Shengli Xu

5.0k total citations
79 papers, 3.5k citations indexed

About

Shengli Xu is a scholar working on Immunology, Molecular Biology and Oncology. According to data from OpenAlex, Shengli Xu has authored 79 papers receiving a total of 3.5k indexed citations (citations by other indexed papers that have themselves been cited), including 47 papers in Immunology, 34 papers in Molecular Biology and 11 papers in Oncology. Recurrent topics in Shengli Xu's work include T-cell and B-cell Immunology (26 papers), Immune Cell Function and Interaction (25 papers) and Immune Response and Inflammation (10 papers). Shengli Xu is often cited by papers focused on T-cell and B-cell Immunology (26 papers), Immune Cell Function and Interaction (25 papers) and Immune Response and Inflammation (10 papers). Shengli Xu collaborates with scholars based in Singapore, China and United States. Shengli Xu's co-authors include Kong‐Peng Lam, Jianxin Huo, Emanuela Castigli, Raif S. Geha, Koon-Guan Lee, Joy Tan, Xijun Ou, Ding Xiang Liu, Stacey R. Dillon and Micah J. Benson and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Journal of Clinical Investigation.

In The Last Decade

Shengli Xu

76 papers receiving 3.5k citations

Peers

Shengli Xu
Hui Shao United States
Vivian H. Gersuk United States
Aaron J. Johnson United States
George Kassiotis United Kingdom
Frank Köentgen Australia
Mireia Sospedra Switzerland
Hui Shao United States
Shengli Xu
Citations per year, relative to Shengli Xu Shengli Xu (= 1×) peers Hui Shao

Countries citing papers authored by Shengli Xu

Since Specialization
Citations

This map shows the geographic impact of Shengli Xu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Shengli Xu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Shengli Xu more than expected).

Fields of papers citing papers by Shengli Xu

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Shengli Xu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Shengli Xu. The network helps show where Shengli Xu may publish in the future.

