Stefanie Kurtz

823 total citations
10 papers, 658 citations indexed

About

Stefanie Kurtz is a scholar working on Molecular Biology, Pulmonary and Respiratory Medicine and Genetics. According to data from OpenAlex, Stefanie Kurtz has authored 10 papers receiving a total of 658 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 4 papers in Pulmonary and Respiratory Medicine and 3 papers in Genetics. Recurrent topics in Stefanie Kurtz's work include Cell death mechanisms and regulation (5 papers), Occupational and environmental lung diseases (4 papers) and Genetic and Clinical Aspects of Sex Determination and Chromosomal Abnormalities (3 papers). Stefanie Kurtz is often cited by papers focused on Cell death mechanisms and regulation (5 papers), Occupational and environmental lung diseases (4 papers) and Genetic and Clinical Aspects of Sex Determination and Chromosomal Abnormalities (3 papers). Stefanie Kurtz collaborates with scholars based in Switzerland, Germany and United States. Stefanie Kurtz's co-authors include Rolf A. Stahel, Uwe Zangemeister‐Wittke, Emanuela Felley‐Bosco, Colette Bigosch, C Ludwig, Daniela Kandioler, Larisa Belyanskaya, Annemarie Ziegler, Ana Paula Simões‐Wüst and Sally Hopkins-Donaldson and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Cell Death and Differentiation and International Journal of Cancer.

In The Last Decade

Stefanie Kurtz

10 papers receiving 648 citations

Peers

Countries citing papers authored by Stefanie Kurtz

Since Specialization

Citations

This map shows the geographic impact of Stefanie Kurtz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Stefanie Kurtz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Stefanie Kurtz more than expected).

Fields of papers citing papers by Stefanie Kurtz

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Stefanie Kurtz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Stefanie Kurtz. The network helps show where Stefanie Kurtz may publish in the future.

Co-authorship network of co-authors of Stefanie Kurtz

This figure shows the co-authorship network connecting the top 25 collaborators of Stefanie Kurtz. A scholar is included among the top collaborators of Stefanie Kurtz based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Stefanie Kurtz. Stefanie Kurtz is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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10 of 10 papers shown

#	Work	Indexed citations
1	Generation of Pigs that Produce Single Sex Progeny 2022 ·Methods in molecular biology ·Björn Petersen, Stefanie Kurtz	1
2	Knockout of the HMG domain of the porcine SRY gene causes sex reversal in gene-edited pigs 2020 ·Proceedings of the National Academy of Sciences ·Stefanie Kurtz, Andrea Lucas‐Hahn, Brigitte Schlegelberger, Gudrun Göhring, Heiner Niemann, Thomas C. Mettenleiter, Björn Petersen	30
3	Pre-determination of sex in pigs by application of CRISPR/Cas system for genome editing 2019 ·Theriogenology ·Stefanie Kurtz, Björn Petersen	20
4	Functional inactivation of NF2/merlin in human mesothelioma 2008 ·Lung Cancer ·(unknown), Isabelle Opitz, Stefanie Kurtz, Walter Weder, Rolf A. Stahel, Emanuela Felley‐Bosco	113
5	TRAIL-induced survival and proliferation of SCLC cells is mediated by ERK and dependent on TRAIL-R2/DR5 expression in the absence of caspase-8 2008 ·Lung Cancer ·Larisa Belyanskaya, Algirdas Ziogas, Sally Hopkins-Donaldson, Stefanie Kurtz, Hans‐Uwe Simon, Rolf A. Stahel, Uwe Zangemeister‐Wittke	37
6	Human agonistic TRAIL receptor antibodies Mapatumumab and Lexatumumab induce apoptosis in malignant mesothelioma and act synergistically with cisplatin 2007 ·Molecular Cancer ·Larisa Belyanskaya, Thomas M. Marti, Sally Hopkins-Donaldson, Stefanie Kurtz, Emanuela Felley‐Bosco, Rolf A. Stahel	59
7	p53-Induced Apoptosis Occurs in the Absence of p14ARF in Malignant Pleural Mesothelioma 2006 ·Neoplasia ·Sally Hopkins-Donaldson, (unknown), Ana Paula Simões‐Wüst, Brigitte Sigrist, Stefanie Kurtz, Uwe Zangemeister‐Wittke, Rolf A. Stahel	36
8	Cisplatin activates Akt in small cell lung cancer cells and attenuates apoptosis by survivin upregulation 2005 ·International Journal of Cancer ·Larisa Belyanskaya, (unknown), Stefanie Kurtz, Ana Paula Simões‐Wüst, Shída Yousefi, Hans‐Uwe Simon, Rolf A. Stahel, Uwe Zangemeister‐Wittke	89
9	Induction of apoptosis and chemosensitization of mesothelioma cells by Bcl‐2 and Bcl‐xL antisense treatment 2003 ·International Journal of Cancer ·(unknown), Richard Cathomas, Ana Paula Simões‐Wüst, Stefanie Kurtz, (unknown), Rolf A. Stahel, Uwe Zangemeister‐Wittke	62
10	Silencing of death receptor and caspase-8 expression in small cell lung carcinoma cell lines and tumors by DNA methylation 2003 ·Cell Death and Differentiation ·(unknown), Annemarie Ziegler, Stefanie Kurtz, Colette Bigosch, Daniela Kandioler, C Ludwig, Uwe Zangemeister‐Wittke, Rolf A. Stahel	211

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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