Stefan Pukatzki

7.2k total citations · 3 hit papers
47 papers, 5.6k citations indexed

About

Stefan Pukatzki is a scholar working on Endocrinology, Molecular Medicine and Immunology. According to data from OpenAlex, Stefan Pukatzki has authored 47 papers receiving a total of 5.6k indexed citations (citations by other indexed papers that have themselves been cited), including 39 papers in Endocrinology, 20 papers in Molecular Medicine and 16 papers in Immunology. Recurrent topics in Stefan Pukatzki's work include Vibrio bacteria research studies (38 papers), Antibiotic Resistance in Bacteria (20 papers) and Escherichia coli research studies (16 papers). Stefan Pukatzki is often cited by papers focused on Vibrio bacteria research studies (38 papers), Antibiotic Resistance in Bacteria (20 papers) and Escherichia coli research studies (16 papers). Stefan Pukatzki collaborates with scholars based in United States, Canada and Germany. Stefan Pukatzki's co-authors include John J. Mekalanos, Sarah T. Miyata, Amy T., Daniel Unterweger, Maya Kitaoka, Benjamin Kostiuk, Andrew T. Revel, John F. Heidelberg, Bryan Krastins and David Sarracino and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Stefan Pukatzki

46 papers receiving 5.6k citations

Hit Papers

align trajectories sort by overperformance

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Identification of a conserved bacterial protein secretion system in Vibrio cholerae using the Dictyostelium host model system

2006 894 citations Stefan Pukatzki, Amy T. et al. Proceedings of the National Academy of Sciences profile →
Type VI secretion system translocates a phage tail spike-like protein into target cells where it cross-links actin

2007 586 citations Stefan Pukatzki, Amy T. et al. Proceedings of the National Academy of Sciences profile →
Type VI secretion apparatus and phage tail-associated protein complexes share a common evolutionary origin

2009 501 citations P.G. Leiman, Marek Basler et al. Proceedings of the National Academy of Sciences profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Stefan Pukatzki United States	31	4.2k	2.1k	1.9k	1.1k	854	47	5.6k
Ina Attrée France	35	1.6k 0.4×	1.3k 0.6×	2.0k 1.0×	1.3k 1.2×	381 0.4×	94	3.7k
Timothy L. Yahr United States	39	1.8k 0.4×	1.7k 0.8×	2.9k 1.5×	1.9k 1.7×	356 0.4×	78	4.3k
E. Fidelma Boyd United States	42	2.8k 0.7×	574 0.3×	2.0k 1.0×	785 0.7×	1.3k 1.6×	97	5.1k
Karen Skorupski United States	29	2.5k 0.6×	499 0.2×	1.7k 0.9×	885 0.8×	1.1k 1.3×	41	3.4k
Wanyin Deng Canada	40	3.0k 0.7×	554 0.3×	1.7k 0.9×	1.6k 1.4×	629 0.7×	72	5.0k
Robert K. Taylor New Zealand	27	2.2k 0.5×	423 0.2×	1.2k 0.6×	801 0.7×	906 1.1×	73	3.9k
Laure Journet France	32	1.9k 0.4×	683 0.3×	1.3k 0.7×	1.4k 1.3×	188 0.2×	46	3.2k
Francisco García‐del Portillo Spain	45	2.0k 0.5×	614 0.3×	2.2k 1.1×	1.5k 1.3×	434 0.5×	133	5.8k
Paul A. Manning Australia	47	3.8k 0.9×	712 0.3×	2.1k 1.1×	1.9k 1.8×	1.3k 1.5×	178	6.3k
Christopher S. Hayes United States	39	1.9k 0.4×	661 0.3×	2.4k 1.2×	1.9k 1.7×	148 0.2×	86	4.2k

Countries citing papers authored by Stefan Pukatzki

Since Specialization

Citations

This map shows the geographic impact of Stefan Pukatzki's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Stefan Pukatzki with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Stefan Pukatzki more than expected).

Fields of papers citing papers by Stefan Pukatzki

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Stefan Pukatzki. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Stefan Pukatzki. The network helps show where Stefan Pukatzki may publish in the future.

