Shin-Long Wu

555 total citations
10 papers, 476 citations indexed

About

Shin-Long Wu is a scholar working on Molecular Biology, Genetics and Surgery. According to data from OpenAlex, Shin-Long Wu has authored 10 papers receiving a total of 476 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 3 papers in Genetics and 2 papers in Surgery. Recurrent topics in Shin-Long Wu's work include Bone health and treatments (2 papers), Heat shock proteins research (2 papers) and Bone Metabolism and Diseases (2 papers). Shin-Long Wu is often cited by papers focused on Bone health and treatments (2 papers), Heat shock proteins research (2 papers) and Bone Metabolism and Diseases (2 papers). Shin-Long Wu collaborates with scholars based in Taiwan, Germany and Netherlands. Shin-Long Wu's co-authors include Jih‐Yang Ko, Feng‐Sheng Wang, Yushan Chen, Pei-Chin Chuang, Lin‐Hsiu Weng, Da‐Wei Yeh, Wei‐Shiung Lian, Jochen Reiser, Mingwen Chen and Pei‐Hsien Lee and has published in prestigious journals such as Journal of the American Society of Nephrology, Cellular and Molecular Life Sciences and Life Sciences.

In The Last Decade

Shin-Long Wu

10 papers receiving 473 citations

Peers

Shin-Long Wu

MB

CR

RHEUM

ONCOL

OSM

Shui Qiu China

Martine Bisson Canada

Claire-Sophie Devignes France

Prem P. Dwivedi Australia

Marika Bergenstock United States

Nabil A. Rashdan United States

Jianxin Qiu China

Tao Mei China

Yuying Kou China

Sarah Franco United States

Shui Qiu China

Shin-Long Wu

316 ×1.0

MB

169 ×1.1

CR

34 ×0.5

RHEUM

61 ×1.2

ONCOL

36 ×0.7

OSM

Citations per year, relative to Shin-Long Wu Shin-Long Wu (= 1×) peers Shui Qiu

Countries citing papers authored by Shin-Long Wu

Since Specialization

Citations

This map shows the geographic impact of Shin-Long Wu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Shin-Long Wu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Shin-Long Wu more than expected).

Fields of papers citing papers by Shin-Long Wu

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Shin-Long Wu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Shin-Long Wu. The network helps show where Shin-Long Wu may publish in the future.

Co-authorship network of co-authors of Shin-Long Wu

This figure shows the co-authorship network connecting the top 25 collaborators of Shin-Long Wu. A scholar is included among the top collaborators of Shin-Long Wu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Shin-Long Wu. Shin-Long Wu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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10 of 10 papers shown

1.

Lian, Wei‐Shiung, Yushan Chen, Shin-Long Wu, et al.. (2024). Trimethylamine-N-oxide accelerates osteoporosis by PERK activation of ATF5 unfolding. Cellular and Molecular Life Sciences. 82(1). 13–13. 3 indexed citations

2.

Lian, Wei‐Shiung, Jih‐Yang Ko, Yushan Chen, et al.. (2018). Chaperonin 60 sustains osteoblast autophagy and counteracts glucocorticoid aggravation of osteoporosis by chaperoning RPTOR. Cell Death and Disease. 9(10). 741–741. 30 indexed citations

3.

Lian, Wei‐Shiung, Jih‐Yang Ko, Yi‐Chih Sun, et al.. (2018). MicroRNA-128a represses chondrocyte autophagy and exacerbates knee osteoarthritis by disrupting Atg12. Cell Death and Disease. 9(9). 919–919. 88 indexed citations

4.

Kuo, Shu‐Jui, Feng‐Sheng Wang, Jiunn‐Ming Sheen, et al.. (2014). Complement component C3: Serologic signature for osteogenesis imperfecta. Analysis of a comparative proteomic study. Journal of the Formosan Medical Association. 114(10). 943–949. 6 indexed citations

5.

Lin, Chun‐Liang, Pei‐Hsien Lee, Yung‐Chien Hsu, et al.. (2014). MicroRNA-29a Promotion of Nephrin Acetylation Ameliorates Hyperglycemia-Induced Podocyte Dysfunction. Journal of the American Society of Nephrology. 25(8). 1698–1709. 168 indexed citations

6.

Ko, Jih‐Yang, Pei-Chin Chuang, Mingwen Chen, et al.. (2013). MicroRNA-29a ameliorates glucocorticoid-induced suppression of osteoblast differentiation by regulating β-catenin acetylation. Bone. 57(2). 468–475. 50 indexed citations

7.

Wu, Re‐Wen, Tzu-Ping Lin, Jih‐Yang Ko, et al.. (2011). Cannabinoid receptor 1 regulates ERK and GSK-3β-dependent glucocorticoid inhibition of osteoblast differentiation in murine MC3T3-E1 cells. Bone. 49(6). 1255–1263. 30 indexed citations

8.

Wang, Feng‐Sheng, Re‐Wen Wu, Jih‐Yang Ko, et al.. (2011). Heat shock protein 60 protects skeletal tissue against glucocorticoid-induced bone mass loss by regulating osteoblast survival. Bone. 49(5). 1080–1089. 23 indexed citations

9.

Wang, Feng‐Sheng, et al.. (2009). Inhibition of glycogen synthase kinase-3β attenuates glucocorticoid-induced bone loss. Life Sciences. 85(19-20). 685–692. 45 indexed citations

10.

Wu, Re‐Wen, Feng‐Sheng Wang, Jih‐Yang Ko, Ching‐Jen Wang, & Shin-Long Wu. (2008). Comparative serum proteome expression of osteonecrosis of the femoral head in adults. Bone. 43(3). 561–566. 33 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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