Sin Young Choi

993 total citations
26 papers, 823 citations indexed

About

Sin Young Choi is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Oncology. According to data from OpenAlex, Sin Young Choi has authored 26 papers receiving a total of 823 indexed citations (citations by other indexed papers that have themselves been cited), including 21 papers in Molecular Biology, 8 papers in Cardiology and Cardiovascular Medicine and 5 papers in Oncology. Recurrent topics in Sin Young Choi's work include Histone Deacetylase Inhibitors Research (8 papers), Cardiac Fibrosis and Remodeling (6 papers) and Signaling Pathways in Disease (5 papers). Sin Young Choi is often cited by papers focused on Histone Deacetylase Inhibitors Research (8 papers), Cardiac Fibrosis and Remodeling (6 papers) and Signaling Pathways in Disease (5 papers). Sin Young Choi collaborates with scholars based in South Korea, China and Germany. Sin Young Choi's co-authors include Hae Jin Kee, Myung Ho Jeong, Yuhee Ryu, Gwi Ran Kim, Jin Li, Simei Sun, Zhe Piao, Jae Yeong Cho, Young Mi Seok and Hyung-Seok Kim and has published in prestigious journals such as PLoS ONE, Scientific Reports and Kidney International.

In The Last Decade

Sin Young Choi

25 papers receiving 818 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Sin Young Choi South Korea 18 471 136 122 78 68 26 823
Yuhee Ryu South Korea 18 444 0.9× 133 1.0× 114 0.9× 78 1.0× 58 0.9× 19 774
Gwi Ran Kim South Korea 18 473 1.0× 142 1.0× 121 1.0× 78 1.0× 64 0.9× 22 818
Farhan Rizvi United States 19 366 0.8× 165 1.2× 67 0.5× 48 0.6× 68 1.0× 34 884
Yanfei Xin China 19 404 0.9× 178 1.3× 106 0.9× 26 0.3× 94 1.4× 39 875
Masafumi Funamoto Japan 16 313 0.7× 127 0.9× 55 0.5× 57 0.7× 38 0.6× 61 732
Rufeng Ma China 16 657 1.4× 48 0.4× 115 0.9× 64 0.8× 89 1.3× 24 1.3k
Sana Irfan Khan India 15 321 0.7× 59 0.4× 94 0.8× 71 0.9× 55 0.8× 31 946
Pei‐Ming Chu Taiwan 18 380 0.8× 80 0.6× 76 0.6× 78 1.0× 92 1.4× 31 910
Chia-Yao Shen Taiwan 19 387 0.8× 164 1.2× 54 0.4× 36 0.5× 58 0.9× 39 836
Chuang-Ye Hong Taiwan 15 409 0.9× 82 0.6× 52 0.4× 51 0.7× 107 1.6× 24 918

Countries citing papers authored by Sin Young Choi

Since Specialization
Citations

This map shows the geographic impact of Sin Young Choi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sin Young Choi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sin Young Choi more than expected).

Fields of papers citing papers by Sin Young Choi

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sin Young Choi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sin Young Choi. The network helps show where Sin Young Choi may publish in the future.

