Shung‐Te Kao

729 citations
30 papers · 609 indexed · h-index 16
Topics
Neuroinflammation and Neurodegeneration Mechanisms (6 papers)Neurological Disease Mechanisms and Treatments (4 papers)Asthma and respiratory diseases (4 papers)
Partner nations
TaiwanCanadaChina

In The Last Decade

Shung‐Te Kao

30 papers receiving 592 citations

Peers

Shung‐Te Kao
Comparison fields: 5 of 96
  • Molecular Biology 239
  • Complementary and alternative medicine 103
  • Pharmacology 93
  • Cardiology and Cardiovascular Medicine 76
  • Pharmacology 75
Replace Piotr Skopiński with:
Piotr Skopiński Poland
Mehul R. Chorawala India
Jianxin Diao China
Xiaoliang Tong China
Shaohui Zong China
Adila A. Hamid Malaysia
Monica Rapino Italy
Eman A. Mohamed Egypt
F. Squadrito Italy
Shung‐Te Kao relative to Piotr Skopiński Poland Piotr Skopiński's profile →
Citations per field
00.5×3.3×
Piotr Skopiński · 1×
Citations per year

Countries citing papers authored by Shung‐Te Kao

Since Specialization
Citations

This map shows the geographic impact of Shung‐Te Kao's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Shung‐Te Kao with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Shung‐Te Kao more than expected).

Fields of papers citing papers by Shung‐Te Kao

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Shung‐Te Kao. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Shung‐Te Kao. The network helps show where Shung‐Te Kao may publish in the future.

Co-authorship network of co-authors of Shung‐Te Kao

This figure shows the co-authorship network connecting the top 25 collaborators of Shung‐Te Kao. A scholar is included among the top collaborators of Shung‐Te Kao based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Shung‐Te Kao. Shung‐Te Kao is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
#WorkIndexed citations
1 2
2 1
3 14
4 18
5 13
6 3
7 22
8 38
9 31
10 6
11 32
12 63
13 18
14 63
15 11
16 73
17 33
18 19
19 11
20 16

About Shung‐Te Kao

Shung‐Te Kao is a scholar working on Complementary and alternative medicine, Neurology and Pharmacology, having authored 30 papers that have together received 609 indexed citations. Recurring topics across this work include Neuroinflammation and Neurodegeneration Mechanisms (6 papers), Neurological Disease Mechanisms and Treatments (4 papers) and Asthma and respiratory diseases (4 papers). The work is most often cited by research in Complementary and alternative medicine (103 citations), Pharmacology (93 citations) and Neurology (45 citations). Shung‐Te Kao has collaborated with scholars based in Taiwan, Canada and China. Frequent co-authors include Juei‐Tang Cheng, I‐Min Liu, Yu‐Chen Lee, Shorong‐Shii Liou, Mei-Fen Chen, I-Min Liu, Tsai‐Chung Li, Chang‐Chi Hsieh, Chiachung Chen and Fun–Jou Chen. Their work appears in journals such as The FASEB Journal, International Journal of Molecular Sciences and Molecules.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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