Shinghua Ding

2.9k total citations
51 papers, 2.4k citations indexed

About

Shinghua Ding is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Neurology. According to data from OpenAlex, Shinghua Ding has authored 51 papers receiving a total of 2.4k indexed citations (citations by other indexed papers that have themselves been cited), including 26 papers in Molecular Biology, 25 papers in Cellular and Molecular Neuroscience and 20 papers in Neurology. Recurrent topics in Shinghua Ding's work include Neuroinflammation and Neurodegeneration Mechanisms (18 papers), Neuroscience and Neuropharmacology Research (12 papers) and Mitochondrial Function and Pathology (10 papers). Shinghua Ding is often cited by papers focused on Neuroinflammation and Neurodegeneration Mechanisms (18 papers), Neuroscience and Neuropharmacology Research (12 papers) and Mitochondrial Function and Pathology (10 papers). Shinghua Ding collaborates with scholars based in United States, China and Austria. Shinghua Ding's co-authors include Frederick Sachs, Gourav Roy Choudhury, Richard Horn, Philip G. Haydon, Xiaowan Wang, Tiannan Wang, Hermann J. Gruber, Nannan Zhang, Hailong Li and Shui Qing Ye and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Neuroscience and PLoS ONE.

In The Last Decade

Shinghua Ding

50 papers receiving 2.3k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Shinghua Ding United States 26 963 784 618 330 295 51 2.4k
Jun Gao China 26 3.6k 3.8× 792 1.0× 520 0.8× 335 1.0× 122 0.4× 63 5.6k
Masafumi Ito Japan 33 2.0k 2.1× 664 0.8× 449 0.7× 146 0.4× 47 0.2× 63 4.3k
Chenjian Li China 24 1.7k 1.8× 1.0k 1.3× 345 0.6× 177 0.5× 78 0.3× 61 4.0k
Uk Namgung South Korea 29 680 0.7× 989 1.3× 270 0.4× 192 0.6× 110 0.4× 73 2.3k
John Woulfe Canada 34 1.9k 2.0× 1.2k 1.6× 826 1.3× 147 0.4× 99 0.3× 119 4.8k
Jaume del Valle Spain 31 601 0.6× 855 1.1× 414 0.7× 374 1.1× 24 0.1× 64 2.3k
Zhijun Zhang China 34 1.0k 1.1× 387 0.5× 1.5k 2.4× 124 0.4× 72 0.2× 68 2.9k
Baorong Zhang China 34 1.1k 1.2× 1.1k 1.3× 810 1.3× 117 0.4× 58 0.2× 216 4.1k
Henrich Cheng Taiwan 33 886 0.9× 1.7k 2.1× 335 0.5× 245 0.7× 43 0.1× 100 3.7k
Wenbin Deng United States 36 1.8k 1.8× 878 1.1× 831 1.3× 267 0.8× 51 0.2× 130 4.1k

Countries citing papers authored by Shinghua Ding

Since Specialization
Citations

This map shows the geographic impact of Shinghua Ding's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Shinghua Ding with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Shinghua Ding more than expected).

Fields of papers citing papers by Shinghua Ding

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Shinghua Ding. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Shinghua Ding. The network helps show where Shinghua Ding may publish in the future.

