Simon Glatigny

1.3k total citations
28 papers, 893 citations indexed

About

Simon Glatigny is a scholar working on Immunology, Rheumatology and Hematology. According to data from OpenAlex, Simon Glatigny has authored 28 papers receiving a total of 893 indexed citations (citations by other indexed papers that have themselves been cited), including 23 papers in Immunology, 10 papers in Rheumatology and 7 papers in Hematology. Recurrent topics in Simon Glatigny's work include Psoriasis: Treatment and Pathogenesis (12 papers), T-cell and B-cell Immunology (12 papers) and Spondyloarthritis Studies and Treatments (10 papers). Simon Glatigny is often cited by papers focused on Psoriasis: Treatment and Pathogenesis (12 papers), T-cell and B-cell Immunology (12 papers) and Spondyloarthritis Studies and Treatments (10 papers). Simon Glatigny collaborates with scholars based in France, United States and Belgium. Simon Glatigny's co-authors include Estelle Bettelli, Rebekka Duhen, Maxime Bréban, Mohamed Oukka, Ingrid Fert, Luiza M. Araujo, Gilles Chiocchia, Tiffany C. Blair, Rik Lories and Cécile Poulain and has published in prestigious journals such as The Journal of Immunology, Scientific Reports and Frontiers in Immunology.

In The Last Decade

Simon Glatigny

26 papers receiving 886 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Simon Glatigny France 15 585 308 187 156 115 28 893
N Karin Israel 13 617 1.1× 88 0.3× 230 1.2× 53 0.3× 82 0.7× 18 941
Toshimasa Aranami Japan 12 619 1.1× 243 0.8× 236 1.3× 70 0.4× 760 6.6× 19 1.4k
Jayagopala Reddy United States 11 1.1k 1.8× 89 0.3× 149 0.8× 34 0.2× 187 1.6× 14 1.3k
Gábor Gyülvészi Switzerland 8 1.1k 1.8× 83 0.3× 186 1.0× 64 0.4× 236 2.1× 9 1.4k
Boli Fan Canada 5 705 1.2× 163 0.5× 91 0.5× 41 0.3× 571 5.0× 6 1.1k
Claudia Haftmann Germany 13 578 1.0× 50 0.2× 243 1.3× 55 0.4× 73 0.6× 17 883
Will G. James Australia 10 440 0.8× 103 0.3× 111 0.6× 34 0.2× 44 0.4× 10 617
Christine Ewing Australia 7 574 1.0× 113 0.4× 158 0.8× 30 0.2× 252 2.2× 8 883
Mirjam Korporal Germany 12 791 1.4× 84 0.3× 117 0.6× 39 0.3× 374 3.3× 12 1.1k
Anna Garefalaki United Kingdom 7 918 1.6× 53 0.2× 260 1.4× 47 0.3× 131 1.1× 7 1.2k

Countries citing papers authored by Simon Glatigny

Since Specialization
Citations

This map shows the geographic impact of Simon Glatigny's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Simon Glatigny with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Simon Glatigny more than expected).

Fields of papers citing papers by Simon Glatigny

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Simon Glatigny. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Simon Glatigny. The network helps show where Simon Glatigny may publish in the future.

