Simon Hammans
- Clinical Biochemistry top 0.2%
- Metabolism and Genetic Disorders 15
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- Genetic Neurodegenerative Diseases 15
- Hereditary Neurological Disorders 4
- Molecular Biology top 5%
- Mitochondrial Function and Pathology 23
- ATP Synthase and ATPases Research 8
- Muscle Physiology and Disorders 7
- Neurology top 10%
- Neurological diseases and metabolism 4
- Neurology top 5%
- Neurological diseases and metabolism 4
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- Glycogen Storage Diseases and Myoclonus 5
- Co-authors
- A. E. HardingJ A Morgan-HughesMartin BrockingtonMary G. SweeneyM G SweeneyIan HoltT. James BeattieE M Brett
- Partner nations
- United KingdomUnited StatesSpain
In The Last Decade
Simon Hammans
45 papers receiving 1.7k citations
Peers
Comparison fields: 5 of 81
- Clinical Biochemistry 825
- Cellular and Molecular Neuroscience 425
- Molecular Biology 1.4k
- Neurology 121
- Neurology 211
Countries citing papers authored by Simon Hammans
This map shows the geographic impact of Simon Hammans's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Simon Hammans with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Simon Hammans more than expected).
Fields of papers citing papers by Simon Hammans
This network shows the impact of papers produced by Simon Hammans. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Simon Hammans. The network helps show where Simon Hammans may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Simon Hammans, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2021 | 1 | |
| 2 | 2017 | 31 | |
| 3 | 2017 | 21 | |
| 4 | 2013 | 28 | |
| 5 | 2010 | 8 | |
| 6 | 2009 | 3 | |
| 7 | 2009 | 47 | |
| 8 | 2005 | 97 | |
| 9 | 2005 | 40 | |
| 10 | 2004 | 48 | |
| 11 | 2001 | 61 | |
| 12 | 2001 | 51 | |
| 13 | 2000 | 33 | |
| 14 | 1998 | 16 | |
| 15 | 1995 | 123 | |
| 16 | 1995 | 69 | |
| 17 | 1994 | 43 | |
| 18 | 1993 | 114 | |
| 19 | 1993 | 61 | |
| 20 | 1992 | 40 |
About Simon Hammans
Simon Hammans is a scholar working on Clinical Biochemistry, Cellular and Molecular Neuroscience and Neurology, having authored 45 papers that have together received 1.7k indexed citations. Recurring topics across this work include Mitochondrial Function and Pathology (23 papers), Metabolism and Genetic Disorders (15 papers), Genetic Neurodegenerative Diseases (15 papers), ATP Synthase and ATPases Research (8 papers), Muscle Physiology and Disorders (7 papers), Glycogen Storage Diseases and Myoclonus (5 papers), Neurological diseases and metabolism (4 papers) and Hereditary Neurological Disorders (4 papers). The work is most often cited by research in Clinical Biochemistry (825 citations), Cellular and Molecular Neuroscience (425 citations) and Molecular Biology (1.4k citations). Simon Hammans has collaborated with scholars based in United Kingdom, United States and Spain. Frequent co-authors include A. E. Harding, J A Morgan-Hughes, Martin Brockington, Mary G. Sweeney, M G Sweeney, Ian Holt, T. James Beattie, E M Brett, A E Harding and David Robinson. Their work appears in journals such as Brain, Neuromuscular Disorders, Neurology, Journal of Neurology Neurosurgery & Psychiatry and Journal of Inherited Metabolic Disease.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.