Sumihisa Imakado

1.6k total citations · 1 hit paper
30 papers, 1.1k citations indexed

About

Sumihisa Imakado is a scholar working on Dermatology, Epidemiology and Molecular Biology. According to data from OpenAlex, Sumihisa Imakado has authored 30 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Dermatology, 11 papers in Epidemiology and 8 papers in Molecular Biology. Recurrent topics in Sumihisa Imakado's work include Cancer and Skin Lesions (6 papers), Nail Diseases and Treatments (4 papers) and Skin and Cellular Biology Research (3 papers). Sumihisa Imakado is often cited by papers focused on Cancer and Skin Lesions (6 papers), Nail Diseases and Treatments (4 papers) and Skin and Cellular Biology Research (3 papers). Sumihisa Imakado collaborates with scholars based in Japan, United States and Australia. Sumihisa Imakado's co-authors include Fujio Otsuka, Dennis R. Roop, Shuhei Noda, Junko Wakabayashi, Ken Itoh, Takanori Harada, Tomoe KOTSUJI, Satoru Takahashi, Nobunao Wakabayashi and James Douglas Engel and has published in prestigious journals such as Nature Genetics, Genes & Development and SHILAP Revista de lepidopterología.

In The Last Decade

Sumihisa Imakado

26 papers receiving 1.1k citations

Hit Papers

Keap1-null mutation leads to postnatal lethality due to c... 2003 2026 2010 2018 2003 200 400 600
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. Keap1-null mutation leads to postnatal lethality due to constitutive Nrf2 activation
    2003 728 citations Nobunao Wakabayashi, Ken Itoh et al. Nature Genetics profile →

Peers

Sumihisa Imakado
Dean Ng United States
Luowei Li United States
S. Arase Japan
Tobias A. Beyer Switzerland
Xiuwei Tang United States
David K. Bol United States
France Bertrand France
Eui Man Jeong South Korea
BI Zhi-gang China
Mingliang Chen China
Dean Ng United States
Sumihisa Imakado
Citations per year, relative to Sumihisa Imakado Sumihisa Imakado (= 1×) peers Dean Ng

Countries citing papers authored by Sumihisa Imakado

Since Specialization
Citations

This map shows the geographic impact of Sumihisa Imakado's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sumihisa Imakado with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sumihisa Imakado more than expected).

Fields of papers citing papers by Sumihisa Imakado

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sumihisa Imakado. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sumihisa Imakado. The network helps show where Sumihisa Imakado may publish in the future.

