Shuntaro Morikawa

572 total citations
27 papers, 416 citations indexed

About

Shuntaro Morikawa is a scholar working on Molecular Biology, Genetics and Endocrinology, Diabetes and Metabolism. According to data from OpenAlex, Shuntaro Morikawa has authored 27 papers receiving a total of 416 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Molecular Biology, 12 papers in Genetics and 11 papers in Endocrinology, Diabetes and Metabolism. Recurrent topics in Shuntaro Morikawa's work include Pancreatic function and diabetes (7 papers), Thyroid Disorders and Treatments (7 papers) and Endoplasmic Reticulum Stress and Disease (6 papers). Shuntaro Morikawa is often cited by papers focused on Pancreatic function and diabetes (7 papers), Thyroid Disorders and Treatments (7 papers) and Endoplasmic Reticulum Stress and Disease (6 papers). Shuntaro Morikawa collaborates with scholars based in Japan, United States and Singapore. Shuntaro Morikawa's co-authors include Toshihiro Tajima, Fumihiko Urano, Akie Nakamura, Katsura Ishizu, Nathaniel J. Hogrebe, Jeffrey R. Millman, Kristina G. Maxwell, Leonardo Velazco-Cruz, Rie Asada and Punn Augsornworawat and has published in prestigious journals such as SHILAP Revista de lepidopterología, The Journal of Clinical Endocrinology & Metabolism and Cell Metabolism.

In The Last Decade

Shuntaro Morikawa

26 papers receiving 412 citations

Peers

Shuntaro Morikawa

SURGE

EDM

GENET

Baroj Abdulkarim Switzerland

Katharina Rickenbach Switzerland

Moritz Gegg Germany

Caroline Arous Switzerland

Irene Miguel-Escalada United Kingdom

Mei‐Chyn Chao Taiwan

Le Min United States

Ayush Midha United States

P Gorden United States

Irina Krits United States

Baroj Abdulkarim Switzerland

Shuntaro Morikawa

283 ×1.2

216 ×1.5

SURGE

65 ×0.5

EDM

119 ×1.0

GENET

115 ×1.3

Citations per year, relative to Shuntaro Morikawa Shuntaro Morikawa (= 1×) peers Baroj Abdulkarim

Countries citing papers authored by Shuntaro Morikawa

Since Specialization

Citations

This map shows the geographic impact of Shuntaro Morikawa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Shuntaro Morikawa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Shuntaro Morikawa more than expected).

Fields of papers citing papers by Shuntaro Morikawa

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Shuntaro Morikawa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Shuntaro Morikawa. The network helps show where Shuntaro Morikawa may publish in the future.

Co-authorship network of co-authors of Shuntaro Morikawa

This figure shows the co-authorship network connecting the top 25 collaborators of Shuntaro Morikawa. A scholar is included among the top collaborators of Shuntaro Morikawa based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Shuntaro Morikawa. Shuntaro Morikawa is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Yamaguchi, Takeshi, Shuntaro Morikawa, Isao Yokota, et al.. (2025). Adiposity rebound and body mass index in Japanese patients with congenital hypothyroidism. Clinical Pediatric Endocrinology. 34(2). 121–130. 1 indexed citations

Tanabe, Katsuya, Wataru Nishimura, Masayuki Hatanaka, et al.. (2025). β cell dedifferentiation, the underlying mechanism of diabetes in Wolfram syndrome. Science Translational Medicine. 17(786). eadp2332–eadp2332. 3 indexed citations

Morikawa, Shuntaro, et al.. (2024). Comprehensive overview of disease models for Wolfram syndrome: toward effective treatments. Mammalian Genome. 35(1). 1–12. 5 indexed citations

Morikawa, Shuntaro, Hui Ling Ko, Ee Chee Ren, et al.. (2024). Functional Analysis of a Novel HNF4A Variant Identified in a Patient With MODY1. Journal of the Endocrine Society. 8(6). bvae090–bvae090.