Co-authorship network of co-authors of Simon Glatigny

This figure shows the co-authorship network connecting the top 25 collaborators of Simon Glatigny. A scholar is included among the top collaborators of Simon Glatigny based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Simon Glatigny. Simon Glatigny is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
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Crémazy, Frédéric, Roula Said‐Nahal, Félicie Costantino, et al.. (2023). STAT1 deficiency underlies a proinflammatory imprint of naive CD4+ T cells in spondyloarthritis. Frontiers in Immunology. 14. 1227281–1227281. 4 indexed citations
4.
Glatigny, Simon, et al.. (2023). Mycobacterium abscessus resists the innate cellular response by surviving cell lysis of infected phagocytes. PLoS Pathogens. 19(3). e1011257–e1011257. 13 indexed citations
5.
Glatigny, Simon, Guillaume Morelle, Benjamin Hagège, et al.. (2023). Interleukin 27 is a novel cytokine with anti-inflammatory effects against spondyloarthritis through the suppression of Th17 responses. Frontiers in Immunology. 13. 1072420–1072420. 5 indexed citations
6.
Gasser, Adeline, et al.. (2022). Extracellular traps formation following cervical spinal cord injury. European Journal of Neuroscience. 57(4). 692–704. 8 indexed citations
7.
Bréban, Maxime, et al.. (2021). Lessons on SpA pathogenesis from animal models. Seminars in Immunopathology. 43(2). 207–219. 14 indexed citations
8.
Fert, Ingrid, Luiza M. Araujo, Simon Glatigny, et al.. (2021). Rodent Models of Spondyloarthritis Have Decreased White and Bone Marrow Adipose Tissue Depots. Frontiers in Immunology. 12. 665208–665208. 4 indexed citations
9.
Bréban, Maxime, et al.. (2021). Intestinal dysbiosis in spondyloarthritis – chicken or egg?. Current Opinion in Rheumatology. 33(4). 341–347. 8 indexed citations
10.
Glatigny, Simon, Barbara Höllbacher, Samantha Motley, et al.. (2019). Abatacept Targets T Follicular Helper and Regulatory T Cells, Disrupting Molecular Pathways That Regulate Their Proliferation and Maintenance. The Journal of Immunology. 202(5). 1373–1382. 45 indexed citations
11.
Araujo, Luiza M., Simon Glatigny, Gilles Chiocchia, et al.. (2019). HLA-B27 alters BMP/TGFβ signalling in Drosophila, revealing putative pathogenic mechanism for spondyloarthritis. Annals of the Rheumatic Diseases. 78(12). 1653–1662. 23 indexed citations
12.
Glatigny, Simon, et al.. (2019). Tolerogenic XCR1+ dendritic cell population is dysregulated in HLA-B27 transgenic rat model of spondyloarthritis. Arthritis Research & Therapy. 21(1). 46–46. 9 indexed citations
13.
Glatigny, Simon & Estelle Bettelli. (2018). Experimental Autoimmune Encephalomyelitis (EAE) as Animal Models of Multiple Sclerosis (MS). Cold Spring Harbor Perspectives in Medicine. 8(11). a028977–a028977. 168 indexed citations
14.
Glatigny, Simon, et al.. (2015). Integrin alpha L controls the homing of regulatory T cells during CNS autoimmunity in the absence of integrin alpha 4. Scientific Reports. 5(1). 7834–7834. 40 indexed citations
15.
Bettelli, Estelle, et al.. (2013). Integrin alpha 4 differentially affect the migration of effector and regulatory T cells (P4113). The Journal of Immunology. 190(Supplement_1). 133.10–133.10. 1 indexed citations
16.
Glatigny, Simon, Ingrid Fert, Rik Lories, et al.. (2011). Proinflammatory Th17 cells are expanded and induced by dendritic cells in spondylarthritis‐prone HLA–B27–transgenic rats. Arthritis & Rheumatism. 64(1). 110–120. 111 indexed citations
17.
Glatigny, Simon, M A Blaton, Sylvie Mistou, et al.. (2010). Treatment of collagen‐induced arthritis by Natura‐α via regulation of Th‐1/Th‐17 responses. European Journal of Immunology. 40(2). 460–469. 12 indexed citations
18.
Dhaenens, Maarten, Ingrid Fert, Simon Glatigny, et al.. (2009). Dendritic cells from spondylarthritis‐prone HLA–B27–transgenic rats display altered cytoskeletal dynamics, class II major histocompatibility complex expression, and viability. Arthritis & Rheumatism. 60(9). 2622–2632. 37 indexed citations
19.
Fert, Ingrid, Simon Glatigny, Cécile Poulain, et al.. (2008). Correlation between dendritic cell functional defect and spondylarthritis phenotypes in HLA–B27/HUMAN β2‐microglobulin–transgenic rat lines. Arthritis & Rheumatism. 58(11). 3425–3429. 29 indexed citations
20.
Glatigny, Simon, M A Blaton, Sylvie Mistou, et al.. (2007). Insights into spatial configuration of a galactosylated epitope required to trigger arthritogenic T-cell receptors specific for the sugar moiety. Arthritis Research & Therapy. 9(5). R92–R92. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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