Co-authorship network of co-authors of Stefan Pukatzki

This figure shows the co-authorship network connecting the top 25 collaborators of Stefan Pukatzki. A scholar is included among the top collaborators of Stefan Pukatzki based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Stefan Pukatzki. Stefan Pukatzki is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Kostiuk, Benjamin, et al.. (2023). Vibrio cholerae Alkalizes Its Environment via Citrate Metabolism to Inhibit Enteric Growth In Vitro. Microbiology Spectrum. 11(2). e0491722–e0491722. 3 indexed citations

Almagro‐Moreno, Salvador & Stefan Pukatzki. (2023). Vibrio spp. Infections. Advances in experimental medicine and biology. 4 indexed citations

Santoriello, Francis J. & Stefan Pukatzki. (2023). Type VI Secretion Systems: Environmental and Intra-host Competition of Vibrio cholerae. Advances in experimental medicine and biology. 1404. 41–63. 4 indexed citations

Teschler, Jennifer K., Hannah Jeckel, Praveen K. Singh, et al.. (2022). VxrB Influences Antagonism within Biofilms by Controlling Competition through Extracellular Matrix Production and Type 6 Secretion. mBio. 13(4). e0188522–e0188522. 13 indexed citations

Kostiuk, Benjamin, et al.. (2021). Type VI secretion system mutations reduced competitive fitness of classical Vibrio cholerae biotype. Nature Communications. 12(1). 6457–6457. 16 indexed citations

Fast, David E., Kristina Petkau, Meghan Ferguson, et al.. (2020). Vibrio cholerae-Symbiont Interactions Inhibit Intestinal Repair in Drosophila. Cell Reports. 30(4). 1088–1100.e5. 30 indexed citations

Fast, David E., Benjamin Kostiuk, Edan Foley, & Stefan Pukatzki. (2018). Commensal pathogen competition impacts host viability. Proceedings of the National Academy of Sciences. 115(27). 7099–7104. 60 indexed citations

Unterweger, Daniel, Benjamin Kostiuk, & Stefan Pukatzki. (2016). Adaptor Proteins of Type VI Secretion System Effectors. Trends in Microbiology. 25(1). 8–10. 51 indexed citations

Unterweger, Daniel, et al.. (2015). Chimeric adaptor proteins translocate diverse type VI secretion system effectors in Vibrio cholerae. The EMBO Journal. 34(16). 2198–2210. 107 indexed citations

10.

Unterweger, Daniel, Sarah T. Miyata, Teresa M. Brooks, et al.. (2014). The Vibrio cholerae type VI secretion system employs diverse effector modules for intraspecific competition. Nature Communications. 5(1). 3549–3549. 152 indexed citations

11.

Brooks, Teresa M., et al.. (2013). Lytic Activity of the Vibrio cholerae Type VI Secretion Toxin VgrG-3 Is Inhibited by the Antitoxin TsaB. Journal of Biological Chemistry. 288(11). 7618–7625. 135 indexed citations

12.

Miyata, Sarah T., Daniel Unterweger, Sydney P. Rudko, & Stefan Pukatzki. (2013). Dual Expression Profile of Type VI Secretion System Immunity Genes Protects Pandemic Vibrio cholerae. PLoS Pathogens. 9(12). e1003752–e1003752. 115 indexed citations

13.

Miyata, Sarah T., Maya Kitaoka, Łukasz Wieteska, et al.. (2010). The Vibrio Cholerae Type VI Secretion System: Evaluating its Role in the Human Disease Cholera. Frontiers in Microbiology. 1. 117–117. 33 indexed citations

14.

Leiman, P.G., Marek Basler, U.A. Ramagopal, et al.. (2009). Type VI secretion apparatus and phage tail-associated protein complexes share a common evolutionary origin. Proceedings of the National Academy of Sciences. 106(11). 4154–4159. 501 indexed citations breakdown →

15.

T., Amy, et al.. (2009). Translocation of a Vibrio cholerae Type VI Secretion Effector Requires Bacterial Endocytosis by Host Cells. Cell Host & Microbe. 5(3). 234–243. 227 indexed citations

16.

Pukatzki, Stefan, et al.. (2007). Type VI secretion system translocates a phage tail spike-like protein into target cells where it cross-links actin. Proceedings of the National Academy of Sciences. 104(39). 15508–15513. 586 indexed citations breakdown →

17.

Smith, Michael G., Tara A. Gianoulis, Stefan Pukatzki, et al.. (2007). New insights into Acinetobacter baumannii pathogenesis revealed by high-density pyrosequencing and transposon mutagenesis. Genes & Development. 21(5). 601–614. 397 indexed citations

18.

Pukatzki, Stefan, Amy T., Bryan Krastins, et al.. (2006). Identification of a conserved bacterial protein secretion system in Vibrio cholerae using the Dictyostelium host model system. Proceedings of the National Academy of Sciences. 103(5). 1528–1533. 894 indexed citations breakdown →

19.

Raskin, David M., R. Seshadri, Stefan Pukatzki, & John J. Mekalanos. (2006). Bacterial Genomics and Pathogen Evolution. Cell. 124(4). 703–714. 101 indexed citations

20.

Pukatzki, Stefan, Richard H. Kessin, & John J. Mekalanos. (2002). The human pathogen Pseudomonas aeruginosa utilizes conserved virulence pathways to infect the social amoeba Dictyostelium discoideum. Proceedings of the National Academy of Sciences. 99(5). 3159–3164. 225 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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