Co-authorship network of co-authors of Sumihisa Imakado

This figure shows the co-authorship network connecting the top 25 collaborators of Sumihisa Imakado. A scholar is included among the top collaborators of Sumihisa Imakado based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sumihisa Imakado. Sumihisa Imakado is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Takeichi, Takuya, Yasushi Ogawa, Sumihisa Imakado, et al.. (2024). Updated mutational spectrum and genotype–phenotype correlations in ichthyosis patients with ABCA12 pathogenic variants. Experimental Dermatology. 33(4). e15072–e15072. 1 indexed citations
2.
Matsumoto, Hideyuki, Akihiko Mitsutake, Junko Yoshimura, et al.. (2018). Toxic Epidermal Necrolysis in a Patient with Autoimmune Limbic Encephalitis with Anti-Glutamate Receptor Antibodies. Case Reports in Neurology. 10(2). 207–212.
3.
Imakado, Sumihisa & Ryo Masuda. (2012). A Case of Carcinoma of the Male Breast Mimicking a Mucinous Carcinoma of the Skin. SHILAP Revista de lepidopterología. 2(3). e61–e61.
4.
Imakado, Sumihisa, et al.. (2012). Metachronous Merkel Cell Carcinoma on Both Cheeks. Acta Dermato Venereologica. 92(1). 54–56. 2 indexed citations
5.
Imakado, Sumihisa, Hiroyuki Sato, & Kazutoshi Hamada. (2008). Two cases of subungual melanoma in situ. The Journal of Dermatology. 35(11). 754–758. 16 indexed citations
6.
Kagami, Shinji, Hidehisa Saeki, Yoshihiro Kuwano, Sumihisa Imakado, & Kunihiko Tamaki. (2006). A probable case of Muckle–Wells syndrome. The Journal of Dermatology. 33(2). 118–121. 10 indexed citations
7.
Kawachi, Yasuhiro, Xuezhu Xu, Eiko Ichikawa, Sumihisa Imakado, & Fujio Otsuka. (2003). Expression of angiogenic factors in neurofibromas. Experimental Dermatology. 12(4). 412–417. 30 indexed citations
8.
Wakabayashi, Nobunao, Ken Itoh, Junko Wakabayashi, et al.. (2003). Keap1-null mutation leads to postnatal lethality due to constitutive Nrf2 activation. Nature Genetics. 35(3). 238–245. 728 indexed citations breakdown →
9.
Otsuka, Fujio, et al.. (2002). Absence of Lisch Nodules in Sporadic Neurofibromatosis Type 1 May Reflect Somatic Mosaicism—Reply. Archives of Dermatology. 138(6). 840–840. 1 indexed citations
10.
Imakado, Sumihisa, et al.. (2002). PDGF‐BB Induces MAP Kinase Phosphorylation and VEGF Expression in Neurofibroma‐Derived Cultured Cells from Patients with Neurofibromatosis 1. The Journal of Dermatology. 29(11). 713–717. 18 indexed citations
11.
Ichikawa, Eiko, et al.. (2001). Sweet's Disease Associated with IgA Myeloma.. The Nishinihon Journal of Dermatology. 63(4). 368–370. 1 indexed citations
12.
Ichikawa, Eriko, et al.. (2001). Hereditary complement (C9) deficiency associated with dermatomyositis. British Journal of Dermatology. 144(5). 1080–1083. 14 indexed citations
13.
Banno, Tomohiro, et al.. (2001). Psoriasis vulgaris and acute guttate psoriasis in a family. International Journal of Dermatology. 40(4). 285–287. 5 indexed citations
14.
Imakado, Sumihisa, et al.. (2000). Sarcoidosis showing a livedo reticularis-like eruption. European Journal of Dermatology. 7(2). 112–114. 1 indexed citations
15.
Fujisawa, Yasuhiro, Yoshiyuki Ishii, Tomohiro Banno, et al.. (2000). Two Cases of Adult Lichen Nitidus.. The Nishinihon Journal of Dermatology. 62(2). 179–181. 1 indexed citations
16.
Ichikawa, Eriko, et al.. (2000). Eccrine syringofibroadenoma in a patient with a burn scar ulcer. British Journal of Dermatology. 143(3). 591–594. 23 indexed citations
17.
Wojcik, Sonja M., Sumihisa Imakado, Mary A. Longley, et al.. (1999). Expression of MK6a dominant-negative and C-terminal mutant transgenes in mice has distinct phenotypic consequences in the epidermis and hair follicle. Differentiation. 65(2). 97–112. 28 indexed citations
18.
Wertz, Philip W., et al.. (1997). Inhibition of Retinoid Signaling in Transgenic Mice Alters Lipid Processing and Disrupts Epidermal Barrier Function. Molecular Endocrinology. 11(6). 792–800. 33 indexed citations
19.
Imakado, Sumihisa, Jackie R. Bickenbach, Donnie S. Bundman, et al.. (1995). Targeting expression of a dominant-negative retinoic acid receptor mutant in the epidermis of transgenic mice results in loss of barrier function.. Genes & Development. 9(3). 317–329. 120 indexed citations
20.
Imakado, Sumihisa, Satoshi Koike, Shigeru Kondo, Masaharu Sakai, & Masami Muramatsu. (1991). The N-Terminal Transactivation Domain of Rat Estrogen Receptor Is Localized in a Hydrophobic Domain of Eighty Amino Acids. The Journal of Biochemistry. 109(5). 684–689. 11 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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