Luiza M. Araujo

613 total citations
21 papers, 401 citations indexed

About

Luiza M. Araujo is a scholar working on Immunology, Rheumatology and Hematology. According to data from OpenAlex, Luiza M. Araujo has authored 21 papers receiving a total of 401 indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Immunology, 9 papers in Rheumatology and 7 papers in Hematology. Recurrent topics in Luiza M. Araujo's work include Psoriasis: Treatment and Pathogenesis (11 papers), Spondyloarthritis Studies and Treatments (9 papers) and Autoimmune and Inflammatory Disorders Research (7 papers). Luiza M. Araujo is often cited by papers focused on Psoriasis: Treatment and Pathogenesis (11 papers), Spondyloarthritis Studies and Treatments (9 papers) and Autoimmune and Inflammatory Disorders Research (7 papers). Luiza M. Araujo collaborates with scholars based in France, Brazil and Japan. Luiza M. Araujo's co-authors include Maxime Bréban, Gilles Chiocchia, Ingrid Fert, Simon Glatigny, Rik Lories, Orlando Ribeiro, Olga M. Ibañez, Marcelo De Franco, Wafa Hanna Koury Cabrera and Carla Eponina Carvalho-Pinto and has published in prestigious journals such as Frontiers in Immunology, Annals of the Rheumatic Diseases and European Journal of Immunology.

In The Last Decade

Luiza M. Araujo

20 papers receiving 399 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Luiza M. Araujo France 10 249 217 112 83 38 21 401
Matthew J Turner United States 7 281 1.1× 226 1.0× 136 1.2× 154 1.9× 32 0.8× 7 518
Jonathan Dixey United Kingdom 10 147 0.6× 110 0.5× 64 0.6× 63 0.8× 23 0.6× 15 304
Nicolaas E. Aerts Belgium 9 218 0.9× 104 0.5× 25 0.2× 32 0.4× 16 0.4× 11 431
Manon A. A. Jansen Netherlands 11 177 0.7× 34 0.2× 19 0.2× 130 1.6× 24 0.6× 31 390
Volker Streit Germany 11 229 0.9× 59 0.3× 23 0.2× 78 0.9× 16 0.4× 19 431
Masaki Akimoto Japan 11 321 1.3× 46 0.2× 17 0.2× 50 0.6× 42 1.1× 24 466
Adam R. Lefferts United States 8 138 0.6× 44 0.2× 13 0.1× 89 1.1× 38 1.0× 10 292
Silke Schmechel Germany 3 335 1.3× 29 0.1× 41 0.4× 61 0.7× 183 4.8× 3 465

Countries citing papers authored by Luiza M. Araujo

Since Specialization
Citations

This map shows the geographic impact of Luiza M. Araujo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Luiza M. Araujo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Luiza M. Araujo more than expected).

Fields of papers citing papers by Luiza M. Araujo

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Luiza M. Araujo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Luiza M. Araujo. The network helps show where Luiza M. Araujo may publish in the future.