Co-authorship network of co-authors of Shinghua Ding

This figure shows the co-authorship network connecting the top 25 collaborators of Shinghua Ding. A scholar is included among the top collaborators of Shinghua Ding based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Shinghua Ding. Shinghua Ding is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Zhang, Nannan, et al.. (2024). Dietary NMN supplementation enhances motor and NMJ function in ALS. Experimental Neurology. 374. 114698–114698. 4 indexed citations
2.
Wu, Guangfu, Nannan Zhang, Xincheng Zhang, et al.. (2022). Wireless, battery-free push-pull microsystem for membrane-free neurochemical sampling in freely moving animals. Science Advances. 8(8). eabn2277–eabn2277. 12 indexed citations
3.
Zhang, Nannan, Zhe Zhang, Ilker Ozden, & Shinghua Ding. (2022). Imaging Mitochondrial Ca<sup>2+</sup> Uptake in Astrocytes and Neurons using Genetically Encoded Ca<sup>2+</sup> Indicators (GECIs). Journal of Visualized Experiments. 1 indexed citations
4.
Ding, Shinghua, et al.. (2021). Glial cell line derived neurotrophic factor in brain repair after focal ischemic stroke. Neural Regeneration Research. 17(8). 1735–1735. 5 indexed citations
5.
Ding, Shinghua, et al.. (2021). NAD+ Metabolism and Diseases with Motor Dysfunction. Genes. 12(11). 1776–1776. 16 indexed citations
6.
Zhang, Nannan, et al.. (2020). The effect of NAMPT deletion in projection neurons on the function and structure of neuromuscular junction (NMJ) in mice. Scientific Reports. 10(1). 99–99. 18 indexed citations
7.
Wang, Xiaowan, Qiao Zhang, Nannan Zhang, et al.. (2017). Deletion of Nampt in Projection Neurons of Adult Mice Leads to Motor Dysfunction, Neurodegeneration, and Death. Cell Reports. 20(9). 2184–2200. 66 indexed citations
8.
Wang, Xiaowan, Hailong Li, & Shinghua Ding. (2016). Pre-B-cell colony-enhancing factor protects against apoptotic neuronal death and mitochondrial damage in ischemia. Scientific Reports. 6(1). 32416–32416. 25 indexed citations
9.
Choudhury, Gourav Roy & Shinghua Ding. (2015). Reactive astrocytes and therapeutic potential in focal ischemic stroke. Neurobiology of Disease. 85. 234–244. 206 indexed citations
10.
11.
Li, Hailong, Gourav Roy Choudhury, Nannan Zhang, & Shinghua Ding. (2015). Photothrombosis-induced Focal Ischemia as a Model of Spinal Cord Injury in Mice. Journal of Visualized Experiments. e53161–e53161. 3 indexed citations
12.
Li, Hailong, et al.. (2014). Histological, cellular and behavioral assessments of stroke outcomes after photothrombosis-induced ischemia in adult mice. BMC Neuroscience. 15(1). 58–58. 110 indexed citations
13.
Ding, Shinghua. (2014). Ca2+ Signaling in Astrocytes and its Role in Ischemic Stroke. Advances in neurobiology. 11. 189–211. 27 indexed citations
14.
15.
Bi, Jing, Hailong Li, Shui Qing Ye, & Shinghua Ding. (2011). Pre‐B‐cell colony‐enhancing factor exerts a neuronal protection through its enzymatic activity and the reduction of mitochondrial dysfunction in in vitro ischemic models. Journal of Neurochemistry. 120(2). 334–346. 45 indexed citations
16.
Lian, Jiazhang, Shinghua Ding, Jin Cai, et al.. (2008). Improving aquaporin Z expression in Escherichia coli by fusion partners and subsequent condition optimization. Applied Microbiology and Biotechnology. 82(3). 463–470. 38 indexed citations
17.
Ding, Shinghua, Tommaso Fellin, Yingzi Zhu, et al.. (2007). Enhanced Astrocytic Ca2+Signals Contribute to Neuronal Excitotoxicity after Status Epilepticus. Journal of Neuroscience. 27(40). 10674–10684. 216 indexed citations
18.
Ding, Shinghua & Richard Horn. (2003). Effect of S6 Tail Mutations on Charge Movement in Shaker Potassium Channels. Biophysical Journal. 84(1). 295–305. 38 indexed citations
19.
Ding, Shinghua & Frederick Sachs. (2002). Evidence for non-independent gating of P2X2 receptors expressed in Xenopusoocytes. BMC Neuroscience. 3(1). 17–17. 35 indexed citations
20.
Ding, Shinghua & Frederick Sachs. (1999). Single Channel Properties of P2X2 Purinoceptors. The Journal of General Physiology. 113(5). 695–720. 154 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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