Adak, Sangeeta, George D. Spyropoulos, Qiang Zhang, et al.. (2023). Palmitoylation couples insulin hypersecretion with β cell failure in diabetes. Cell Metabolism. 35(2). 332–344.e7. 41 indexed citations

Morikawa, Shuntaro, et al.. (2022). Loss of Function of WFS1 Causes ER Stress-Mediated Inflammation in Pancreatic Beta-Cells. Frontiers in Endocrinology. 13. 849204–849204. 20 indexed citations

Mahadevan, Jana, Shuntaro Morikawa, Takuya Yagi, et al.. (2020). A soluble endoplasmic reticulum factor as regenerative therapy for Wolfram syndrome. Laboratory Investigation. 100(9). 1197–1207. 9 indexed citations

Yamaguchi, Takeshi, et al.. (2020). Targeted Next-Generation Sequencing for Congenital Hypothyroidism With Positive Neonatal TSH Screening. The Journal of Clinical Endocrinology & Metabolism. 105(8). e2825–e2833. 44 indexed citations

Maxwell, Kristina G., Punn Augsornworawat, Leonardo Velazco-Cruz, et al.. (2020). Gene-edited human stem cell–derived β cells from a patient with monogenic diabetes reverse preexisting diabetes in mice. Science Translational Medicine. 12(540). 137 indexed citations

10.

Suzuki, Erina, Hirohito Shima, Masayo Kagami, et al.. (2019). (Epi)genetic defects of MKRN3 are rare in Asian patients with central precocious puberty. Human Genome Variation. 6(1). 7–7. 9 indexed citations

11.

Morikawa, Shuntaro, Akie Nakamura, Kumihiro Matsuo, et al.. (2018). Four Japanese Patients with Congenital Nephrogenic Diabetes Insipidus due to the AVPR2 Mutations. SHILAP Revista de lepidopterología. 2018(1). 6561952–6561952. 8 indexed citations

12.

Yamaguchi, Takeshi, et al.. (2018). Hypoglycemia in type 1A diabetes can develop before insulin therapy: A retrospective cohort study. Diabetes Research and Clinical Practice. 147. 87–92. 2 indexed citations

13.

Yamaguchi, Takeshi, et al.. (2018). A Japanese patient with congenital central hypothyroidism caused by a novel IGSF1 mutation. Journal of Pediatric Endocrinology and Metabolism. 31(3). 355–359. 5 indexed citations

14.

Hamazaki, Takashi, et al.. (2016). A Japanese Family with Central Hypothyroidism Caused by a Novel IGSF1 Mutation. Thyroid. 26(12). 1701–1705. 12 indexed citations

15.

Kawano, Osamu, Akie Nakamura, Shuntaro Morikawa, et al.. (2015). Spondyloepiphyseal dysplasia congenita caused by double heterozygous mutations in COL2A1. American Journal of Medical Genetics Part A. 167(7). 1578–1581. 3 indexed citations

16.

Nakamura, Akie, et al.. (2014). A Japanese family with nonautoimmune hyperthyroidism caused by a novel heterozygous thyrotropin receptor gene mutation. Pediatric Research. 75(6). 749–753. 7 indexed citations

17.

Morikawa, Shuntaro, Akie Nakamura, Masaru Fukushi, et al.. (2014). Results from 28 Years of Newborn Screening for Congenital Adrenal Hyperplasia in Sapporo. Clinical Pediatric Endocrinology. 23(2). 35–43. 18 indexed citations

18.

Morikawa, Shuntaro, et al.. (2013). A case of recurrent rhabdomyolysis associated with childhood Sjögren’s syndrome. Open Journal of Pediatrics. 3(3). 276–278. 1 indexed citations

19.

Morikawa, Shuntaro, et al.. (1984). [Pathological study of endometrial carcinogenesis in androgen-sterilized-rats].. PubMed. 36(12). 2561–6. 1 indexed citations

20.

Morikawa, Shuntaro, et al.. (1981). [Study on uterine peroxidase as a specific marker of estrogen-dependence in rat (author's transl)].. PubMed. 33(4). 499–505. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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