Sven Potelle

508 total citations
12 papers, 418 citations indexed

About

Sven Potelle is a scholar working on Molecular Biology, Cell Biology and Physiology. According to data from OpenAlex, Sven Potelle has authored 12 papers receiving a total of 418 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 8 papers in Cell Biology and 4 papers in Physiology. Recurrent topics in Sven Potelle's work include Cellular transport and secretion (8 papers), Glycosylation and Glycoproteins Research (7 papers) and Erythrocyte Function and Pathophysiology (3 papers). Sven Potelle is often cited by papers focused on Cellular transport and secretion (8 papers), Glycosylation and Glycoproteins Research (7 papers) and Erythrocyte Function and Pathophysiology (3 papers). Sven Potelle collaborates with scholars based in France, Belgium and United States. Sven Potelle's co-authors include François Foulquier, André Klein, Gert Matthijs, Willy Morelle, Marie‐Ange Krzewinski‐Recchi, Sandrine Duvet, Geoffroy de Bettignies, Dorothée Vicogne, Dominique Legrand and Jaak Jaeken and has published in prestigious journals such as Journal of Biological Chemistry, The Journal of Clinical Endocrinology & Metabolism and Chemical Communications.

In The Last Decade

Sven Potelle

12 papers receiving 417 citations

Peers

Sven Potelle
Sven Potelle
Citations per year, relative to Sven Potelle Sven Potelle (= 1×) peers Didier Demaegd

Countries citing papers authored by Sven Potelle

Since Specialization
Citations

This map shows the geographic impact of Sven Potelle's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sven Potelle with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sven Potelle more than expected).

Fields of papers citing papers by Sven Potelle

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sven Potelle. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sven Potelle. The network helps show where Sven Potelle may publish in the future.

Co-authorship network of co-authors of Sven Potelle

This figure shows the co-authorship network connecting the top 25 collaborators of Sven Potelle. A scholar is included among the top collaborators of Sven Potelle based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sven Potelle. Sven Potelle is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

12 of 12 papers shown
1.
Morava, Éva, Ulrich A. Schatz, Pernille Mathiesen Tørring, et al.. (2021). Impaired glucose-1,6-biphosphate production due to bi-allelic PGM2L1 mutations is associated with a neurodevelopmental disorder. The American Journal of Human Genetics. 108(6). 1151–1160. 11 indexed citations
2.
Potelle, Sven, et al.. (2021). The promiscuous binding pocket of SLC35A1 ensures redundant transport of CDP-ribitol to the Golgi. Journal of Biological Chemistry. 296. 100789–100789. 12 indexed citations
3.
Houdou, Marine, Sven Potelle, Geoffroy de Bettignies, et al.. (2019). Dissection of TMEM165 function in Golgi glycosylation and its Mn2+ sensitivity. Biochimie. 165. 123–130. 24 indexed citations
4.
Houdou, Marine, Anne Garat, Sandrine Duvet, et al.. (2018). Involvement of thapsigargin– and cyclopiazonic acid–sensitive pumps in the rescue of TMEM165‐associated glycosylation defects by Mn 2+. The FASEB Journal. 33(2). 2669–2679. 26 indexed citations
5.
Morelle, Willy, Sven Potelle, Peter Witters, et al.. (2017). Galactose Supplementation in Patients With TMEM165-CDG Rescues the Glycosylation Defects. The Journal of Clinical Endocrinology & Metabolism. 102(4). 1375–1386. 68 indexed citations
6.
Yu, Shin‐Yi, Marine Houdou, Geoffroy de Bettignies, et al.. (2017). Investigating the function of Gdt1p in yeast Golgi glycosylation. Biochimica et Biophysica Acta (BBA) - General Subjects. 1862(3). 394–402. 27 indexed citations
7.
Krzewinski‐Recchi, Marie‐Ange, Sven Potelle, Anne‐Marie Mir, et al.. (2017). Evidence for splice transcript variants of TMEM165, a gene involved in CDG. Biochimica et Biophysica Acta (BBA) - General Subjects. 1861(4). 737–748. 7 indexed citations
8.
Potelle, Sven, Leslie K. Climer, Sandrine Duvet, et al.. (2017). Manganese-induced turnover of TMEM165. Biochemical Journal. 474(9). 1481–1493. 47 indexed citations
10.
Potelle, Sven, Willy Morelle, Sandrine Duvet, et al.. (2016). Glycosylation abnormalities in Gdt1p/TMEM165 deficient cells result from a defect in Golgi manganese homeostasis. Human Molecular Genetics. 25(8). 1489–1500. 90 indexed citations
11.
Lion, Cédric, Dorothée Vicogne, Thierry Levade, et al.. (2015). A sequential bioorthogonal dual strategy: ManNAl and SiaNAl as distinct tools to unravel sialic acid metabolic pathways. Chemical Communications. 52(11). 2318–2321. 39 indexed citations
12.
Potelle, Sven, André Klein, & François Foulquier. (2015). Golgi post‐translational modifications and associated diseases. Journal of Inherited Metabolic Disease. 38(4). 741–751. 32 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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