Co-authorship network of co-authors of Shengli Xu

This figure shows the co-authorship network connecting the top 25 collaborators of Shengli Xu. A scholar is included among the top collaborators of Shengli Xu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Shengli Xu. Shengli Xu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Wang, Chelsia Qiuxia, Lei Yang, Shengli Xu, et al.. (2025). Nucleofection-based screening of chimeric antigen receptor candidates in human natural killer cells. Frontiers in Immunology. 16. 1557766–1557766.
2.
Li, Yuxing, Wenjing Chen, Rui Zhang, et al.. (2023). The histone H2B ubiquitination regulator Wac is essential for plasma cell differentiation. FEBS Letters. 597(13). 1748–1760. 2 indexed citations
3.
Neo, Shiyong, et al.. (2023). Harnessing novel strategies and cell types to overcome immune tolerance during adoptive cell therapy in cancer. Journal for ImmunoTherapy of Cancer. 11(4). e006434–e006434. 10 indexed citations
4.
Tan, Chee Wah, Feng Zhu, Wan Ni Chia, et al.. (2023). Distinctive serotypes of SARS-related coronaviruses defined by convalescent sera from unvaccinated individuals. PubMed. 1(1). 26–34. 19 indexed citations
5.
Li, Yuxing, Wenjing Chen, Rui Zhang, et al.. (2023). The RNA-binding protein hnRNP F is required for the germinal center B cell response. Nature Communications. 14(1). 1731–1731. 7 indexed citations
6.
Zhang, Rui, Ruisi Wang, Yuxing Li, et al.. (2023). Spliceosome component PHD finger 5A is essential for early B lymphopoiesis. Development. 151(2). 1 indexed citations
7.
Li, Yuxing, Wenjing Chen, Rui Zhang, et al.. (2022). Mettl14-Mediated m6A Modification Is Essential for Germinal Center B Cell Response. The Journal of Immunology. 208(8). 1924–1936. 36 indexed citations
8.
Guan, Di, et al.. (2021). Bruton’s tyrosine kinase regulates gut immune homeostasis through attenuating Th1 response. Cell Death and Disease. 12(5). 431–431. 8 indexed citations
9.
Tan, Andy Hee‐Meng, Gloria Hoi Wan Tso, Biyan Zhang, et al.. (2020). TACI Constrains TH17 Pathogenicity and Protects against Gut Inflammation. iScience. 23(11). 101707–101707. 3 indexed citations
10.
Chen, Ping, Shengli Xu, He Xiao, et al.. (2018). Blebbistatin modulates prostatic cell growth and contrapctility through myosin II signaling. Clinical Science. 132(20). 2189–2205. 6 indexed citations
11.
Ou, Xijun, Jianxin Huo, Yuhan Huang, et al.. (2018). Transcription factor YY1 is essential for iNKT cell development. Cellular and Molecular Immunology. 16(6). 547–556. 18 indexed citations
12.
Wu, Xiaoli, Ming Zhou, Hui Ding, et al.. (2013). Myelin oligodendrocyte glycoprotein induces aquaporin-4 autoantibodies in mouse experimental autoimmune encephalomyelitis. Journal of Neuroimmunology. 261(1-2). 1–6. 4 indexed citations
13.
Xu, Shengli, Jianxin Huo, Merry Gunawan, I-hsin Su, & Kong‐Peng Lam. (2009). Activated Dectin-1 Localizes to Lipid Raft Microdomains for Signaling and Activation of Phagocytosis and Cytokine Production in Dendritic Cells. Journal of Biological Chemistry. 284(33). 22005–22011. 62 indexed citations
14.
Benson, Micah J., Stacey R. Dillon, Emanuela Castigli, et al.. (2008). Cutting Edge: The Dependence of Plasma Cells and Independence of Memory B Cells on BAFF and APRIL. The Journal of Immunology. 180(6). 3655–3659. 357 indexed citations
15.
Lee, Koon-Guan, et al.. (2008). Bruton's Tyrosine Kinase Separately Regulates NFκB p65RelA Activation and Cytokine Interleukin (IL)-10/IL-12 Production in TLR9-stimulated B Cells. Journal of Biological Chemistry. 283(17). 11189–11198. 69 indexed citations
16.
Xu, Shengli, et al.. (2006). Phospholipase Cγ2 Dosage Is Critical for B Cell Development in the Absence of Adaptor Protein BLNK. The Journal of Immunology. 176(8). 4690–4698. 8 indexed citations
17.
Ng, Lai Guan, Chee H. Ng, Andrew P. R. Sutherland, et al.. (2006). BAFF costimulation of Toll‐like receptor‐activatedB‐1 cells. European Journal of Immunology. 36(7). 1837–1846. 62 indexed citations
18.
Xu, Shengli & Kong‐Peng Lam. (2002). Delayed Cellular Maturation and Decreased Immunoglobulin κ Light Chain Production In Immature B Lymphocytes Lacking B Cell Linker Protein. The Journal of Experimental Medicine. 196(2). 197–206. 20 indexed citations
19.
Tan, Joy, Siew Cheng Wong, Samuel Ken‐En Gan, Shengli Xu, & Kong‐Peng Lam. (2001). The Adaptor Protein BLNK Is Required for B Cell Antigen Receptor-induced Activation of Nuclear Factor-κB and Cell Cycle Entry and Survival of B Lymphocytes. Journal of Biological Chemistry. 276(23). 20055–20063. 88 indexed citations
20.
Xu, Shengli, Siew Cheng Wong, & Kong‐Peng Lam. (2000). Cutting Edge: B Cell Linker Protein Is Dispensable for the Allelic Exclusion of Immunoglobulin Heavy Chain Locus But Required for the Persistence of CD5+ B Cells. The Journal of Immunology. 165(8). 4153–4157. 21 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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