Shuichi Otabe

1.6k total citations
22 papers, 700 citations indexed

About

Shuichi Otabe is a scholar working on Surgery, Molecular Biology and Epidemiology. According to data from OpenAlex, Shuichi Otabe has authored 22 papers receiving a total of 700 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Surgery, 8 papers in Molecular Biology and 8 papers in Epidemiology. Recurrent topics in Shuichi Otabe's work include Diabetes and associated disorders (5 papers), Adipokines, Inflammation, and Metabolic Diseases (5 papers) and Pancreatic function and diabetes (5 papers). Shuichi Otabe is often cited by papers focused on Diabetes and associated disorders (5 papers), Adipokines, Inflammation, and Metabolic Diseases (5 papers) and Pancreatic function and diabetes (5 papers). Shuichi Otabe collaborates with scholars based in Japan and France. Shuichi Otabe's co-authors include Kentaro Yamada, Nobuhiko Wada, Toshihiko Hashinaga, Hitomi Nakayama, Tomoka Fukutani, Xiaohong Yuan, Yasumichi Mori, Kazuki Yasuda, Takashi Kadowaki and Philippe Froguel and has published in prestigious journals such as PLoS ONE, Diabetes and Biochemical and Biophysical Research Communications.

In The Last Decade

Shuichi Otabe

21 papers receiving 688 citations

Peers

Shuichi Otabe
Su Sung Kim South Korea
Zoi Michailidou United Kingdom
Sonal Harbaran United States
Beate Enigk Germany
Shuichi Otabe
Citations per year, relative to Shuichi Otabe Shuichi Otabe (= 1×) peers Nobuhiko Wada

Countries citing papers authored by Shuichi Otabe

Since Specialization
Citations

This map shows the geographic impact of Shuichi Otabe's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Shuichi Otabe with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Shuichi Otabe more than expected).

Fields of papers citing papers by Shuichi Otabe

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Shuichi Otabe. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Shuichi Otabe. The network helps show where Shuichi Otabe may publish in the future.

Co-authorship network of co-authors of Shuichi Otabe

This figure shows the co-authorship network connecting the top 25 collaborators of Shuichi Otabe. A scholar is included among the top collaborators of Shuichi Otabe based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Shuichi Otabe. Shuichi Otabe is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Kita, Shunbun, Shiro Fukuda, Shuichi Otabe, et al.. (2023). Chronic hyperadiponectinemia induced by transgenic overexpression increases plasma exosomes without significantly improving glucose and lipid metabolism. Endocrine Journal. 70(6). 635–645. 2 indexed citations
2.
Nakayama, Hitomi, Shuichi Otabe, Toshihiko Hashinaga, et al.. (2017). Pivotal Role of TNF-α in the Development and Progression of Nonalcoholic Fatty Liver Disease in a Murine Model. Hormone and Metabolic Research. 50(1). 80–87. 121 indexed citations
3.
Otabe, Shuichi, et al.. (2016). Haemoglobin variants may cause significant differences in haemoglobin A1c as measured by high-performance liquid chromatography and enzymatic methods in diabetic patients: a cross-sectional study. Annals of Clinical Biochemistry International Journal of Laboratory Medicine. 54(4). 432–437. 3 indexed citations
4.
Kamohara, Ryotaro, Haruyoshi Yamaza, Tomoshi Tsuchiya, et al.. (2015). Overexpression of the adiponectin gene mimics the metabolic and stress resistance effects of calorie restriction, but not the anti-tumor effect. Experimental Gerontology. 64. 46–54. 3 indexed citations
5.
Wada, Nobuhiko, Toshihiko Hashinaga, Shuichi Otabe, et al.. (2013). Selective Modulation of Wnt Ligands and Their Receptors in Adipose Tissue by Chronic Hyperadiponectinemia. PLoS ONE. 8(7). e67712–e67712. 16 indexed citations
6.
Hashinaga, Toshihiko, Nobuhiko Wada, Shuichi Otabe, et al.. (2012). Modulation by adiponectin of circadian clock rhythmicity in model mice for metabolic syndrome. Endocrine Journal. 60(4). 483–492. 25 indexed citations
7.
Ueno, Takato, Anna Nakamura, Hitomi Nakayama, et al.. (2011). Adiponectin suppresses endoplasmic reticulum stress in nonalcoholic steatohepatitis. Experimental and Therapeutic Medicine. 2(6). 1035–1040. 13 indexed citations
8.
Mitsui, Yasuhiro, Masafumi Gotoh, Nobuhiro Fukushima, et al.. (2011). Hyperadiponectinemia enhances bone formation in mice. BMC Musculoskeletal Disorders. 12(1). 18–18. 31 indexed citations
9.
Nakayama, Hitomi, Shuichi Otabe, Xiaohong Yuan, et al.. (2009). Effects of adiponectin transgenic expression in liver of nonalcoholic steatohepatitis model mice. Metabolism. 58(7). 901–908. 15 indexed citations
10.
Otabe, Shuichi, Hitomi Nakayama, Tomoka Fukutani, et al.. (2009). Excessive maternal transmission of diabetes in Japanese families with young-onset type 2 diabetes and insulin secretion defect according to clinical features. Acta Diabetologica. 47(S1). 133–138. 11 indexed citations
11.
Otabe, Shuichi, Xiaohong Yuan, Tomoka Fukutani, et al.. (2007). Overexpression of human adiponectin in transgenic mice results in suppression of fat accumulation and prevention of premature death by high-calorie diet. American Journal of Physiology-Endocrinology and Metabolism. 293(1). E210–E218. 104 indexed citations
12.
Otabe, Shuichi, Hitomi Nakayama, Tomoka Fukutani, et al.. (2007). Clinical and genetic features of childhood-onset Type 2 diabetes in Japan. Acta Diabetologica. 44(4). 181–185. 5 indexed citations
13.
Nakayama, Hitomi, Shuichi Otabe, Takato Ueno, et al.. (2007). Transgenic mice expressing nuclear sterol regulatory element–binding protein 1c in adipose tissue exhibit liver histology similar to nonalcoholic steatohepatitis. Metabolism. 56(4). 470–475. 60 indexed citations
14.
Otabe, Shuichi, et al.. (2006). Sequential activation of caspases and synergistic β-cell cytotoxicity by palmitate and anti-Fas antibodies. Life Sciences. 79(13). 1312–1316. 13 indexed citations
16.
Mori, Yasumichi, Shuichi Otabe, Christian Dina, et al.. (2002). Genome-Wide Search for Type 2 Diabetes in Japanese Affected Sib-Pairs Confirms Susceptibility Genes on 3q, 15q, and 20q and Identifies Two New Candidate Loci on 7p and 11p. Diabetes. 51(4). 1247–1255. 195 indexed citations
17.
Otabe, Shuichi, Kazuki Yasuda, Yasumichi Mori, et al.. (1999). Molecular and Histological Evaluation of Pancreata from Patients with a Mitochondrial Gene Mutation Associated with Impaired Insulin Secretion. Biochemical and Biophysical Research Communications. 259(1). 149–156. 22 indexed citations
19.
Yamada, Kentaro, et al.. (1993). Pancreatic β-Cell–Selective Production of Tumor Necrosis Factor-α Induced by Interleukin-1. Diabetes. 42(7). 1026–1031. 27 indexed citations
20.
Otabe, Shuichi, Shigeaki Muto, Yasushi Asano, et al.. (1991). Selective Hypoaldosteronism in a Patient with Sjögren’s Syndrome: Insensitivity to Angiotensin II. ˜The œNephron journals/Nephron journals. 59(3). 466–470. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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