Co-authorship network of co-authors of Luiza M. Araujo

This figure shows the co-authorship network connecting the top 25 collaborators of Luiza M. Araujo. A scholar is included among the top collaborators of Luiza M. Araujo based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Luiza M. Araujo. Luiza M. Araujo is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
2.
Rincheval, Vincent, et al.. (2024). TGFβ signaling pathway is altered by HLA-B27 expression, resulting in pathogenic consequences relevant for spondyloarthritis. Arthritis Research & Therapy. 26(1). 131–131. 2 indexed citations
3.
Crémazy, Frédéric, Roula Said‐Nahal, Félicie Costantino, et al.. (2023). STAT1 deficiency underlies a proinflammatory imprint of naive CD4+ T cells in spondyloarthritis. Frontiers in Immunology. 14. 1227281–1227281. 4 indexed citations
4.
Araujo, Luiza M., et al.. (2021). Effects of a low-dose IL-2 treatment in HLA-B27 transgenic rat model of spondyloarthritis. Arthritis Research & Therapy. 23(1). 193–193. 7 indexed citations
5.
Bréban, Maxime, et al.. (2021). Lessons on SpA pathogenesis from animal models. Seminars in Immunopathology. 43(2). 207–219. 14 indexed citations
6.
Fert, Ingrid, Luiza M. Araujo, Simon Glatigny, et al.. (2021). Rodent Models of Spondyloarthritis Have Decreased White and Bone Marrow Adipose Tissue Depots. Frontiers in Immunology. 12. 665208–665208. 4 indexed citations
7.
Araujo, Luiza M., et al.. (2021). Axial spondyloarthritis: emerging drug targets. Expert Opinion on Therapeutic Targets. 25(8). 633–644. 5 indexed citations
8.
Araujo, Luiza M., Simon Glatigny, Gilles Chiocchia, et al.. (2019). HLA-B27 alters BMP/TGFβ signalling in Drosophila, revealing putative pathogenic mechanism for spondyloarthritis. Annals of the Rheumatic Diseases. 78(12). 1653–1662. 23 indexed citations
9.
Glatigny, Simon, et al.. (2019). Tolerogenic XCR1+ dendritic cell population is dysregulated in HLA-B27 transgenic rat model of spondyloarthritis. Arthritis Research & Therapy. 21(1). 46–46. 9 indexed citations
10.
Rauner, Martina, Sylvia Thiele, Ingrid Fert, et al.. (2015). Loss of bone strength in HLA-B27 transgenic rats is characterized by a high bone turnover and is mainly osteoclast-driven. Bone. 75. 183–191. 9 indexed citations
11.
12.
Araujo, Luiza M., et al.. (2014). Increased Production of Interleukin‐17 Over Interleukin‐10 by Treg Cells Implicates Inducible Costimulator Molecule in Experimental Spondyloarthritis. Arthritis & Rheumatology. 66(9). 2412–2422. 31 indexed citations
13.
Glatigny, Simon, Ingrid Fert, Rik Lories, et al.. (2011). Proinflammatory Th17 cells are expanded and induced by dendritic cells in spondylarthritis‐prone HLA–B27–transgenic rats. Arthritis & Rheumatism. 64(1). 110–120. 111 indexed citations
14.
Smaniotto, Salete, Daniella Arêas Mendes‐da‐Cruz, Carla Eponina Carvalho-Pinto, et al.. (2009). Combined role of extracellular matrix and chemokines on peripheral lymphocyte migration in growth hormone transgenic mice. Brain Behavior and Immunity. 24(3). 451–461. 22 indexed citations
15.
Araujo, Luiza M., et al.. (2001). Effect of genetic modificationsby selection for immunological toleranceon fungus infection in mice. Microbes and Infection. 3(3). 215–222. 16 indexed citations
16.
Cabrera, Wafa Hanna Koury, Luiza M. Araujo, Orlando Ribeiro, et al.. (2000). Pristane-induced arthritis in mice selected for maximal or minimal acute inflammatory reaction. European Journal of Immunology. 30(2). 431–437. 37 indexed citations
17.
Araujo, Luiza M., Anne Puel, Christine Gouarin, et al.. (2000). NKT lymphocyte ontogeny and function are impaired in low antibody-producer Biozzi mice: gene mapping in the interval-specific congenic strains raised for immunomodulatory genes. International Immunology. 12(11). 1613–1622. 6 indexed citations
18.
Cabrera, Wafa Hanna Koury, et al.. (2000). Pristane-induced arthritis in mice selected for maximal or minimal acute inflammatory reaction. European Journal of Immunology. 30(2). 431–437. 3 indexed citations
19.
Araujo, Luiza M., Orlando Ribeiro, Marcelo De Franco, et al.. (1998). Innate resistance to infection by intracellular bacterial pathogens differs in mice selected for maximal or minimal acute inflammatory response. European Journal of Immunology. 28(9). 2913–2920. 46 indexed citations
20.
Massa, Solange, Olga M. Ibañez, Luiza M. Araujo, et al.. (1994). Genetic control of innate and acquired immunity. Ciencia e cultura. 46. 363–367. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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