Da‐Wei Yeh

1.0k total citations
23 papers, 816 citations indexed

About

Da‐Wei Yeh is a scholar working on Immunology, Oncology and Molecular Biology. According to data from OpenAlex, Da‐Wei Yeh has authored 23 papers receiving a total of 816 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Immunology, 8 papers in Oncology and 7 papers in Molecular Biology. Recurrent topics in Da‐Wei Yeh's work include Immune Response and Inflammation (7 papers), Cancer Immunotherapy and Biomarkers (3 papers) and Immune Cell Function and Interaction (3 papers). Da‐Wei Yeh is often cited by papers focused on Immune Response and Inflammation (7 papers), Cancer Immunotherapy and Biomarkers (3 papers) and Immune Cell Function and Interaction (3 papers). Da‐Wei Yeh collaborates with scholars based in Taiwan, United States and China. Da‐Wei Yeh's co-authors include Jih‐Yang Ko, Tsung‐Hsien Chuang, Feng‐Sheng Wang, Yiling Liu, Chao-Yang Lai, Chih‐Hao Lu, S.-L. Catherine Jin, Keigo Machida, Guann‐Yi Yu and Chiou‐Hwa Yuh and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Nature Communications.

In The Last Decade

Da‐Wei Yeh

23 papers receiving 805 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Da‐Wei Yeh Taiwan 14 365 247 196 102 82 23 816
Bongkun Choi South Korea 22 531 1.5× 346 1.4× 233 1.2× 151 1.5× 48 0.6× 43 1.2k
André Bouchot France 11 266 0.7× 191 0.8× 126 0.6× 74 0.7× 45 0.5× 15 694
Bei Wu China 10 302 0.8× 252 1.0× 139 0.7× 75 0.7× 92 1.1× 29 748
Kayvan Zainabadi United States 14 339 0.9× 58 0.2× 95 0.5× 108 1.1× 72 0.9× 29 747
Rolf Drivdahl United States 16 379 1.0× 120 0.5× 77 0.4× 68 0.7× 48 0.6× 21 766
Élaine Beaulieu Australia 15 262 0.7× 319 1.3× 126 0.6× 98 1.0× 52 0.6× 18 722
Guozhe Yang United States 12 251 0.7× 123 0.5× 100 0.5× 23 0.2× 137 1.7× 15 613
Carlos Pipaón Spain 15 484 1.3× 362 1.5× 112 0.6× 104 1.0× 34 0.4× 26 995
Morteza Motallebnezhad Iran 19 390 1.1× 382 1.5× 230 1.2× 98 1.0× 47 0.6× 37 1.1k

Countries citing papers authored by Da‐Wei Yeh

Since Specialization
Citations

This map shows the geographic impact of Da‐Wei Yeh's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Da‐Wei Yeh with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Da‐Wei Yeh more than expected).

Fields of papers citing papers by Da‐Wei Yeh

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Da‐Wei Yeh. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Da‐Wei Yeh. The network helps show where Da‐Wei Yeh may publish in the future.