Co-authorship network of co-authors of Sin Young Choi

This figure shows the co-authorship network connecting the top 25 collaborators of Sin Young Choi. A scholar is included among the top collaborators of Sin Young Choi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sin Young Choi. Sin Young Choi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
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Kim, Gyeong Dae, So-I Shin, Su Woong Jung, et al.. (2024). Cell Type– and Age-Specific Expression of lncRNAs across Kidney Cell Types. Journal of the American Society of Nephrology. 35(7). 870–885. 6 indexed citations
4.
Shin, So-I, Kyoung-Dong Kim, Sin Young Choi, et al.. (2023). Chromatin accessibility analysis and architectural profiling of human kidneys reveal key cell types and a regulator of diabetic kidney disease. Kidney International. 105(1). 150–164. 9 indexed citations
5.
Yoon, Cheol‐Hee, Sin Young Choi, Jung‐Eun Kim, et al.. (2021). Transcriptome Analysis Identifies Altered Biological Processes and Novel Markers in Human Immunodeficiency Virus-1 Long-Term Non-Progressors. Infection and Chemotherapy. 53(3). 489–489. 5 indexed citations
6.
Bai, Liyan, Hae Jin Kee, Sin Young Choi, et al.. (2020). HDAC5 inhibition reduces angiotensin II-induced vascular contraction, hypertrophy, and oxidative stress in a mouse model. Biomedicine & Pharmacotherapy. 134. 111162–111162. 18 indexed citations
7.
Kee, Hae Jin, Yuhee Ryu, Young Mi Seok, et al.. (2019). Selective inhibition of histone deacetylase 8 improves vascular hypertrophy, relaxation, and inflammation in angiotensin II hypertensive mice. Clinical Hypertension. 25(1). 13–13. 32 indexed citations
8.
Sun, Simei, Hae Jin Kee, Yuhee Ryu, et al.. (2019). Gentisic acid prevents the transition from pressure overload-induced cardiac hypertrophy to heart failure. Scientific Reports. 9(1). 3018–3018. 17 indexed citations
9.
Ryu, Yuhee, Hae Jin Kee, Simei Sun, et al.. (2019). Class I histone deacetylase inhibitor MS-275 attenuates vasoconstriction and inflammation in angiotensin II-induced hypertension. PLoS ONE. 14(3). e0213186–e0213186. 35 indexed citations
10.
Choi, Sin Young, Hae Jin Kee, Simei Sun, et al.. (2019). Histone deacetylase inhibitor LMK235 attenuates vascular constriction and aortic remodelling in hypertension. Journal of Cellular and Molecular Medicine. 23(4). 2801–2812. 23 indexed citations
11.
Li, Jin, Simei Sun, Yuhee Ryu, et al.. (2018). Gallic acid improves cardiac dysfunction and fibrosis in pressure overload-induced heart failure. Scientific Reports. 8(1). 9302–9302. 70 indexed citations
12.
Choi, Sin Young, Hae Jin Kee, Jin Li, et al.. (2018). Inhibition of class IIa histone deacetylase activity by gallic acid, sulforaphane, TMP269, and panobinostat. Biomedicine & Pharmacotherapy. 101. 145–154. 43 indexed citations
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Li, Jin, Zhe Piao, Simei Sun, et al.. (2017). Gallic acid attenuates pulmonary fibrosis in a mouse model of transverse aortic contraction-induced heart failure. Vascular Pharmacology. 99. 74–82. 18 indexed citations
15.
Li, Jin, Zhe Piao, Simei Sun, et al.. (2017). Gallic Acid Reduces Blood Pressure and Attenuates Oxidative Stress and Cardiac Hypertrophy in Spontaneously Hypertensive Rats. Scientific Reports. 7(1). 15607–15607. 87 indexed citations
16.
Kim, Gwi Ran, Soo‐Na Cho, Hyung‐Seok Kim, et al.. (2016). Histone deacetylase and GATA-binding factor 6 regulate arterial remodeling in angiotensin II-induced hypertension. Journal of Hypertension. 34(11). 2206–2219. 28 indexed citations
17.
Ryu, Yuhee, Jin Li, Hae Jin Kee, et al.. (2016). Gallic acid prevents isoproterenol-induced cardiac hypertrophy and fibrosis through regulation of JNK2 signaling and Smad3 binding activity. Scientific Reports. 6(1). 34790–34790. 88 indexed citations
18.
Choi, Sin Young, Yuhee Ryu, Hae Jin Kee, et al.. (2015). Tubastatin A suppresses renal fibrosis via regulation of epigenetic histone modification and Smad3-dependent fibrotic genes. Vascular Pharmacology. 72. 130–140. 73 indexed citations
19.
Kee, Hae Jin, Soo‐Na Cho, Gwi Ran Kim, et al.. (2014). Gallic acid inhibits vascular calcification through the blockade of BMP2–Smad1/5/8 signaling pathway. Vascular Pharmacology. 63(2). 71–78. 46 indexed citations
20.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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