinergic Signaling (9 papers)Glycosylation and Glycoproteins Research (6 papers)
- Cited by
- NephrologyImmunologyHematology
- Journals
- Journal of Biological ChemistryJournal of Clinical InvestigationThe Journal of Experimental Medicine
- Partner nations
- JapanUnited States
In The Last Decade
Tadashi Kagimoto
39 papers receiving 745 citations
Peers
Comparison fields: 5 of 75
- Immunology 390
- Molecular Biology 269
- Hematology 185
- Nephrology 134
- Genetics 116
Countries citing papers authored by Tadashi Kagimoto
This map shows the geographic impact of Tadashi Kagimoto's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tadashi Kagimoto with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tadashi Kagimoto more than expected).
Fields of papers citing papers by Tadashi Kagimoto
This network shows the impact of papers produced by Tadashi Kagimoto. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tadashi Kagimoto. The network helps show where Tadashi Kagimoto may publish in the future.
Co-authorship network of co-authors of Tadashi Kagimoto
This figure shows the co-authorship network connecting the top 25 collaborators of Tadashi Kagimoto. A scholar is included among the top collaborators of Tadashi Kagimoto based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tadashi Kagimoto. Tadashi Kagimoto is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 21 | |
| 2 | 41 | |
| 3 | 7 | |
| 4 | 17 | |
| 5 | 31 | |
| 6 | 30 | |
| 7 | Altered expression of protein kinase C in adult T-cell leukemia cells. | 6 |
| 8 | 8 | |
| 9 | 26 | |
| 10 | 12 | |
| 11 | 10 | |
| 12 | 2 | |
| 13 | 33 | |
| 14 | 23 | |
| 15 | 37 | |
| 16 | 4 | |
| 17 | 16 | |
| 18 | 38 | |
| 19 | 7 | |
| 20 | 6 |
About Tadashi Kagimoto
Tadashi Kagimoto is a scholar working on Physiology, Immunology and Nephrology, having authored 39 papers that have together received 789 indexed citations. Recurring topics across this work include Complement system in diseases (13 papers), Adenosine and Purinergic Signaling (9 papers) and Glycosylation and Glycoproteins Research (6 papers). The work is most often cited by research in Nephrology (134 citations), Immunology (390 citations) and Hematology (185 citations). Tadashi Kagimoto has collaborated with scholars based in Japan and United States. Frequent co-authors include K. Uyeda, Hideki Nakakuma, Kiyoshi Takatsuki, Shoichi Nagakura, Kentaro Horikawa, Norihiro Iwamoto, Tatsuya Kawaguchi, Kosaku Uyeda, Michihiro Hidaka and Akhteruzzaman Molla. Their work appears in journals such as Journal of Biological Chemistry, Journal of Clinical Investigation and The Journal of Experimental Medicine.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.