Co-authorship network of co-authors of Da‐Wei Yeh

This figure shows the co-authorship network connecting the top 25 collaborators of Da‐Wei Yeh. A scholar is included among the top collaborators of Da‐Wei Yeh based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Da‐Wei Yeh. Da‐Wei Yeh is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Yeh, Da‐Wei, Xuyao Zhao, Hifzur R. Siddique, et al.. (2023). MSI2 promotes translation of multiple IRES-containing oncogenes and virus to induce self-renewal of tumor initiating stem-like cells. Cell Death Discovery. 9(1). 141–141. 6 indexed citations
2.
Yeh, Da‐Wei, Joel Marh, Hye Yeon Choi, et al.. (2022). Activated and nonactivated MSCs increase survival in humanized mice after acute liver injury through alcohol binging. Hepatology Communications. 6(7). 1549–1560. 6 indexed citations
3.
Carlos, Anthony J., Dat P. Ha, Da‐Wei Yeh, et al.. (2021). The chaperone GRP78 is a host auxiliary factor for SARS-CoV-2 and GRP78 depleting antibody blocks viral entry and infection. Journal of Biological Chemistry. 296. 100759–100759. 118 indexed citations
4.
Lin, Yen‐Yu, Yu‐Chao Wang, Da‐Wei Yeh, et al.. (2021). Gene Expression Profile in Primary Tumor Is Associated with Brain-Tropism of Metastasis from Lung Adenocarcinoma. International Journal of Molecular Sciences. 22(24). 13374–13374. 11 indexed citations
5.
Wu, Chiao‐En, Da‐Wei Yeh, Wen‐Kuan Huang, et al.. (2020). Chromosomal Instability May Not Be a Predictor for Immune Checkpoint Inhibitors from a Comprehensive Bioinformatics Analysis. Life. 10(11). 276–276. 12 indexed citations
6.
Choi, Hye Yeon, Hifzur R. Siddique, Yi Kou, et al.. (2020). p53 destabilizing protein skews asymmetric division and enhances NOTCH activation to direct self-renewal of TICs. Nature Communications. 11(1). 3084–3084. 22 indexed citations
7.
Wu, Chiao‐En, Yu‐Chao Wang, Yi‐Chen Yeh, et al.. (2020). Establishment of a novel gene panel as a biomarker of immune checkpoint inhibitor response. Clinical & Translational Immunology. 9(7). e1145–e1145. 8 indexed citations
9.
Lu, Chih‐Hao, Da‐Wei Yeh, Chao-Yang Lai, et al.. (2018). USP17 mediates macrophage-promoted inflammation and stemness in lung cancer cells by regulating TRAF2/TRAF3 complex formation. Oncogene. 37(49). 6327–6340. 68 indexed citations
10.
Lu, Chih‐Hao, Chao-Yang Lai, Da‐Wei Yeh, et al.. (2018). Involvement of M1 Macrophage Polarization in Endosomal Toll-Like Receptors Activated Psoriatic Inflammation. Mediators of Inflammation. 2018. 1–14. 66 indexed citations
11.
Yeh, Da‐Wei, Ping‐Hui Tseng, Chiou‐Hwa Yuh, et al.. (2017). CpG-oligodeoxynucleotides developed for grouper toll-like receptor (TLR) 21s effectively activate mouse and human TLR9s mediated immune responses. Scientific Reports. 7(1). 17297–17297. 27 indexed citations
12.
Lai, Chao-Yang, Da‐Wei Yeh, Chih‐Hao Lu, et al.. (2016). Thiostrepton inhibits psoriasis-like inflammation induced by TLR7, TLR8, and TLR9. The Journal of Immunology. 196(1_Supplement). 124.41–124.41. 2 indexed citations
13.
Yeh, Da‐Wei, et al.. (2016). Interplay between Inflammation and Stemness in Cancer Cells: The Role of Toll-Like Receptor Signaling. Journal of Immunology Research. 2016. 1–14. 48 indexed citations
14.
Yeh, Da‐Wei, Cheng–Yuan Kao, Huan-Yuan Chen, et al.. (2015). Identification of Thiostrepton as a Novel Inhibitor for Psoriasis-like Inflammation Induced by TLR7–9. The Journal of Immunology. 195(8). 3912–3921. 26 indexed citations
15.
Yeh, Da‐Wei, et al.. (2014). Follicular Lymphoma in Bilateral Submandibular Glands and Thyroid Gland. Journal of Medical Ultrasound. 22(3). 170–172. 1 indexed citations
16.
Ko, Jih‐Yang, et al.. (2011). Cannabinoid receptor 1 mediates glucocorticoid‐induced bone loss in rats by perturbing bone mineral acquisition and marrow adipogenesis. Arthritis & Rheumatism. 64(4). 1204–1214. 25 indexed citations
17.
Wu, Re‐Wen, Tzu-Ping Lin, Jih‐Yang Ko, et al.. (2011). Cannabinoid receptor 1 regulates ERK and GSK-3β-dependent glucocorticoid inhibition of osteoblast differentiation in murine MC3T3-E1 cells. Bone. 49(6). 1255–1263. 30 indexed citations
18.
Wang, Feng‐Sheng, Re‐Wen Wu, Jih‐Yang Ko, et al.. (2011). Heat shock protein 60 protects skeletal tissue against glucocorticoid-induced bone mass loss by regulating osteoblast survival. Bone. 49(5). 1080–1089. 23 indexed citations
19.
Wang, Feng‐Sheng, et al.. (2009). Inhibition of glycogen synthase kinase-3β attenuates glucocorticoid-induced bone loss. Life Sciences. 85(19-20). 685–692. 45 indexed citations
20.
Wang, Feng‐Sheng, et al.. (2008). Modulation of Dickkopf-1 Attenuates Glucocorticoid Induction of Osteoblast Apoptosis, Adipocytic Differentiation, and Bone Mass Loss. Endocrinology. 149(4). 1793–1801. 139 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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